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Hee Zoo Kim,Seung Hwan Oh,Chung Guk Park,Eun Hye Koo,Hye Ran Oh,Mi Kyoung Lee,Sang Ho Lim Korean Society of Critical Care Medicine 2005 Acute and Critical Care Vol.20 No.1
We had done one-lung ventilation using 9 Fr wire-guided endobronchial blocker and outer diameter 41-mm flexible fiberoptic bronchoscope in ruptured esophageal patient who expected difficult tracheal intubation and in esophageal cancer patient who was in need of mechanical ventilation during and after the operation.
Hee-Tae. Roh,Hyoung Zoo,Ha․,Jin-Hee,Woo․,Yul-Hyo,Lee Kangeun,Ko․,Ju-Yong. Bae 한국유화학회 2018 한국응용과학기술학회지 Vol.35 No.3
We investigated the effect of different exercise intensities on biomarkers of oxidant-antioxidant balance, inflammation, and muscle damage. Eighteen healthy and untrained male subjects were enrolled.Subjects were randomly and equally assigned to a moderate-intensityexercise(MIE, 65%VO 2 max) group(n=9) or a high-intensity exercise(HIE, 85%VO 2 max) group(n=9).Blood samples were collectedimmediately pre-exercise, post-exercise, and 60min post-exercisetoexamine oxidant-antioxidant balance(d-ROMs; BAP), inflammation(CRP; fibrinogen), muscle damage(CK; LDH), and lactate. Serum d-ROMs and BAP levels were significantly increased post-exercise compared with pre-exercise levels in HIE group(p<0.05). Lactate levels were significantly increased post-exercise compared pre-exercise levels in both the MIE and HIE groups(p<0.05). In addition, post-exercise serum d-ROMs and plasma lactate levels were significantly higher in the HIE group than in the MIE group(p<0.05). These results suggest that although relatively high-intensity exercises may increase oxidative stress levels in the body, they do not produce inflammatory response and/or muscle damage.
Roh, Hee-Tae,Ha, Hyoung Zoo,Woo, Jin-Hee,Lee, Yul-Hyo,Ko, Kangeun,Bae, Ju-Yong The Korean Society of Applied Science and Technolo 2018 한국응용과학기술학회지 Vol.35 No.3
We investigated the effect of different exercise intensities on biomarkers of oxidant-antioxidant balance, inflammation, and muscle damage. Eighteen healthy and untrained male subjects were enrolled.Subjects were randomly and equally assigned to a moderate-intensityexercise(MIE, $65%VO_2max$) group(n=9) or a high-intensity exercise(HIE, $85%VO_2max$) group(n=9).Blood samples were collectedimmediately pre-exercise, post-exercise, and 60min post-exercisetoexamine oxidant-antioxidant balance(d-ROMs; BAP), inflammation(CRP; fibrinogen), muscle damage(CK; LDH), and lactate. Serum d-ROMs and BAP levels were significantly increased post-exercise compared with pre-exercise levels in HIE group(p<0.05). Lactate levels were significantly increased post-exercise compared pre-exercise levels in both the MIE and HIE groups(p<0.05). In addition, post-exercise serum d-ROMs and plasma lactate levels were significantly higher in the HIE group than in the MIE group(p<0.05). These results suggest that although relatively high-intensity exercises may increase oxidative stress levels in the body, they do not produce inflammatory response and/or muscle damage.
Jeong Hee Han,Bing-Zhe Hong,Young Geun Kwak,Kuichang Yuan,Woo Hyun Park,Sung Zoo Kim,Suhn Hee Kim 대한생리학회-대한약리학회 2007 The Korean Journal of Physiology & Pharmacology Vol.11 No.3
Gap junction protein, connexin, is expressed in endothelial cells of vessels, glomerulus, and renin secreting cells of the kidney. The purpose of this study was to investigate the role of gap junction in renin secretion and its underlying mechanisms using As 4.1 cell line, a renin-expressing clonal cell line. Renin release was increased proportionately to incubation time. The specific gap junction inhibitor, 18-beta glycyrrhetinic acid (GA) increased renin release in dose-dependent and time- dependent manners. Heptanol and octanol, gap junction blockers, also increased renin release, which were less potent than GA. GA-stimulated renin release was attenuated by pretreatment of the cells with amiloride, nifedipine, ryanodine, and thapsigargin. GA dose-dependently increased intracellular Ca<SUP>2+</SUP> concentration, which was attenuated by nifedipine, nimodipine, ryanodine, and thapsigargin. However, RP-cAMP, chelerythrine, tyrphostin A23, or phenylarsine oxide did not induced any signi</SUP>ficant change in GA-stimulated increase of intracellular Ca2+ concentration. These results suggest that gap junction plays an important role on the regulation of renin release and intracellular Ca<SUP>2+</SUP> concentration in As 4.1 cells.
증례보고 : 승모판 치환술 후 발생한 의인성 대동맥 박리
김희주 ( Hee Zoo Kim ),임상호 ( Sang Ho Lim ),박성우 ( Sung Woo Park ),김난숙 ( Nan Suk Kim ),이미경 ( Mi Kyoung Lee ) 대한마취과학회 2007 Korean Journal of Anesthesiology Vol.53 No.4
Iatrogenic aortic dissection (IAD) is a life-threatening complication that can occur during open heart surgery, therefore IAD requires early diagnosis and prompt management. We describe here a case of IAD that occurred during mitral valve replacement. The transesophageal echocardiography (TEE) evaluation revealed features indicative of acute aortic dissection and the tear was successfully repaired by interposition of a graft. (Korean J Anesthesiol 2007; 53: 524~7)
Jin, Young-Hee,Jung, Ji-Hun,Jeon, Su-Jin,Lee, Jae-Kyoo,Oh, Young-Hee,Choi, Sung-Min,Chae, Young-Zoo The Korean Society for Microbiology 2012 Journal of Bacteriology and Virology Vol.42 No.3
A total of 91 non-typhoid Salmonella isolated from pediatric patients with diarrhea in Seoul from 2003 to 2009 was tested for antimicrobial susceptibility of nalidixic acid (NA). Forty strains of NA resistance or intermediate susceptible non-typhoid Salmonella were identified and their minimum inhibitory concentrations (MICs) of NA, ciprofloxacin (CIP), and norfloxacin (NOR) were determined. Of the 40 isolates, 26 were resistant to NA (MIC > $256{\mu}g/ml$). Only one isolate was high-level resistant to CIP ($12{\mu}g/ml$) and NOR ($48{\mu}g/ml$). Mutations in gyrA and parC genes were studied by PCR and sequencing. All NA-resistant isolates carried point mutations in the gyrA quinolone resistance determining regions (QRDR) at codon 83 or 87 (MICs of NA, > $256{\mu}g/ml$; MICs of CIP, $0.047{\sim}0.25{\mu}g/ml$; MICs of NOR, $0.38{\sim}1.5{\mu}g/ml$). A double change in GyrA was found in one Salmonella Enteritidis (MIC of CIP, $12{\mu}g/ml$; MIC of NOR, $48{\mu}g/ml$). In respect of the ParC protein, a single change at Thr57${\rightarrow}$Ser was found in 3 isolates (MICs of NA, > $256{\mu}g/ml$; MICs of CIP, $0.19{\sim}0.25{\mu}g/ml$; MICs of NOR, $1{\mu}g/ml$). At the same time, these strains changed from Ser83 to Tyr in the gyrA. The result of the investigation for the prevalence of plasmid-mediated quinolone resistance (PMQR) genes, 14 isolates harbored qnr gene among 40 isolates. All of 14 isolates showed decreased susceptibility at NA (MICs $4{\sim}16{\mu}g/ml$) and except one strain, all of qnr genes were identified as qnrB. Mutations in the gyrA gene and production of PMQR determinants were critical for quinolone resistance and decreased susceptibility to fluoroquinolone in these isolates.
Han, Jeong-Hee,Hong, Bing-Zhe,Kwak, Young-Geun,Yuan, Kui-Chang,Park, Woo-Hyun,Kim, Sung-Zoo,Kim, Suhn-Hee The Korean Society of Pharmacology 2007 The Korean Journal of Physiology & Pharmacology Vol.11 No.3
Gap junction protein, connexin, is expressed in endothelial cells of vessels, glomerulus, and renin secreting cells of the kidney. The purpose of this study was to investigate the role of gap junction in renin secretion and its underlying mechanisms using As 4.1 cell line, a renin-expressing clonal cell line. Renin release was increased proportionately to incubation time. The specific gap junction inhibitor, 18-beta glycyrrhetinic acid (GA) increased renin release in dose-dependent and time-dependent manners. Heptanol and octanol, gap junction blockers, also increased renin release, which were less potent than GA. GA-stimulated renin release was attenuated by pretreatment of the cells with amiloride, nifedipine, ryanodine, and thapsigargin. GA dose-dependently increased intracellular $Ca^{2+}$ concentration, which was attenuated by nifedipine, nimodipine, ryanodine, and thapsigargin. However, RP-cAMP, chelerythrine, tyrphostin A23, or phenylarsine oxide did not induced any significant change in GA-stimulated increase of intracellular $Ca^{2+}$ concentration. These results suggest that gap junction plays an important role on the regulation of renin release and intracellular $Ca^{2+}$ concentration in As 4.1 cells.
오현희(Hyun Hee Oh),황금택(Keum Taek Hwang),김무열(Muyeol Kim),이희권(Hee Kwon Lee),김성주(Sung Zoo Kim) 한국식품영양과학회 2008 한국식품영양과학회지 Vol.37 No.6
본 연구에서는 국내에서 재배되거나 야생의 나무딸기류 20 종류를 수집하여 무게, 수분함량, 과즙의 당도, pH, 산도, pH를 측정하고, 과즙의 당 조성 및 유기산 조성 분석하여 각각의 특성을 검토하였다. 실험에 사용한 나무딸기류 과일의 무게는 0.9~7.8 g이었고, 과일의 수분함량은 82.0~90.3%였다. 나무딸기류 과즙의 당도는 6.9~16.9%(Brix)로 다양하였다. HPLC로 분석한 나무딸기류 과즙의 당은 glucose(총당 함량의 40.7~84.0%(w/w))와 fructose(6.5~57.5%)가 주를 이루고 있었고, 일부 과일에는 sucrose와 xylose도 미량 존재하였다. 과즙의 pH는 2.8~3.9였고, 적정 산도는 0.6~2.9%(w/v, citric acid)였다. 나무딸기류 과즙의 유기산을 HPLC로 분석한 결과, citric acid(총산 함량의 14.6~87.5%(w/w))와 malic acid (9.6~87.1%)가 주를 이루고 있었고, 일부 과일에는 tartaric acid, succinic acid, oxalic acid 등이 함유되어 있었다. In this study, 20 kinds of raspberries and blackberries, cultivated or wild in Korea, were collected. Weight, moisture content, sugar content, sugar composition, pH, titratable acidity and acid composition of the fruits were determined. Weight of each of the fruits and moisture contents in the fruits were 0.9~7.8 g and 26~62%, respectively. The sugar contents in the fruit juices were 6.9~16.9% (Brix). The major sugars in the fruits were glucose (40.7~84.0% (w/w) of the total sugars) and fructose (6.5~57.5%). Sucrose and xylose were also detected in a small quantity. pH and titratable acidity of the berry juices were 2.8~3.9% (w/v, citric acid) and 0.6~2.9%, respectively. The major organic acids were citric acid (14.6~87.5% (w/w) of the total acids) and malic acid (9.6~87.1%). Succinic, tartaric and oxalic acids were also detected in a small amount.