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한용진,조봉균,정재훈,이수빈,박해윤,이태기,채희백,홍진수 순천향대학교 기초과학연구소 1998 순천향자연과학연구 논문집 Vol.4 No.2
Thin amorphous carbon films were deposited by a Q-switched Nd;YAG 532nm with beam power density of ?? on the high purity graphite (99.7%). The pressure of vacuum chamber was ?? Torr. In order to estimate the quality of the Pulsed Laser Deposition films one of the most important optical properties, bandgap energy, was characterized by transmission and reflection in the range of the visible, and an optical direct bandgap energy of 2.45eV and indirect bandgap energy 0.36eV were obtained. Surface morphology of amorphous film was investigated by AFM (Atomic Force Microscopy). Its surface roughness is 70nm. Comparing our results with the published values in the literature we have confidence that our films show the good quality for optical properties.
김태윤 ( Tae Yoon Kim ),권윤희 ( Yoon Hee Kwon ),이동원 ( Dong Won Lee ),백승철 ( Seung Churl Baek ),조백기 ( Baik Kee Cho ) 대한피부과학회 1996 대한피부과학회지 Vol.34 No.6
Acrodermatitis enteropathica is a rare autosomal recessive disorder characterized by alopecia, a ral and periorificial derma.titis and gastro-intestinal disturbances such as diarrhea. This syndrome is caused by malab, orption of zinc. The mean serum zinc level is usually low in untreated patients and the oral administration of zinc sulfate can clear the skin lesions and other symptoms. We report a three-month-ol:~3 female baby with acrodermatitis enteropathica showing a normal serum zinc level in whom diapnosis was confirmed by typical skin lesions, clinical symptoms and response to oral zinc therapy. (Kor J Dermatol 1996;34(5): 984 987)
전교통 동맥류의 분류와 수술을 시행한 135 예에 대한 임상보고
백민우,김달수,김영,박춘근,김문찬,이상원,조태훈,조병일,이재수,윤석훈,하영수,강준기,송진언 대한신경외과학회 1986 Journal of Korean neurosurgical society Vol.15 No.1
During the 10-year period up to December 1984, 176 patients with anterior communicating aneurysm (ACOMA) among total 467 patients of intracranial aneurysms were admitted to this Catholic Medical Center. Of these, 135 cases of ACOMA were operated by direct intracranial procedures. To analyse the factors influencing the mortality involving in surgery of 135 patients with ACOMA, a classification of ACOMA was attempted. The origins and projections of aneurysms, anatomical variations were analysed with the aid of angiography, intraoperative findings and intraoperative photographs which permitted the establishment of a classification of ACOMA with their direction. Our classification of operated 135 cases of ACOMA are seven types: 32 anterior(23.7%), 30 anterior-rostral(22.2%), 42 anterior-caudal (31.1%), 3 posterior(2.2%), 18 posterior-rostral(13.4%), 4 posteriorcaudal (3.0%), caudal 6(4.4%), respectively. Most of ACOMA projecting anteriorly, anterio-rostrally were situated above or between the optic nerve, and the less frequent posterior-caudal and inferior aneurysms were in close proximity to hypothalamic branches of the anterior communicating artery and A₂ segment. There was on overall operative mortality of 6%. The surgical morbidity and mortality were significantly higher in the posteriorly projecting group, garticulary in the posterior-caudal direction. In direct surgery of ACOMA, subpial resection of the gyrus rectus was sffective for not only anteriorly, but also posteriorly directing aneurysms.
비열탄성형 Fe-X%Mn-Y%Si HCP 마르텐사이트 합금의 진동감쇠기능에 관한 연구
백승한,윤태영,전중환,최종술,윤준현 연세대학교 산업기술연구소 1993 논문집 Vol.25 No.2
The specific damping capacities of Fe-X%Mn-Y%Si non-thermoelastic martensitic alloys were investigated, using Fopple-Pertz type torsional pendulum apparatus ?? alloy showed the highest damping capacity in the Fe-X%Mn system. The addition of silicon deteriorated the damping capacity of the ?? alloy. Although the transformation repetition. γ↔ε, was done by heating and cooling between 285℃ and room temperature, the damping capacity of the Fe-17%Mn alloy was not improved by the thermal cycles. However the damping capacity of Fe-17%Mn-1%Si was increased by one or two cycles, showing no improvement on further cycles.
백봉수,손윤호,손태중,변진석 大韓成形外科學會 1989 Archives of Plastic Surgery Vol.16 No.2
Progress in therapy has virtually eliminated death from primary shock and early infection following burns, but late mortality of patients from extensive burns has not been diminished. One of the lethal sequelae observed in patients with severe skin burns is lung damage. However, the pathophysiologic mechanism responsible for this sequela is not clearly elucidated. The purpose of this study is to investigate the ultrastructural changes of the lung injury and its mechanism in severly burned experimental animals. Experimental animals were divided into 5 groups;groupⅠ, 5 Sprague-Dawley rats served as a control'groupⅡ,20 rats subjected to burns and sacrificed 1,3,5 and 7 days postburn intervals;groupⅢ,5 rats injured by burns and teated with dimethyl sulfoxide(DMSO), 1.5ml/kg administered intraperitoneally for 5 days, sacrifeced 5 days postburn;groupⅣ,5 rats injured by burns and treated with gabexate mesilate,50 mg/kg administered intraperitoneally for 5 days, sacrificed 5 days postburn;groupⅤ 5 rats subjucted to burns and treated with both DMSO and gabexate mesilate. The lungs of experimental animals were examined by electron imcroscopy. In order to observe the changes of alveolar capillary permeability in each group, vascular clearance test was performed with horseradish peroxidase(HRP). The results were as follows; Although minimal changes of the lungs began to appear from the 3rd day postburn, marked changes, such as alveolar capillary endothelial swelling, neutrophil and platelet infiltration, mitochondrial swelling of typeⅡ pneumocytes, along with interstitial edema were observed from the 5th day postburn. These changes were apparently alleviated in experimental groups treated with DMSO, gabexate mesilate, and DMSO plus gabexate mesilate. Alveolar capillary permeability was markedly incresed from the 5th day postburn,demonstrated by leakage of HRP into the interstitium,which was diministhed in the experimental groups treated with DMSO,gabexate mesilate,and DMSO plus gabexate mesilate. These findings suggest that the ultrastructural changes of the lungs induced by severe skin burns are significantly improved by absorption of hydroxyl radicals and by blocking of complement activation. It can be concluded that lung injury after severe skin burns is induced by activation of humoral mediator system.