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위 ( 胃 ) 에 발생 ( 發生 ) 한 혈관외피세포종 ( 血管外皮細胞腫 ) ( 例 )
이계희(Kye Heui Lee),박경주(Kyung Joo Park),김동린(Dong Chan Kim),이수영(Su Young Lee),경춘숙(Choon suk Kyoung),최상전(Sang Jeon Choi),손인(In Sohn),박성훈(Seong Hoon Park),전호경(Ho Kyung Chun),최신은(Shin Eun Choi) 대한소화기학회 1990 대한소화기학회지 Vol.22 No.3
Hemangiopericytoma of stomach is an extremely rare tumor, Recentely we experienced a case of gastric. hemangiopericytoma of a 51-year-old man with massive upper gastrointestinal bleeding, The rarity of thie case is emphasized and the literature reviewed, The hemangiopericytoea of stomach is characteril by mild epigaetric pain, slowly growing mass and upper gaetrointestinl bleeding, In the limited exrience reported so far, partial gastric resection may be an effective form of treitment.
혈청 제4형 콜라겐 7S Domain 측정에 의한 만성 간염의 섬유화 정도 예측
이계희(Kye Heui Lee),박성훈(Seong Hoon Park),최신은(Shin Eun Choi),김안나(An Na Kim),김성용(sung Yong Kim),임선희(Sun Hee Lim),임용성(Yong Sung Lim),손인(In Son) 대한소화기학회 1993 대한소화기학회지 Vol.25 No.2
N/A The degree of fibrosis is a major prognostic factor in chronic liver disease. Although liver biopsy is essential to assess the degree of fibrosis, a more simple diagnostic method would be helpful for its invasive nature. Type IV collagen is one of major constituents of basement membrane and serum collagen IV-7S level reflects the degree of fibrosis. To evaluate the usefulness of serum collagen IV-7S in assessing the state of hepatic fibrosis, we compared the level of serum collagen IV-7S in various liver diseases and normal volunteers. The normal value of the serum collagen IV-7S was 3.6+- 1.2 ng/ml. There was no statistically significant difference in serum collagen IV-7S levels among the group of acute hepatitis, fatty liver, chronic persistent hepatitis and the normal controls. And there was no significant difference between the group of chronic active hepatitis and liver cirrhosis, either. But the level of serum collagen IV-7S of the group of chronic active hepatitis and liver cirrhosis was significantly higher than that of the group of acute viral hepatitis, fatty liver and chronic persistent hepatitis. These results suggest that the measurement of serum collagen IV-7S is a valuable aid for evaluating the state of fibrrosis in chronic liver disease.
우리나라 원발성 간세포암 조직에서 중합효소 연쇄반응을 이용한 B 형 간염 바이러스 DNA 의 검출
이계희(Kye Heui Lee),고광철(Kwang Cheol Koh),이효석(Hyo Suk Lee),송인성(In Sung Song),김정룡(Chung Yong Kim) 대한내과학회 1994 대한내과학회지 Vol.46 No.3
Objectives : The advances in molecular biological technique enable us to detect the integrated HBV DNA in the chromosomal DNA of hepatocellular carcinoma (HCC) cells by Southern blot analysis. The recent development of the polymerase chain reaction made possible the detection of single-copy HBV DNA in a hepatocyte. Regarding the oncogenesis of HBV-related HCC, X-gene is highly suggested to be oncogenic in a transgenic mice model. We investigate the detection rate of each HBV subgenome in the HCC tissues to approach to the human hepatocarcinogenesis using the most sensitive polymerase chain reaction (PCR). Methods: Thirteen formalin-fixed paraffin-embedded HCC-tissue blocks which were aseptically obtained by surgical resection in Korea were studied; 10 were positive for serum HBsAg and one was positive for serum anti-HBc alone, and two were negative for all HBV serological markers. Cellular DNA was extracted from a deparaffinized slice of each tissue block, and then each subgenomic HBV DNA was amplified by PCR using a pair of primers corresponding to each subgenome. The amplified HBV DNA subgenomic fragment was detected by Southern blot hybridization. Results : Among 10 HBsAg-positive cases, the s-gene, c-gene and x-gene nucleotide sequences were detected in 5(50%), 4(40%) and 9(90%) cases, respectively, All 3 gene sequences were detectable in 4(40%) cases. However, HBV DNA subgenomic fragment was not found in the anti-HBc-only case and the cases without any HBV serological marker. Conclusion: It was concluded that HBV may not be involved at all in the development of HBsAg-negative HCC while HBV, especially x-gene, played a major etiologic role in HBsAg-positive HCC in human hepatocarcinogenesis.
( Yong Jik Lee ),( Su Hak Heo ),( Kye Heui Lee ),( Young Sook Lee ),( Ji Young Kim ),( Ji Eun Choi ),( Yoon Hwa Jeong ) 대한소화기학회 2007 SIDDS Vol.9 No.-
Background/Aims: Nonalcoholic fatty liver disease is related with obesity and hyperlipidemia. PPAR α is highly expressed and promotes fatty acid oxidation by PPAR α agonists or inhibits fat denaturalization in liver. The aim of this study is to elucidate the effect of PPAR α agonist on rat liver and fatty acid oxidative metabolism of hepatocytes in hyperlipidemic condition. Methods: Male Splague Dawley rats were divided into 3 groups; fed normal diet, high fat diet (containing 40% fat), and high fat diet with fenofibrate (100 mg/day) for 10 weeks. After sacrificing, the concentrations of adiponectin, leptin, and HDL in serum was measured by ELISA or RIA. The fat contents in liver were determined by H & E staining method. The effects of PPAR α agonist in HepG2 cells exposed to high fatty acid (0.1 mM palmitate) were also determined by RT-PCR for fatty acid binding protein (FABP), carnitine palmitoyltransferase I (CPT I), medium chain acyl-CoA dehydrogenase (MCAD) and Western blot analyses for MCAD and PPAR α. Results: Diet intake, body weight, and fat weight in the group fed PPAR α agonist were decreased by 22.4%, 39.4% and 30.7% respectively compared to the group fed only high fat diet. Adiponectin, leptin, and HDL were increased by 370%, 141%, and 196% in group fed PPAR α agonist. Lipid droplets were clearly observed in liver of the rats fed high fat diet, however, they were never viewed in liver treated by PPAR α agonist In HepG2 cells, the mRNA levels of MCAD, FABP and CPT I decreased by hyperlipidemic condition were increased significantly by treatments of PPAR α agonist, and the protein expression levels of MCAD and PPAR α were elevated by PPAR α agonist in hyperlipidemic condition. Conclusions: In hyperlipidemic condition, PPAR α agonist seems to reduce appetite through increasing of leptin level and inhibit fat accumulation via elevating of adiponectin in rat and may ameliorate fatty acid oxidative metabolism in HepG2 cells. Consequently, it was supposed that PPAR α agonist can inhibit early fatty liver disease.
이재용(Jae Yong Lee),성천모(Chun Mo Sung),경춘숙(Choon Suk Kyeong),이수영(Su Young Lee),김동찬(Dong Chan Kim),이계희(Kye Heui Lee),최상전(Sang Jeon Choi),손인(In Son),박성훈(Seong Hoon Park) 대한내과학회 1991 대한내과학회지 Vol.40 No.6
Due to the high propensity of rupture and cardiac death, early diagnosis of pseudoaneurysms of the left ventricle is clinically very important. Diagnosis can be carried out by noninvasive techniques, such as echocardiography and radioisotope gated blood pool scan, if the disease is suspected. We report a case of left ventricular pseudoaneruysm after acute myocardial infarction of the inferoposterolateral wall, with the findings of echocardiography, radioisotope blood pool scan, and chest computerized tomography.
Kim Jeong-Min,Kim Heui Man,Lee Eun Jung,Jo Hye Jun,Yoon Youngsil,Lee Nam-Joo,Son Junseock,Lee Ye-Ji,Kim Mi Seon,Lee Yong-Pyo,Chae Su-Jin,Park Kye Ryeong,Cho Seung-Rye,Park Sehee,Kim Su Jin,Wang Eunbye 질병관리본부 2020 Osong Public Health and Research Persptectives Vol.11 No.3
Objectives Coronavirus Disease-19 (COVID-19) is a respiratory infection characterized by the main symptoms of pneumonia and fever. It is caused by the novel coronavirus severe acute respiratory syndrome Coronavirus-2 (SARS-CoV-2), which is known to spread via respiratory droplets. We aimed to determine the rate and likelihood of SARS-CoV-2 transmission from COVID-19 patients through non-respiratory routes. Methods Serum, urine, and stool samples were collected from 74 hospitalized patients diagnosed with COVID-19 based on the detection of SARS-CoV-2 in respiratory samples. The SARS-CoV-2 RNA genome was extracted from each specimen and real-time reverse transcription polymerase chain reaction performed. CaCo-2 cells were inoculated with the specimens containing the SARS-COV-2 genome, and subcultured for virus isolation. After culturing, viral replication in the cell supernatant was assessed. Results Of the samples collected from 74 COVID-19 patients, SARS-CoV-2 was detected in 15 serum, urine, or stool samples. The virus detection rate in the serum, urine, and stool samples were 2.8% (9/323), 0.8% (2/247), and 10.1% (13/129), and the mean viral load was 1,210 ± 1,861, 79 ± 30, and 3,176 ± 7,208 copy/µL, respectively. However, the SARS-CoV-2 was not isolated by the culture method from the samples that tested positive for the SARS-CoV-2 gene. Conclusion While the virus remained detectable in the respiratory samples of COVID-19 patients for several days after hospitalization, its detection in the serum, urine, and stool samples was intermittent. Since the virus could not be isolated from the SARS-COV-2-positive samples, the risk of viral transmission via stool and urine is expected to be low.