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※ 단, EBSCO ASC/BSC(오후 5시~익일 오전 9시 무료 이용)
저자들은 완전 관해된 급성골수성 백혈병 환자에서 콩팥에 발생한 과립성 육종 1예를 경험하였기에 문헌 고찰과 함께 보고하는 바이다. Granulocytic sarcoma is an extramedullary tumor composed of granulocytic precursor cells such as myeloblast or monoblast. These tumor were originally celled "Chloroma" because of the green color imparted by the heavy concentration of the enzyme myeloperoxidase present in myelogenous leukemic cells. The tumor usually develops during the course of myelogenous leukemia or myeloproliferative disorders and may represent the inital manifestation of leukemia. Rarely, granulocytic sarcoma is recognized as an isolated tumor without any evidence of leukemia. However, in such cases, leukemia generally develops within 1 to 2 years of the diagnosis of granulocytic sarcoma. Accurate inital diagnosis of granulocytic sarcoma in a nonleukemic patient may reduce the risk of subsequent acte myelocytic leukemia if appropriate chemotherapy is begun in time. We are reporting a case of a 21-year-old man who was diagnosed as having an isolated granulocytic sarcoma of the right kidney. Eight months ago, he was treated with induction chemotherapy due to acute myelocytic leukemia and complete remission was occurred.
Crohn's disease and ulcerative colitis known as inflammatory bowel diseases, are chronic inflammatory disorders involving the gastrointestinal tract. Because the cause of both disease remains unknown, a curative therapy has been lacking. Until recently, conventional treatments have focused on nonspecific suppression of the inflammatory process and are often effective in achieving symptomatic control, but frequently fail to prevent relapse when administered over time. Recent advances in basic research have provided new insights into the role of specific immune cells and their mediators in intestinal inflammation. Such understanding has led to the development of biologic treatments directed at altering specific pathogenic mechanisms that have the potential to modifiy the natural course of these disorders. The compounds directed against important regulatory cytokines such as tumor necrosis factor-alpha (TNF-α) have demonstrated the greatest clinical efficacy to date. These clinical findings are consistent with scientific observations that have suggested a central role of TNF-α in chronic intestinal inflammation. It is probable that biologic therapy will play an important role in the future treatment of inflammatory bowel diseases.