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ALICE Collaboration,Abelev, B.,Adam, J.,Adamova, D.,Adare, A.M.,Aggarwal, M.M.,Aglieri Rinella, G.,Agocs, A.G.,Agostinelli, A.,Aguilar Salazar, S.,Ahammed, Z.,Ahmad, N.,Ahmad Masoodi, A.,Ahn, S.A.,Ahn North-Holland Pub. Co 2012 Physics letters: B Vol.718 No.2
The p<SUB>T</SUB>-differential inclusive production cross section of the prompt charm-strange meson D<SUB>s</SUB><SUP>+</SUP> in the rapidity range |y|<0.5 was measured in proton-proton collisions at s=7 TeV at the LHC using the ALICE detector. The analysis was performed on a data sample of 2.98x10<SUP>8</SUP> events collected with a minimum-bias trigger. The corresponding integrated luminosity is L<SUB>int</SUB>=4.8 nb<SUP>-1</SUP>. Reconstructing the decay D<SUB>s</SUB><SUP>+</SUP>→φπ<SUP>+</SUP>, with φ→K<SUP>-</SUP>K<SUP>+</SUP>, and its charge conjugate, about 480 D<SUB>s</SUB><SUP>+/-</SUP> mesons were counted, after selection cuts, in the transverse momentum range 2<p<SUB>T</SUB><12 GeV/c. The results are compared with predictions from models based on perturbative QCD. The ratios of the cross sections of four D meson species (namely D<SUP>0</SUP>, D<SUP>+</SUP>, D<SUP>@?+</SUP> and D<SUB>s</SUB><SUP>+</SUP>) were determined both as a function of p<SUB>T</SUB> and integrated over p<SUB>T</SUB> after extrapolating to full p<SUB>T</SUB> range, together with the strangeness suppression factor in charm fragmentation. The obtained values are found to be compatible within uncertainties with those measured by other experiments in e<SUP>+</SUP>e<SUP>-</SUP>, ep and pp interactions at various centre-of-mass energies.
D -Meson Azimuthal Anisotropy in Midcentral Pb-Pb Collisions at sNN=5.02 TeV
Acharya, S.,Adamová,, D.,Adolfsson, J.,Aggarwal, M. M.,Aglieri Rinella, G.,Agnello, M.,Agrawal, N.,Ahammed, Z.,Ahmad, N.,Ahn, S. U.,Aiola, S.,Akindinov, A.,Alam, S. N.,Alba, J. L. B.,Albuquerque American Physical Society 2018 Physical Review Letters Vol.120 No.10
<P>The azimuthal anisotropy coefficient v(2) of prompt D-0, D+, D*+, and D-s(+) mesons was measured in midcentral (30%-50% centrality class) Pb-Pb collisions at a center-of-mass energy per nucleon pair root s(NN)=5.02 TeV, with the ALICE detector at the LHC. The D mesons were reconstructed via their hadronic decays at midrapidity, |y| < 0.8, in the transverse momentum interval 1 < p(T) < 24 GeV/c. The measured D-meson v(2) has similar values as that of charged pions. The D-s(+) v(2), measured for the first time, is found to be compatible with that of nonstrange D mesons. The measurements are compared with theoretical calculations of charm-quark transport in a hydrodynamically expanding medium and have the potential to constrain medium parameters.</P>
Choe, N.,Kwon, D. H.,Shin, S.,Kim, Y. S.,Kim, Y. K.,Kim, J.,Ahn, Y.,Eom, G. H.,Kook, H. Elsevier Science B.V., Amsterdam. 2017 FEBS letters Vol.591 No.7
<P>S100 calcium-binding protein A4 (S100A4) induces proliferation and migration of vascular smooth muscle cells (VSMCs). We aimed to find the microRNA regulating S100A4 expression. S100A4 transcripts are abruptly increased in the acute phase of carotid arterial injury 1 day later (at day 1) but gradually decreases at days 7 and 14. Bioinformatics analysis reveals that miR-124 targets S100A4. VSMC survival is attenuated by miR-124 mimic but increased by miR-124 inhibitor. miR-124 decreases immediately after carotid arterial injury but dramatically increases at days 7 and 14. miR-124 inhibitor-induced cell proliferation is blocked by S100A4 siRNA, whereas miR-124-induced cell death is recovered by S100A4. Our findings suggest that miR-124 is a novel regulator of VSMC proliferation and may play a role in the development of neointimal proliferation.</P>
Acharya, S.,Adamová,, D.,Adolfsson, J.,Aggarwal, M.M.,Aglieri Rinella, G.,Agnello, M.,Agrawal, N.,Ahammed, Z.,Ahn, S.U.,Aiola, S.,Akindinov, A.,Al-Turany, M.,Alam, S.N.,Albuquerque, D.S.D.,Aleks North-Holland Pub. Co 2018 Physics letters. Section B Vol.783 No.-
<P><B>Abstract</B></P> <P>The ALICE Collaboration reports the measurement of semi-inclusive distributions of charged-particle jets recoiling from a high-transverse momentum trigger hadron in p–Pb collisions at <SUB> s NN </SUB> = 5.02 TeV. Jets are reconstructed from charged-particle tracks using the anti- <SUB> k T </SUB> algorithm with resolution parameter R = 0.2 and 0.4. A data-driven statistical approach is used to correct the uncorrelated background jet yield. Recoil jet distributions are reported for jet transverse momentum 15 < p T , jet ch < 50 GeV / c and are compared in various intervals of p–Pb event activity, based on charged-particle multiplicity and zero-degree neutral energy in the forward (Pb-going) direction. The semi-inclusive observable is self-normalized and such comparisons do not require the interpretation of p–Pb event activity in terms of collision geometry, in contrast to inclusive jet observables. These measurements provide new constraints on the magnitude of jet quenching in small systems at the LHC. In p–Pb collisions with high event activity, the average medium-induced out-of-cone energy transport for jets with R = 0.4 and 15 < p T , jet ch < 50 GeV / c is measured to be less than 0.4 GeV / c at 90% confidence, which is over an order of magnitude smaller than a similar measurement for central Pb–Pb collisions at <SUB> s NN </SUB> = 2.76 TeV . Comparison is made to theoretical calculations of jet quenching in small systems, and to inclusive jet measurements in p–Pb collisions selected by event activity at the LHC and in d–Au collisions at RHIC.</P>
Choi, S.-K.,Ahn, D.-S.,Lee, Y.-H. Oxford University Press 2009 Cardiovascular research Vol.82 No.2
<P>AIMS: Although stimulation with sphingosylphosphorylcholine (SPC) or sphingosine-1-phosphate (S1P) generally leads to similar vascular responses, the contractile patterns and their underlying signalling mechanisms are often distinct. We investigated the different reliance upon Ca2+-dependent and Ca2+-sensitizing mechanisms of constriction in response to SPC or S1P in coronary arteries. METHODS AND RESULTS: Contractile responses, changes in [Ca2+]i, and phosphorylation of myosin light chain phosphatase-targeting subunit (MYPT1) were measured. SPC induced a concentration-dependent sustained contraction. S1P evoked a rapid rise in force (initial transient), which was followed by a secondary sustained force. In the absence of extracellular Ca2+, the concentration dependency of constriction to SPC was shifted to the right, but with no change in maximum force, whereas S1P-induced contraction was significantly blunted. Cyclopiazonic acid (CPA) significantly decreased the initial transient force induced by S1P. In isolated single cells, S1P markedly increased [Ca2+]i, whereas only a modest elevation was noted with SPC. The S1P-induced elevation of [Ca2+]i was abolished by pre-treatment with CPA and was significantly reduced in the absence of extracellular Ca2+. In beta-escin-permeabilized strips, SPC augmented pCa 6.3-induced force; this was significantly inhibited by fasudil hydrochloride. S1P induced little or no augmentation of pCa 6.3-induced force. In intact arteries, SPC-induced contraction was completely inhibited by fasudil hydrochloride. Fasudil hydrochloride had no effect on the initial transient force induced by S1P but significantly inhibited the secondary sustained force. SPC induced a several-fold increase in Thr696 and Thr853 phosphorylation of MYPT1, but S1P did not affect phosphorylation of MYPT1. CONCLUSION: Our results suggest that constriction of coronary arteries in response to the bioactive lipid S1P or SPC occurs by distinct signalling pathways. Activation of the RhoA/RhoA-associated kinase pathway and subsequent phosphorylation of MYPT1 play a key role in SPC-induced coronary contraction, whereas elevation of [Ca2+]i is crucial for S1P-induced coronary constriction.</P>
Acharya, S.,Adamová,, D.,Adhya, S.P.,Adler, A.,Adolfsson, J.,Aggarwal, M.M.,Aglieri Rinella, G.,Agnello, M.,Agrawal, N.,Ahammed, Z.,Ahmad, S.,Ahn, S.U.,Akindinov, A.,Al-Turany, M.,Alam, S.N.,Alb Elsevier 2019 Physics letters: B Vol.797 No.-
<P><B>Abstract</B></P> <P>An improved value for the lifetime of the (anti-)hypertriton has been obtained using the data sample of Pb–Pb collisions at <SUB> s NN </SUB> = 5.02 TeV collected by the ALICE experiment at the LHC. The (anti-)hypertriton has been reconstructed via its charged two-body mesonic decay channel and the lifetime has been determined from an exponential fit to the d<I>N</I>/d(<I>ct</I>) spectrum. The measured value, τ = 242 − 38 + 34 (stat.) ± 17 (syst.) ps, is compatible with representative theoretical predictions, thus contributing to the solution of the longstanding hypertriton lifetime puzzle.</P>
Agostini, M.,Lim, D.-H.,Sadd, M.,Hwang, J.-Y.,Brutti, S.,Heo, J. W.,Ahn, J. H.,Sun, Y. K.,Matic, A. Wiley (John WileySons) 2018 ChemSusChem Vol.11 No.17
<P>We report a new Li-S cell concept based on an optimized F-free catholyte solution and a high loading nanostructured C/S composite cathode. The Li2S8 present in the electrolyte ensures both buffering against active material dissolution and Li+ conduction. The high S loading is obtained by confining elemental S (approximate to 80%) in the pores of a highly ordered mesopores carbon (CMK3). With this concept we demonstrate stabilization of a high energy density and excellent cycling performance over 500 cycles. This Li-S cell has a specific capacity that reaches over 1000 mA hg(-1), with an overall S loading of 3.6 mg cm(-2) and low electrolyte volume (i.e., 10 mu L cm(-2)), resulting in a practical energy density of 365 Wh kg(-1). The Li-S system proposed thus meets the requirements for large scale energy storage systems and is expected to be environmentally friendly and have lower cost compared with the commercial Li-ion battery thanks to the removal of both Co and F from the overall formulation.</P>
Ahn, S.J.,Song, Y.D.,Mah, S.J.,Cho, E.J.,Kook, J.K. Association for Dental Sciences of the Republic of 2014 Journal of dental sciences Vol.9 No.3
Background/purpose: In prior studies, we induced the antimicrobial activity of deglycyrrhizinated licorice root extract (DG-LRE) by inhibiting the growth and biofilm formation of Streptococcus mutans UA159. Here, we used clinical strains of mutans streptococci (MS) collected from Koreans to determine the optimal concentration of DG-LRE for oral hygiene products to prevent dental caries. Materials and methods: Antimicrobial effects of DG-LRE against 14 clinical strains of MS were evaluated through the minimum inhibitory concentration, minimum bactericidal concentration, time-kill assay, and biofilm-forming assay. Results: Minimum inhibitory concentration and minimum bactericidal concentration values of DG-LRE against the clinical strains of MS ranged from 4 μg/mL to 8 μg/mL and from 8 μg/mL to 16 μg/mL, respectively. Time-kill assay demonstrated that the antimicrobial effects of DG-LRE primarily resulted from bactericidal activity. DG-LRE significantly decreased the biofilm formation of S. mutans ranging from 57.6% to 92.8% at 16 μg/mL. Conclusion: These findings reveal that a DG-LRE concentration of 16 μg/mL may be used to prevent dental caries in Koreans.