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韓國 自生 범부채(Belamcanda chinensis (L.) DC.)의 種子發芽에 關한 硏究
朴潤点,劉成吾,崔權雄,鄭然玉 한국화훼연구회 1995 화훼연구 Vol.4 No.1
These studies were carried out to investigate the seed germination of Belamcanda chinensis (L.) DC. The results obtained are as follows : 1.Optimum temperature of seed germination was 25℃, and the seed germination rate was raised to 92% by 20/30℃ alternative temperature treatment. 2.Under the condition of moist low temperature storage, the germination rate was 53% at 42 days after sowing, but showed the germination rate of 11% at same period with dry low temperature storage. 3.The germination rate was higher in the light condition than in the dark condition. 4.The germination rate was higher when the seed coat was removed than when the seed coat was not removed or the seed coat was stratificated. 5.The germination rate was 80% with 100ppm GA3 treatment, and 76% wihh soaking treatment of 1% KOH solution for 24 hours.
김용구,박일웅,안윤근,배애란,최상덕 國立麗水大學校 環境問題硏究所 2004 環境硏究論文集 Vol.6 No.-
This present study was conducted to determine the effects of sea water quality and sediment quality on growth and body composition in poly culture system, Paralichthys olivaceus and Urechis unicinctus. The seawater temperature of the experimental groups was ranges of 17.1~23.8℃, the lowest in 0 day and the highest in 70 day. The concentration of COD was 1.2~1.6㎎/ℓ, DIN 6.19~28.89㎎/ℓ in the D group. Mean IL concentration was 5.42~5.07% and it was maximum in 0 day and relatively minimum 70day. The concentration of AVS was 0.36~0.22㎎/g-dry, COD 4.36~4.08㎎/g-dry in the D group. Growth and feed intake of fish were affected by diets and feeding frequencies(P<0.05). There were differences at all experimental groups especially C group and control group but A, B group and C, D group were not difference. The lowest increased body weight was observed 4.7g in the D experimental group and the highest experimental group was observed 8.6g in the C group.
Choi, Dukhyun,Lee, Keun Young,Jin, Mi-Jin,Ihn, Soo-Ghang,Yun, Sungyoung,Bulliard, Xavier,Choi, Woong,Lee, Sang Yoon,Kim, Sang-Woo,Choi, Jae-Young,Kim, Jong Min,Wang, Zhong Lin Royal Society of Chemistry 2011 ENERGY AND ENVIRONMENTAL SCIENCE Vol.4 No.11
<P>In this paper, we present a simple, low-cost and flexible hybrid cell that converts individually or simultaneously low-frequency mechanical energy and photon energy into electricity using piezoelectric zinc oxide (ZnO) in conjunction with organic solar cell design. Since the hybrid cell is designed by coupled piezoelectric and photoconductive properties of ZnO, this is a naturally hybrid architecture without crosstalk and an additional assembling process to create multi-type energy scavengers, thus differing from a simple integration of two different energy generators. It is demonstrated that the behavior of a piezoelectric output is controlled from alternating current (AC) type to direct current (DC)-like type by tailoring mechanical straining processes both in the dark and under light illumination. Based on such controllability of output modes, it is shown that the performance of the hybrid cell is synergistically enhanced by integrating the contribution made by a piezoelectric generator with a solar cell under a normal indoor level of illumination. Our approach clearly demonstrates the potential of the hybrid approach for scavenging multi-type energies whenever and wherever they are available. Furthermore, this work establishes the methodology to harvest solar energy and low-frequency mechanical energies such as body movements, making it possible to produce a promising multi-functional power generator that could be embedded in flexible architectures.</P> <P>Graphic Abstract</P><P>Naturally hybrid flexible energy generator that converts individually or simultaneously low-frequency mechanical energies and photon energy into electricity was developed. <IMG SRC='http://pubs.rsc.org/services/images/RSCpubs.ePlatform.Service.FreeContent.ImageService.svc/ImageService/image/GA?id=c1ee02080c'> </P>
Three-Dimensional LiDAR Data Classifying to Extract Road Point in Urban Area
Yun-Woong Choi,Young-Woon Jang,Hyo-Jong Lee,Gi-Sung Cho IEEE 2008 IEEE geoscience and remote sensing letters Vol.5 No.4
<P>The Light Detection and Ranging (LiDAR) system is one of the best ways to accurately and effectively gather 3-D terrain information. However, it is complicated to process the LiDAR cloud data due to its irregularity and large number of collected data points. This letter proposes a novel method to automatically extract urban road network from 3-D LiDAR data. This method uses height and reflectance of LiDAR data, and clustered road point information. Geometric information of general roads is also applied to correctly extract road points group. The proposed method has been tested on various urban areas which contain complicated road networks. The results demonstrate that the integration of height, reflectance, and geometric information of roads is a crucial factor that distinguishes the proposed method in its ability to reliably and correctly classify road points.</P>
Choi, Jae-Woong,Kim, Jong-Wook,Nguyen, Lap P.,Nguyen, Huu C.,Park, Eun-Mee,Choi, Dong Hwa,Han, Kang Min,Kang, Sang Min,Tark, Dongseob,Lim, Yun-Sook,Hwang, Soon B. Korean Society for Molecular and Cellular Biology 2020 Molecules and cells Vol.43 No.5
Hepatitis C virus (HCV) propagation is highly dependent on cellular proteins. To identify the host factors involved in HCV propagation, we previously performed protein microarray assays and identified the LIM and SH3 domain protein 1 (LASP-1) as an HCV NS5A-interacting partner. LASP-1 plays an important role in the regulation of cell proliferation, migration, and protein-protein interactions. Alteration of LASP-1 expression has been implicated in hepatocellular carcinoma. However, the functional involvement of LASP-1 in HCV propagation and HCV-induced pathogenesis has not been elucidated. Here, we first verified the protein interaction of NS5A and LASP-1 by both in vitro pulldown and coimmunoprecipitation assays. We further showed that NS5A and LASP-1 were colocalized in the cytoplasm of HCV infected cells. NS5A interacted with LASP-1 through the proline motif in domain I of NS5A and the tryptophan residue in the SH3 domain of LASP-1. Knockdown of LASP1 increased HCV replication in both HCV-infected cells and HCV subgenomic replicon cells. LASP-1 negatively regulated viral propagation and thereby overexpression of LASP-1 decreased HCV replication. Moreover, HCV propagation was decreased by wild-type LASP-1 but not by an NS5A binding-defective mutant of LASP-1. We further demonstrated that LASP-1 was involved in the replication stage of the HCV life cycle. Importantly, LASP-1 expression levels were increased in persistently infected cells with HCV. These data suggest that HCV modulates LASP-1 via NS5A in order to regulate virion levels and maintain a persistent infection.
Salmonella typhi 에 의한 감염성 심내막염 1 예
윤정호,최재웅,신익균,안태훈,이성광,최윤영,장호열,윤형선,강영숙,최인석 대한내과학회 1996 대한내과학회지 Vol.51 No.4
We report a first case of subacute infective endocarditis due to S. typhi in Korea. The patient is a 58-year-old man and despite of antibiotic therapy, he presented with intermittent fever and chills for about 8 weeks after dental extraction in the Philippines. Salmonella typhi was isolated from blood culture and 2-D echocardiogram showed a 12㎜×4㎜ sized vegetation on ventricular surface of the aortic right coronary cusp. He was treated with intravenous cefotaxime for 24 days successfully and after discharge took oral ofloxacine for 4 weeks without any complication.
Choi, He Yun,Park, Ji Hye,Jang, Woong Bi,Ji, Seung Taek,Jung, Seok Yun,Kim, Da Yeon,Kang, Songhwa,Kim, Yeon Ju,Yun, Jisoo,Kim, Jae Ho,Baek, Sang Hong,Kwon, Sang-Mo The Korean Society of Applied Pharmacology 2016 Biomolecules & Therapeutics(구 응용약물학회지) Vol.24 No.4
Cardiovascular disease is the most common cause of death in diabetic patients. Hyperglycemia is the primary characteristic of diabetes and is associated with many complications. The role of hyperglycemia in the dysfunction of human cardiac progenitor cells that can regenerate damaged cardiac tissue has been investigated, but the exact mechanism underlying this association is not clear. Thus, we examined whether hyperglycemia could regulate mitochondrial dynamics and lead to cardiac progenitor cell dysfunction, and whether blocking glucose uptake could rescue this dysfunction. High glucose in cardiac progenitor cells results in reduced cell viability and decreased expression of cell cycle-related molecules, including CDK2 and cyclin E. A tube formation assay revealed that hyperglycemia led to a significant decrease in the tube-forming ability of cardiac progenitor cells. Fluorescent labeling of cardiac progenitor cell mitochondria revealed that hyperglycemia alters mitochondrial dynamics and increases expression of fission-related proteins, including Fis1 and Drp1. Moreover, we showed that specific blockage of GLUT1 improved cell viability, tube formation, and regulation of mitochondrial dynamics in cardiac progenitor cells. To our knowledge, this study is the first to demonstrate that high glucose leads to cardiac progenitor cell dysfunction through an increase in mitochondrial fission, and that a GLUT1 blocker can rescue cardiac progenitor cell dysfunction and downregulation of mitochondrial fission. Combined therapy with cardiac progenitor cells and a GLUT1 blocker may provide a novel strategy for cardiac progenitor cell therapy in cardiovascular disease patients with diabetes.
( He Yun Choi ),( Ji Hye Park ),( Woong Bi Jang ),( Seung Taek Ji ),( Seok Yun Jung ),( Da Yeon Kim ),( Songhwa Kang ),( Yeon Ju Kim ),( Jisoo Yun ),( Jae Ho Kim ),( Sang Hong Baek ),( Sang Mo Kwon ) 한국응용약물학회 2016 Biomolecules & Therapeutics(구 응용약물학회지) Vol.24 No.4
Cardiovascular disease is the most common cause of death in diabetic patients. Hyperglycemia is the primary characteristic of diabetes and is associated with many complications. The role of hyperglycemia in the dysfunction of human cardiac progenitor cells that can regenerate damaged cardiac tissue has been investigated, but the exact mechanism underlying this association is not clear. Thus, we examined whether hyperglycemia could regulate mitochondrial dynamics and lead to cardiac progenitor cell dysfunction, and whether blocking glucose uptake could rescue this dysfunction. High glucose in cardiac progenitor cells results in reduced cell viability and decreased expression of cell cycle-related molecules, including CDK2 and cyclin E. A tube formation assay revealed that hyperglycemia led to a significant decrease in the tube-forming ability of cardiac progenitor cells. Fluorescent labeling of cardiac progenitor cell mitochondria revealed that hyperglycemia alters mitochondrial dynamics and increases expression of fission-related proteins, including Fis1 and Drp1. Moreover, we showed that specific blockage of GLUT1 improved cell viability, tube formation, and regulation of mitochondrial dynamics in cardiac progenitor cells. To our knowledge, this study is the first to demonstrate that high glucose leads to cardiac progenitor cell dysfunction through an increase in mitochondrial fission, and that a GLUT1 blocker can rescue cardiac progenitor cell dysfunction and downregulation of mitochondrial fission. Combined therapy with cardiac progenitor cells and a GLUT1 blocker may provide a novel strategy for cardiac progenitor cell therapy in cardiovascular disease patients with diabetes.