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남궁미경,NamGoong, Mee-Kyung 대한소아신장학회 2007 Childhood kidney diseases Vol.11 No.1
Many children with microscopic hematuria have been found on school screening examinations. There are not, however, nation-wide criteria for us(specifically, pediatric nephrologists) how to take care of them. Recently, quite a few research papers concerning microalbuminuria with microscopic hematuria, which can predict the renal pathological findings, are published. Here I have reviewed articles on microalbuminuria which gives us the information how to manage microscopic hematuria.
남궁미경,NamGoong, Mee-Kyung 대한소아신장학회 2011 Childhood kidney diseases Vol.15 No.2
급성신부전은 그 단어 의미에 약한 신장 손상을 포함하지 못한다. 또한 급성신부전에 대한 기준이 연구자마다 달라서 연구결과 해석상에 어려움을 주었다. 따라서 급성신부전보다는, 급성신손상이란 용어가, 신손상의 모든 정도를 포함하는 의미로 사용될 수 있다. 2002년에, 급성신손상을 자세히 분류하고 진단하기위해, 혈청크레아티닌, 사구체 여과율과 요량을 기준으로 하여, RIFLE 기준이 제안되었다. 2007년에는, RIFLE 기준을 변형하여, AKIN 기준이 제안되었다. 소아를 위해 소아-RIFLE 기준도 제안되었다. 저자는 여기서 이들 기준과 각각에 대한 비교를 기술하였다. Acute renal failure means that the word does not contain a mild kidney injury. In addition, the criteria for acute renal failure per researcher are different, and it is difficult in interpreting the results of research on acute renal failure. Therefore, rather than acute renal failure, a new term "acute kidney injury" meaning to include all the levels of injury is introduced. In 2002, to diagnose by means of serum creatinine, glomerular filtration rate and urine output, a detailed classification of acute kidney injury, the RIFLE criteria has been proposed. In 2007, the RIFLE criteria by transforming, AKIN criteria has been proposed. The pediatric RIFLE criteria for children has also been proposed. The author reviews here these criteria by comparing them.
( Ki-taek Oh ),( Mee-kyung Namgoong ),( Yeong-myong Yoo ),( Byoung-kook Lee ) 대한주산의학회 2021 Perinatology Vol.32 No.1
Renin-angiotensin system (RAS) has a significant role in the regulation of blood pressure. The use of RAS blocker (angiotensin converting enzyme inhibitor or angiotensin II receptor blocker [ARB]) for hypertension is common, but this medication during pregnancy may lead to dysgenesis of renal tubule, hypoperfusion and hypoplastic growth of skull and pulmonary vascular hypoplasia in fetus. We report a female baby born to a mother who took ARB up to gestational age 35 weeks, and then did not take ARB for 10 days until delivery on IUP 36+3 weeks. The baby suffered from hypotension and anuria for few days but suffered severe respiratory difficulty and pulmonary hypertension requiring mechanical ventilation with inhalation of nitric oxide. The baby had also limb contracture, defects of skull ossification, and thrombocytosis. After weaning of mechanical ventilation, the baby had a polyuria and was diagnosed with transient partial nephrogenic diabetes insipidus. The baby showed polycythemia with normal kidney function at 20 months of age. The baby showed normal growth and development based on the results of the Korean Development Screening Test with 20 months of corrected ages.
Eosinophil Cationic Protein in HSP and in IgA Nephropathy
Kim, Jong Soo,Namgoong, Mee Kyung 대한신장학회 1997 Kidney Research and Clinical Practice Vol.16 No.1
Interleukin-5 (IL-5), which is known to be an activator of human eosinophil, increases in IgA nephropathy. In order to find out the relationship between activated eosinophil function and the pathogenesis of Henoch-Schonlein purpura (HSP) and IgA nephropathy, serum esosinophil cationic protein (ECP) was analyzed using a monoclonal antibody Besides, the soluble IL-2 receptor (sIL-2R) was analyzed to clarify if there was a positive correlation between T cells and activated eosinophils. As anticipated, the levels of ECP, in detail, were significantly higher among HSP patients with a mean of 9.71.8pg/L than in a control group with a mean of 4.6?0.7pg/L. The HSP patients were also classified as one group with normal urine and another group with abnormal urine. The latter showed higher levels of ECP than the former. On the other hand, the levels of ECP were higher in IgA nephropathy patients than in the control group,: however, there was no significance in statistics. The sIL-2R levels were higher in HSP patients tan those in serums of IgA nephropathy patients and the control group. Thus, this study came to a conclusion that the activated eosinophil might be one of the pathogeneses in HSP but not in IgA nephropathy.
천명을 동반한 영유아에서 말초혈액 T 림프구아형에 관한 연구
최진영(Jin Young Choi),남궁미경(Mee Kyung Namgoong),최원규(Won Kyu Choi),김종수(Jong Soo Kim) 대한소아알레르기호흡기학회(구 대한소아알레르기 및 호흡기학회) 1993 소아알레르기 및 호흡기학회지 Vol.3 No.2
N/A There is some evidence in bronchial asthma that the number and the function of T4 (+) cells and of T8 (+) cells decrease but that T8 (+) cells decrease less than T4 (+) cells. These decreases represent one of the pathogenesis of excessive production of IgE and of bronchial asthma. We studied whether or not the subtypes of T cells also change in wheezing infants and toddlers. The results were as follows: 1. Compared to those of control patients, the percentages of T4 (+) cell and T8 (+) cell were high and the ratio of T4/T8 was low in w patients but there was significance in the percentage of T8 (+) cell (21.3% versus 31.0%, p<0.05). 2. Compared to those of acute period the percentages of T4 (+) cell and T8 (+) cell and the ratio of T4/T8 were significantly decreased in convalescent period. 3. There was a significant correlation between the percentage of T8 (+) cell and the levels of IgE in Wheezy patients. 4. There was a significant negative correlation between the T4 (+) cell (%) and age (month) in wheezy patients.
폐 동정맥 기형과 비장 동정맥 기형을 동반한 유전 출혈 모세혈관 확장증 1례
김미현 ( Mi Hyun Kim ),남궁미경 ( Mee Kyung Namgoong ),김영주 ( Young Ju Kim ),정순희 ( Soon Hee Jung ),어영 ( Young Uh ),이해용 ( Hae Yong Lee ) 대한소아알레르기호흡기학회(구 대한소아알레르기 및 호흡기학회) 2012 소아알레르기 및 호흡기학회지 Vol.22 No.4
유전 출혈 모세혈관 확장증은 혈관 생성 유전자의 이상에 의해 나타나는 상염색체 우성 질환으로, 국내에서는 산발적으로 보고되고 있다. 저자들은 평소 비출혈이 있으며 우연히 발견된 폐 결절이 있는 환아에서 영상 소견과 유전자 검사로 폐 및 비장 동정맥 기형을 동반한 유전 출혈 모세혈관 확장증을 진단하고 치료하였기에 보고하는 바이다. Hereditary hemorrhagic telangiectasia (HHT), also known as Rendu-Osler-Weber disease, is a rare autosomal dominant vascular disorder involving arteriovenous malformation (AVM) due to angiodysplasia. HHT is characterized by mucocutaneous hemorrhagic telangiectasia and AVM of various different organs. The mutated genes in HHT encode proteins that mediate signaling by the transforming growth factor-β superfamily, leading to migration and proliferation of endothelial cells to finally result in AVMs of various organs. Amongst various affected organs, pulmonary AVM is the most common. This 11-year-old female patient with a past and familial history of frequent epistaxis visited the hospital for abdominal pain and vomiting which developed on the same day. Nodular opacity was found in chest X-ray. Subsequently, AVMs in the lungs and the spleen were recognized in chest computed tomography along with detection of ENG gene mutation. Here we report a case of hereditary hemorrhagic telangiectasia diagnosed by incidental chest X-ray findings followed by genetic and radiologic studies. [Pediatr Allergy Respir Dis(Korea) 2012;22:438-443 ]
신생검에서 급성 사구체신염 양상을 보인 Henoch-$Sch{\ddot{o}}nlein$ 자반증 2례
이상흔,남궁미경,차병호,김종수,Lee Sang Heun,Namgoong Mee Kyung,Cha Byung Ho,Kim Jong Soo 대한소아신장학회 1998 Childhood kidney diseases Vol.2 No.2
[ $\beta$ ]-streptococci have been reported as an etiology of Henoch-$Sch{\ddot{o}}nlein$ purpura(HSP), but the role of $\beta$-streptococci in HSP is not yet confirmed. Our report of two HSP patients is as follows : Although the two patients' clinical symptoms resembled those symptoms of HSP, whereas serum $C_3$ decreased in one case, ASO titer increased in both cases and a histological examination of renal biopsy specimens revealed the fetures commonly described in poststreptococcal glomerulonephritis.