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상용 S/W를 이용한 소형가스터빈엔진 회전체의 동적 구조해석 및 검증
정혁진(Chung, Hyuk-Jin),이종원(Lee, Chong-Won),홍성욱(Hong, Seong-Wook),유태규(Yoo, Tae-Gyu) 한국소음진동공학회 2010 한국소음진동공학회 논문집 Vol.20 No.1
The accurate prediction of dynamic characteristics of high speed rotors, such as gas turbines, is important to avoid the possibility of operating the machinery near the critical speeds or unstable speed regions. However, the dynamic analysis methods and softwares for gas turbines have been developed in the process of producing many gas turbines by manufacturers and most of them have seldom been disclosed to the public. Recently, commercial FEM softwares, such as SAMCEF, ANSYS and NASTRAN, started supporting some rotordynamics analysis modules based on 3-D finite elements. In this paper, the dynamic analysis method using commercial S/W, especially ANSYS, is attempted for the small-size gas turbine engine rotor, and the analysis capability and limitations of its rotordyamics module are evaluated for further improvement of the module. As the preliminary procedure, the rotordyamic analysis capability of ANSYS was tested and evaluated with the reference models of the well-known dynamics. The limitations in application of the rotordynamics module were then identified. Under the current capability and limitations of ANSYS, it is shown that Lee diagram, a new frequency-speed diagram enhanced with the concept of $H{\infty}$ in rotating machinery, can be indirectly obtained from FRFs computed from harmonic response analysis of ANSYS. Finally, it is demonstrated based on the modeling and analysis method developed in the process of the S/W verification that the conventional Campbell diagram, Lee diagram, mode shapes and critical speeds of the small-size gas turbine engine rotor can be computed using the ANSYS rotordynamics module.
저산소 상태로 유도된 백서 뇌세포 배양에서 Minocycline의 뇌보호 효과
하경아,양범석,김진경,김홍태,하성진,이종원,정혜리,김우택,Ha, Kyung A,Yang, Bum Seok,Kim, Jin Kyung,Kim, Hong Tae,Ha, Sung Jin,Lee, Jong Won,Chung, Hai Lee,Kim, Woo Taek 대한소아청소년과학회 2003 Clinical and Experimental Pediatrics (CEP) Vol.46 No.11
목 적 : 미노사이클린이 in vivo 연구에서 뇌보호 효과가 있는것으로 알려져 있어 본 연구에서는 미노사이클린이 저산소 상태로 유발된 백서 뇌세포 배양에서 고사를 억제하는 뇌보호 효과가 있는지를 알아보고자 하였다. 방 법: 임신 18일된 백서의 대뇌피질 신경세포를 배양하여 정상산소 상태군과 저산소 상태군으로 두 군으로 나누고, 정상산소 상태는 5% $CO_2$ 배양기에, 저산소 상태는 1% $CO_2$ 배양기에서 세포 수를 세면서 수일간 처리하여 현미경하에서 충분한 손상이 있다고 판단되면 두 군을 각각 대조군, 미노사이클린 $1{\mu}g/mL$, $10{\mu}g/mL$로 처리한 군으로 나누어서 실험하였다. TUNEL 및 DAPI 염색으로 세포 고사 상태를 통계학적으로 처리하였다. 결 과: 정상산소 상태군에서 대조군과 미노사이클린 $1{\mu}g/mL$ 투여군, 미노사이클린 $10{\mu}g/mL$ 투여군 3군 모두에서 통계학적으로 유의한 차이가 있었다(P<0.01). 저산소 상태군에서 대조군과 미노사이클린 투여군에서 통계학적으로 유의한 차이가 있었으나(P<0.01), 미노사이클린 투여군 간에는 통계학적으로 유의한 차이가 없었다(P>0.05). 정상산소 상태군과 저산소 상태군간의 대조군과 미노사이클린 $1{\mu}g/mL$ 투여군의 비교에서는 정상산소 상태군이 저산소 상태군보다 평균이 낮아 통계학적으로 유의한 차이가 있었다(P<0.01). 정상산소 상태군과 저산소 상태군간의 미노사이클린 $10{\mu}g/mL$ 투여군의 비교에서는 정상산소 상태군이 저산소 상태군보다 다소 평균이 낮았으나 통계학적으로 유의성은 없었다(P>0.05). 결 론 : 결론적으로 미노사이클린은 고사를 억제하는 기전으로 저 산소 상태나 정상 산소 상태에서 뇌보호 효과가 있었다. Purpose : In vivo, minocycline appears to be neuroprotective. Thus, the neuroprotective effects of minocycline were studied in a rat brain cortical cell culture induced by hypoxia. Methods : Cultured cells from the brains of Sprague-Dawley rats were divided into two sets of groups : normoxia groups treated with 5% $CO_2$ and hypoxia groups treated with 1% $CO_2$. After several days of incubation, the control groups were not treated with minocycline, while the sample groups were treated with either 1 or $10{\mu}g/mL$ of minocycline. The damaged cells were observed under a microscope, while apoptosis was detected using a TUNEL assay control-stained with DAPI. Results : Among the normoxia groups, the control and sample groups treated with 1 and $10{\mu}g/mL$ of minocycline were all statistically significantly different from each other. Meanwhile, among the hypoxia groups, although the control was significantly different from the sample groups, there was no statistically significant difference between the sample groups. When comparing the normoxia and hypoxia groups, there was a statistically significant difference between the control groups and sample groups treated with $1{\mu}g/mL$ of minocycline, yet no significant difference between the sample groups treated with $10{\mu}g/mL$ of minocycline. Conclusion : Minocycline was found to be neuroprotective in normoxia and hypoxia induced rat brain cortical cell cultures.
정진욱,오득영,서제원,문석호,이중호,이종원,안상태,김창현,Chung, Jin-Wook,Oh, Deuk-Young,Seo, Je-Won,Moon, Suk-Ho,Lee, Jung-Ho,Rhie, Jong-Won,Ahn, Sang-Tae,Kim, Chang-Hyun 대한성형외과학회 2010 Archives of Plastic Surgery Vol.37 No.5
Purpose: Central giant cell granuloma is a rare, benign giant cell tumor which commonly develops in areas near the teeth. It accounts for approximately less than 7% of benign tumors of the mandible. Clinically, central giant cell granuloma is classifed into aggressive and non-aggressive type, and usually requires surgical treatment. There has been no report of central giant cell granuloma in plastic surgery field of the country, and we report a case with a brief review of the diagnosis and treatment of the disease. Methods: A 23-year-old male presented with a hard, non-tender, growing mass with the size of $4.0{\times}3.0\;cm$ on mandible for several months. Computed tomography scan showed a solid mass within thinned outer cortex on mandible. The thinned outer cortex was excised with the mass and the inner cortex was partially removed burring. After the tumor removal, mandible was fixed by reconstruction plate. Results: Pathologic report showed numerous large multinucleated giant cells, diffusely distributed in a background of ovoid-to-spindle-shaped mononuclear cells. There was no evidence of recurrence after 1 year follow up. Bony defect was regenerated and we removed the reconstruction plate. Conclusion: Removal of central giant cell granuloma results in defect of outer cortex, which can be reconstructed by using reconstruction plate, autologous bone graft or bone cement. We used reconstruction plate as a conservative method to induce secondary healing of the outer cortical defect area, which resulted in normal mastication and occlusion with no recurrence.