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미국 기술유출 관련 법률 및 판례 분석을 통한 우리나라 대응방안 연구- 형사적 쟁점을 중심으로 -
송규영 법조협회 2019 法曹 Vol.68 No.3
미국은 2013년 기술유출을 억제하기 위한 정부 전략을 발표하면서, 자국기업을 상대로 한 위법한 기술유출에 강력하게 대응하여 이를 척결하겠다는 굳은 의지를 밝혔다. 특히 입법·행정·사법을 아우르는 통일적이고 유기적인 시스템을 구축하여 신속하고 효율적으로 대처함으로써 국부유출을 막으려는 시도와 함께 경제스파이법(EEA) 관련 의미 있는 판결들이 속속 등장하면서 기술유출에 대한 관심은 나날이 증가하고 있다. 먼저, 경제스파이법과 영업비밀보호법(DTSA) 및 PRO-IP법을 중심으로 입법적 노력에 관하여 살펴보고, 그에 따른 행정부의 지적재산권 조정관 설치, 연방검찰 및 FBI의 조치와 활동을 간단히 검토한 다음, 경제스파이법이 적용된 사건 중 논란이 되었던 골드만삭스 사건 및 소시에떼 제네랄르 사건의 수사 및 재판 진행 경과 등을 살펴보았다. 다음으로, 「부정경쟁방지 및 영업비밀보호에 관한 법률」 및 「산업기술의 유출방지 및 보호에 관한 법률」개정을 통한 우리나라의 입법적․행정적 대응 노력을 살펴보고, 신속한 피해회복을 위한 사법적 개선방안을 제시하였다. 특히, ‘영업비밀’ 인정기준 완화 및 영업비밀 보호체계 개정에 대한 타당성, 국가적 컨트롤타워 설치와 각 부처 간 협력체계 구축, 피해회사의 내부조사비용을 배상명령에 포함시키는 방안 등에 대해 연구하고, 도입 필요성 등을 검토하였다. 지적재산권 강국으로의 위상을 지키기 위해서는 기술유출 사범에 대한 적극적인 대응이 필요하고, 실무상 여러 쟁점들이 문제될 것으로 예상되는데, 본 연구논문이 도움이 되었으면 한다. The United States has launched "Administration Strategy on Mitigating The Theft of U.S. Trade Secrets" in February 2013. As the Strategy emphasizes, the government will continue to act vigorously to combat the theft of trade secrets that could be used by foreign companies or foreign governments. Appearing meaningful rulings under the Economic Espionage Act, more attention is gathered on protecting the trade secret. In the United States, Economic Espionage Act in 1996 and Prioritizing Resources and Organization for Intellectual Property Act in 2008 were enacted. Under the trade secret laws, the law enforcement agencies have reformed their organizations. Recently, the court made opposite decisions on the Golman Sachs case and the Société Générale case which had similar factual backgrounds. Republic of Korea also has strived to alleviate the theft of trade secret. Unfair Competition Prevention and Trade Secret Protection Act was revised to facilitate proving the trade secret in 2019. And the minister of Trade, Industry and Energy has been in charge of the government committee for protecting trade secret since 2015. However, the revised act broadens the scope of trade secret excessively. And the government control tower for protecting Intellectual Property including trade secrets still remains absent. Additionally, it is necessary to accept the restitution to the victim's expenses incurred during participation in the investigation on the trade secret cases.
UV , TPA , heterologous DNA inserts 가 동물세포에서 homologous recombination 에 미치는 영향
송규영,Kucherlapati, Raju 한국유전학회 1992 Genes & Genomics Vol.14 No.1
We have studied the plasmid x plasmid recombination system to examine factors that might increase the frequency of homologous recombination. Treatment of input DNA with UV doses up to 100 Jm^(-2) did not have a significant effect on recombination, on the other hand, treating the recipient EJ cells with UV light at a dose of 1 Jm^(-2) prior to transfection resulted in a two fold increase in recombination. The tumor promoter TPA treatment did not have an effect on recombination or transformation. We have studied the effect of heterologous inserts on homologous recombination and observed that as the length of insert increased, there was a decline in the frequency of recombination.
Lamin A/C mutations associated with familial and sporadiccases of dilated cardiomyopathy in Koreans
송규영,Marie-Pierre Dub?,임지영,황일선,이인철,김재종 생화학분자생물학회 2007 Experimental and molecular medicine Vol.39 No.1
Dilated cardiomyopathy (DCM) is characterized by cardiac dilation and systo lic dysfunction. So far sixteen genes have been shown to cause autosomal dominant familial dilated cardiomyopathy (FDC). We identified a large Korean family from the Jeju island showing a clear Mendelian inheritance of FDC. A genomewide linkage scan at 9 cM marker density identified a peak multipoint LOD score of 2.82 at D1S195. Haplotyping of the region with 15 additional markers defined a candidate interval that included a known candidate gene en coding the lamin A/C (LMNA). Sequencing of the LMNA exons revealed one missense mutation at C568T (Arg190Trp) in the -helical rod domain of the LMNAgating with FDC with conduction-system disease. The same mutation was found in patients of another Korean family with FDC without conduction-system disease. Upon screening 14 sporadic DCM cases, we found three LMNA mutations including a case having a previously described (Glu161Lys) mutation and two having novel mutations (Glu53Val and Glu186Lys). Our results sugest that variable genotypes of laminopathy ar e implicated in not only familial but also considerable proportion of sporadic DCM.