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좌측 앞대뇌동맥 뇌경색에 의한 무동무언증에 대한 치험 1례
홍혜정,류혜랑,김경민,김영균,Hong, Hae-jeong,Ryu, Hae-rang,Kim, Kyung-min,Kim, Young-kun 대한한방내과학회 2016 大韓韓方內科學會誌 Vol.37 No.2
Objectives: This clinical study was conducted to evaluate the effectiveness of traditional Korean medicine (TKM) on a patient with akinetic mutism.Method: A patient with akinetic mutism diagnosed with left-anterior cerebral artery infarction was treated with acupuncture and herbal medication; the patient was then evaluated for any improvements in clinical symptoms.Results: Improvements in akinetic mutism symptoms were observed following the TKM treatment.Conclusion: This study shows that TKM treatment for akinetic mutism due to left-anterior cerebral artery infarction may be an effective treatment option.
Akinetic Mutism and Cognitive-Affective Syndrome Caused by Unilateral PICA Infarction
양영순,김정은,이정석,김상윤 대한신경과학회 2007 Journal of Clinical Neurology Vol.3 No.4
A 42-year-old man with left posterior inferior cerebellar artery (PICA) infarction presented with akinetic mutism and cognitive impairment. Initially he suffered from akinetic mutism and MRI-documented infarction in the distribution of the left PICA. Twelve days later he developed cognitive impairment. Neuropsychological tests were performed, with the results corrected for age and education being compared with published Korean norms. Impaired performances were evident on executive function testing, with difficulties in planning, abstract reasoning, set-shifting, and perseveration. Akinetic mutism and cognitive-affective syndrome may be a manifestation of unilateral PICA infarction.
무동무언증과 인지기능장애를 주소로 하는 급성 백질뇌증 환자 치험 1례
김보은 ( Bo Eun Kim ),정대규 ( Dae Kyoo Chung ),김상호 ( Sang Ho Kim ),이경석 ( Kyung Suk Lee ) 대한한방신경정신과학회 2013 동의신경정신과학회지 Vol.24 No.4
This case report presents a 34-year-old female patient. Her chief complaints are akinetic mutism and cognitive dysfunctions. We have diagnosed her with leukoencephalopathy. She was treated with Korean traditional herbal medicines (Joojackwhan) for 33 days continuously. The effects of treatment were measured by the Korean Mini - Mental State Examination and Modified Bathel Index. After treatment, her chief complaints have been improved. Furthermore, the K-MMSE, MBI scores increased. From this case, we deduced that the Korean traditional herbal medicines (Joojackwhan) could be effective for cognitive dysfunctions and akinetic mutism caused by leukoencephalopathy.
이익성,김영도,권세윤,김중석 대한신경과학회 2011 Journal of Clinical Neurology Vol.7 No.4
Background Oriental medicines have been associated with severe psychiatric, neurological, and other adverse medical events. These medicines occasionally cause a typical reversible toxic encephalopathy, but most such cases are not recognized because these adverse events are complex and are associated with other systemic signs and symptoms. Case Report We describe a married couple with rapid progressive cognitive impairment and akinetic mutism after taking the same oriental medicines on the same day. Brain magnetic resonance images of the couple showed typical leukoencephalopathy in the periventricular whitematter and basal ganglia regions, bilaterally. Conclusions The development of neurobehavioral symptoms and toxic leukoencephalopathy in both patients following the ingestion of oriental medicines is suggestive of a cause-and-effect association, although such a relationship needs to be verified.
Neuroanatomical Mechanism of Cerebellar Mutism After Stroke
Lee, Sekwang,Na, Yoon Hye,Moon, Hyun Im,Tae, Woo Suk,Pyun, Sung-Bom Korean Academy of Rehabilitation Medicine 2017 Annals of Rehabilitation Medicine Vol.41 No.6
<P>Cerebellar mutism (CM) is a rare neurological condition characterized by lack of speech due to cerebellar lesions. CM is often reported in children. We describe a rare case of CM after spontaneous cerebellar hemorrhage. The patient showed mutism, irritability, decreased spontaneous movements and oropharyngeal apraxia. Diffusion tensor imaging revealed significant volume reduction of medial frontal projection fibers from the corpus callosum. In Tracts Constrained by UnderLying Anatomy (TRACULA) analysis, forceps major and minor and bilateral cingulum-angular bundles were not visualized. Cerebello-frontal pathway reconstructed from the FMRIB Software Library showed continuity of fibers, with decreased number of fibers on qualitative analysis. These results suggest that cerebello-frontal disconnection may be a neuroanatomical mechanism of CM. Damage of brain network between occipital lobe, cingulate and cerebellum caused by hemorrhage may also have role in the mechanism of CM in our case.</P>