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Md. Ismail Hossain,Golam M. Mathbor,Renata Semenza 한국사회복지학회 2013 Asian Social Work and Policy Review Vol.7 No.3
Two contested arguments persist in explaining the causes of feminization and labor vulnerability. Some scholars argue that global industries are dominated by female workers as a result of thesearch for cheap labor. On the other hand, some scholars claim that the primary cause of feminizationand labor vulnerability is the gendered discourses of work. Drawing views from readymadegarment (RMG) industries of Bangladesh, this paper argues that both economic choices of cheaplabor and gendered discourses of work collaboratively contribute to feminizing the labor force. This feminization of the workforce enables the violation of labor rights and benefits capitalists.
Karamian Brian,Kothari Parth,Toci Gregory,Lambrechts Mark James,Canseco Jose,Mao Jennifer,Narayan Raj,Alfonsi Samuel,Sirch Francis,Kheir Nadim,Semenza Nicholas,Woods Barrett,Rihn Jeffrey,Kurd Mark,Rad 대한척추외과학회 2023 Asian Spine Journal Vol.17 No.2
Study Design: Single-center retrospective cohort.Purpose: To compare surgical outcomes of patients based on lumbar drain variables relating to output and duration. Overview of Literature: The use of drains following lumbar spine surgery, specifically with respect to hospital readmission, postoperative hematoma, postoperative anemia, and surgical site infections, has been controversial.Methods: Patients aged ≥18 years who underwent lumbar fusion with a postoperative drain between 2017 and 2020 were included and grouped based on hospital readmission status, last 8-hour drain output (<40 mL cutoff), or drain duration (2 days cutoff). Total output of all drains, total output of the primary drain, drain duration in days, drain output per day, last 8-hour output, penultimate 8-hour output, and last 8-hour delta (last 8-hour output subtracted by penultimate 8-hour output) were collected. Continuous and categorical data were compared between groups. Multivariate logistic regression analysis and receiver operating characteristic (ROC) analysis were performed to determine whether drain variables can predict hospital readmission, postoperative blood transfusions, and postoperative anemia. Alpha was 0.05.Results: Our cohort consisted of 1,166 patients with 111 (9.5%) hospital readmissions. Results of regression analysis did not identify any of the drain variables as independent predictors of hospital readmission, postoperative blood transfusion, or postoperative anemia. ROC analysis demonstrated the drain variables to be poor predictors of hospital readmission, with the highest area under curve of 0.524 (drain duration), corresponding to a sensitivity of 61.3% and specificity of 49.9%.Conclusions: Drain output or duration did not affect readmission rates following lumbar spine surgery.
Negative Regulation of Hypoxic Responses via Induced Reptin Methylation
Lee, Jason S.,Kim, Yunho,Kim, Ik Soo,Kim, Bogyou,Choi, Hee June,Lee, Ji Min,Shin, Hi-Jai R.,Kim, Jung Hwa,Kim, Ji-Young,Seo, Sang-Beom,Lee, Ho,Binda, Olivier,Gozani, Or,Semenza, Gregg L.,Kim, Minhyung Elsevier 2010 Molecular cell Vol.39 No.1
<P><B>Summary</B></P><P>Lysine methylation within histones is crucial for transcriptional regulation and thus links chromatin states to biological outcomes. Although recent studies have extended lysine methylation to nonhistone proteins, underlying molecular mechanisms such as the upstream signaling cascade that induces lysine methylation and downstream target genes modulated by this modification have not been elucidated. Here, we show that Reptin, a chromatin-remodeling factor, is methylated at lysine 67 in hypoxic conditions by the methyltransferase G9a. Methylated Reptin binds to the promoters of a subset of hypoxia-responsive genes and negatively regulates transcription of these genes to modulate cellular responses to hypoxia.</P> <P><B>Highlights</B></P><P>► Reptin is a target of G9a methyltransferase ► Reptin K67 methylation is induced by hypoxia ► Genome-wide identification of hypoxia target genes negatively regulated by Reptin ► Hypoxia-driven Reptin methylation negatively regulates tumorigenic behavior in vivo</P>