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정숙금(Seuk Kum Chung),설미영(Mee Young Sol),이선경(Sun Kyung Lee) 대한소화기학회 1996 대한소화기학회지 Vol.28 No.5
N/A Background/Aims: Although the incidence of intrahepatic biliary stone in Korea is relatively high, the pathogenetic role of intrahepatic biliary ]esion on the genesis of intrahepatic stone is not clearly understood yet. This study was perfomed to investigate the patbological changes of intrahepatic bile ducts in hepatolithiasis and compare with other reports, thus get some information about the pathophysiologic role of those lesions. Methods: Patho]ogical and histochemical studies were done on 43 cases of liver resected for the treatment of intrahepatic biliary stones during last 16 years. Results: The highest incidence was at the 6th decade. Male to female ratio was 1:1.4 (18cases to 25 cases) and mean age was 45.4 years. More than half of the cases complained less than 2 months of biliary symptom duration. Majority of the cases(38 cases, 88.3%) revealed stones in the left intrahepatic bile ducts. Histological changes of the intrahepatic bile ducts harboring biliary stones showed superficial epithelial hyperplasia in 20 cases(46.5%), proliferation of intramural glands in 36 (83.7%), extramural glandular hyperplasia in 42(97.7%), pseudopyloric glands in 3(7.0%)and pancreatic acinus in 3 cases(7.0%), and chronic inflammatory reaction in all the cases examined. Mucin in the pseudopyloric glands were mixture of class m mucin and sialo- and sulphomucin. Conclusions: It is concluded that histologic lesions associated with the intrahepatic biliary stones were chronic inflammatory reaction and proliferation of intrahepatic ductal epithelium and peribiliary glands secondary to the inflammation. Hypersecreted glandular secretion of peribiliary glands can be an augumenting factor of the increment of the biliary stones. (Korean J Gastroenterol 1996; 28: 697 - 704)
양승학(SH Yang),정숙금(SG Jeong),허만하(MH Huh),박은동(ED Park) 대한산부인과학회 1993 Obstetrics & Gynecology Science Vol.36 No.6
Retroperitoneal cysts are rare intra-abdominal tumors. They can appear as chronic abdominal pain, a painless abdominal mass, or acute abdomen. It is difficult to differentiate from ovarian tumor by pelvic examination. Diagnostic aids include pelvic computed tomography and ultrasound. We present a case of retroperitoneal lymphatic cyst required differential diagnosis.
이식신에 발생한 Polyoma Virus 간질성 신염 1예
박민,김미선,윤대현,박용기,정현주,허동,이시래,김중경,장익득,신용훈,정숙금 대한신장학회 1999 Kidney Research and Clinical Practice Vol.18 No.6
We report one case of renal PV infection after renal allograft transplantation leading to graft dysfunction. According to prior reports, PV induced interstitial nephritis might be a cause of graft loss. Pathologic findings show varying degrees of interstitial infiltration and tubular degenerative changes, which resemble acute cellular rejection. Therapeutic strategies have not yet been developed. Case ; A 23 years old male underwent renal transplantation from his HLA haploidentical 25 year old sister. His renal function had been good with cyclosporin, steroid and azathioprine until 9 months after transplantation, when his serum creatinine level rose to 2.2mg/dl. The renal biopsy revealed diffuse lymphocyte infiltration in the interstitium and feature of the tubulitis. Also, giant tubular epithelial cells with large, hyperchromic nuclei were present. Despite steroid pulsing and OKT3, renal function progressively deteriorated. After 10 days of OKT3 therapy, the patient suffered from high fever, dyspnea and general aches. A chest X-ray revealed interstitial infiltration in both lung fields and the cytomegalovirus PCR (polymerase chain reaction) test of serum and blood was positive. Intravenous ganciclorvir was administered and immunosuppressants were tapered. 4 months after admission, he lost his graft function and underwent hemodialysis. The aforementioned renal biopsy was retested immunohistochemically. Nuclear inclusions in renal tubular epithelial cells were shown and these inclusions were reacted positively with PV monoclonal antibodies.