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강진경(Jin Kyung Kang),최흥재(Heung Jai Choi),이상인(Sang In Lee),박용준(Yong Jun Park),김경희(Kyung Hee Kim),허균(Kyoon Huh) 대한소화기학회 1982 대한소화기학회지 Vol.14 No.2
N/A Eosinophilic gastroenteritis is an infiltrative disease of unknown etiology which can involve the entire G-I tract and associated with peripheral eosinophilia. Eosinophilic gastroenteritis was first recognized by Kaijser in 1937, since then little more than 100 case reports have appeared in the worlds literature but rare in korean literature. A 28 year old male patient was referred for evaluation of indigestion and abdominal distension of 15 days duration He denied episodes of allergic disorder of him and his family members. On physical exam. His abdomen was soft but morderately distended with shifting dullness and fluid wave. Paracentesis was performed, peritoneal fluid was slightly bloody and direct smear with stain revealed 100% of eosinophile. WBC count was 11,000/mm with 41% eosinophils. He underwent Ba enema and small bowel series which revealed mucosal thickening of entire jejunum and ileum and seperation of intestinal loops. He was treated with corticosteroid and responded dramatically with a complete resolution of ascites and other G-I symptoms.
대뇌 침범이 확인된 Henoch - Schonlein 자반증 1 예
정경섭(Kyung Sup Chung),김경철(Kyung Cheol Kim),조현명(Hyeon Myeong Cho),최규헌(Kyu Hun Choi),한광협(Kwang Hyup Han),홍천수(Chein Soo Hong),허균(Kyoon Huh),홍순원(Soon Won Hong),최인준(In Joon Choi) 대한내과학회 1991 대한내과학회지 Vol.40 No.3
Henoch-Schonlein purpura is a generalized small vessel vasculitis of the hypersensitivity type characterized by the association of cutaneous lesions with gastrointestinal, joint and/or renal symptoms. The syndrome may occur in infants as well as in adults, but it occurs most commonly in childhood. Neurologic involvement was recognized in 1914 by Osler who described a child with allergic purpura associated with a hemiplegia. Since then, relatively little has been written in the medical literature concerning the neurologic manifestations of this illness. The neurologic symptoms and signs may be due to the involvement of the meningeal and cerebral parenchymal vessels by the fibrinoid necrotizing arteriolitis or arteritis of this disease causing the diffuse cerebral ischemia. And it may be aggrevated by a consequence of the hypertension and metabolic derangements that can accompany renal involvement. We report a case of Henoch-Schonlein purpura with neurologic involvement, which was menifestated by typical clinical pictures including severe headache and generalized seizure associated with multifocal low density on computed tomography and abnormal EEG findings.
SH-SY5Y Human Neuroblastoma Cell에서 에스트로겐의 신경세포 보호효과에 대한 기전
염지현,김희,홍현석,방오영,허균,묵인희 대한치매학회 2003 Dementia and Neurocognitive Disorders Vol.2 No.1
Apoptosis is one major mechanism underlying neuronal cell death in degenerative diseases in the central nervous system. Previous studies have shown that estrogen has neuroprotective effects in several neuronal cell death model systems. In the present study, we established a staurosporine-induced apoptotic neuronal cell death system with human SH-SY5Y cell line. 17b-estradiol(E2) blocked staurosporine-induced apoptosis of SH-SY5Y cells as revealed by MTT as well as LDH assay. The cells showed typical DNA fragmentation at 4 h and 8 h following staurosporine treatment, which was attenuated by E2 pretreatment. Staurosporine-induced chromatin condensation was also reduced by E2 treatment. Because apoptotic stimuli are known to induce caspase-3 activation and PARP cleavage, we examined whether E2 blocks these processes. E2 markedly attenuated staurosporine-induced casapase-3 activation and PARP cleavage. This study shows that E2 blocks staurosporine-induced apoptosis in SH-SY5Y human neuroblastoma cells, suggesting that E2 is a neuroprotective agent that may be used for the treatment of neurodegenerative diseases such as Alzheimer's disease and Parkinson's disease.