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박상석,박태선,송현모,백홍선,이대열 대한내분비학회 1996 Endocrinology and metabolism Vol.11 No.1
Backgrounds: Thyroid hormones play a fundamental role in the initiation and maintenance of somatic growth in mammalian species, and the insulin-like growth factors(IGFs) occupy a position of central importance in the growth of all tissues. To evaluate the changes in serum insulin-like growth factor-I(IGF-I) and insulin-like growth factor binding proteins in hyperthyroid and hypothyroid patients, sera was obtained from 19 hyperthyroid patients, 9 hypothyroid patients, and 10 healthy volunteers. Methods: IGF-I concentration was determined by radioimmunoassay, and changes in IGFBPs were assesed by Western Ligand Blotting. To evaluate the binding pattern of IGF-I & IGFBPs, autoradiographs were obtained. Results & Conclusion: IGF-I levels were increased significantly in hyperthyroid patients(mean ± SE, 267.88±19.80 ng/ml, p$lt;0.05) and decreased significantly in hypothyroid patients(154.81±11.43 ng/ml, p $lt;0.01) compaired to healthy control group(209.45±8.60 ng/ml). Autoradiograph of serum IGFBPs from patients wiyperthyroidism and hypothyroidism did not show any change in the intensity of IGFBP-3 bands(40-45 KD) and IGFBP-1 bands, but in hyperthyroid patients, it showed increased intensity of IGFBP-2 band compared to healthy control group and hypothyroid patients(1 Kor Soc Endocrinol 11:52-60, 1996).
T 형 만성 림프구성 백혈병에 동반된 순수 적혈구 형성부전증 1 예
박상석(Sang Seuk Park),이홍(Hong Lee),장재호(Jae Ho Jang),송현모(Hyun Mo Song),이성중(Seung Jung Lee),곽재용(Jae Yong Kwak),임창열(Chang Yeol Yim),최병문(Byung Moon Choi),조용곤(Yong Gon Cho),이미경(Mi Kyung Lee),김윤정(Yeun Jeong K 대한내과학회 1997 대한내과학회지 Vol.52 No.2
Pure red cell aplasia(PRCA) is characterized by anemia, absence of reticulocytes in the peripherial blood and selective erythroid hypoplasia in the bone marrow. Acquired PRCA is often associated with thymoma but may also occur in many diverse conditions, such as chromic lymphocytic leukemia, systemic lupus erythematosus, autoimmune disorders, T gamma lymphocytosis, acquired hypogammaglobulinemia, acquired immunodeficiency syndrome. Recently we experienced a case of T cell chronic lymphocytic leukemia associated with pure red cell aplasia. A 65-year-old man was presented with severe anemia. absolute reticulocytopenia, hepatosplenomegaly and lymphocytosis(T cell marker' CD2, CD7 positive). Bone marrow findings showed a marked decrease in erythroid precursors and normal maturations of granulocytic and megakaryocytic series, which were consistent with pure red cell aplasia. So we report this case with a review of the literature.
심한 저알부민증과 혈소판감소증을 동반한 특발성 다발성근염 1 예
박상석(Sang Seuk Park),류완희(Wan Hee Yoo),문성기,김재헌(Jae Hean Kim),송현모(Hyun Mo Song),곽재용,임창열(Chang Yeol Yim) 대한내과학회 1997 대한내과학회지 Vol.52 No.1
Primary idiopathic polymyositis is a condition of presumed autoimmune etiology in which the skeletal muscle is damaged by a nonsuppurative inflammatory process dominated by lymphocytic infiltration. We recently experienced a patient with primary idiopathic Polymyositis associated with hypoalbuminemia and thrombocytopenia. About 4 months prior to admission, he was diagnosed as primary idiopathic polymyositis, and improved by treatment with prednisolone 60 mg/day. During steroid tapering, muscle weakness was recurred and accompanied by hypoalbuminemia and thrombocytopenia. Despite retreatment with prednisolone 60 mg/day, muscle weakness, hypoalbuminemia and thrombocytopenia persisted. He was then started to administer methotrexate(MTX) 15 mg/week which resulted in prompt improvement of muscle weakness, hypoalbuminemia and thrombocytopenia. These findnigs suggest that primary idiopathic polymyositis is one of the cause of hypoalbuminemia and thrombocytopenia, and that the hypoalbuminemia and thrombocytopenia can be improved promptly by methotrexate treatment.
만성 폐쇄성 폐질환 , 기관지천식 환자에서의 운동부하 폐기능검사
이용철(Yong Chul Lee),김원(Won Kim),임석태(Seok Tae Lim),이흥범(Heung Bum Lee),김현중(Hyun Jung Kim),이제경(Je Kyung Lee),송현모(Hyun Mo Song),이양근(Yang Keun Rhee) 대한내과학회 1996 대한내과학회지 Vol.50 No.6
Objectives: Exercise tests are being widely applied to objective measurement of work capacity, identification and quantification of cardiopulmonary mechanics in the cardiac and pulmonary disorders. To evaluate patients with exertional dyspnea in bronchial asthma and chronic obstructive pulmonary disease (COPD), clinicians need to know the range of expected performance during exercise. To define the various response to exercise, we studied exercise tests in clinically stable asthma and chronic obstructive pulmonary disease. Methods: Twenty healthy subjects, twenty controlled bronchial asthma patients, and twenty five chronic airway obstructive patients underwent progressively incremental exercise test to the symptom-limited maximum. Measurement were made of O₂uptake and CO₂ ouptput, heart rate, breathing rate, anaerobic threshold, minute ventilation, maximal O₂pulse and dead space during exercise test. Results: 1) The maximal oxygen uptake, maximal CO₂production, maximal oxygen pulse, minute ventilation, and breathing reserve in COPD patients were significantly lower than that of normal subjects. 2) The heart rate reserve and dead space at maximal exercise in COPD patients were significantly highter than those of normal controls. 3) The anaerobic threshold was detectable in 8 of 25 COPD patients. The ratio of anaerobic threshold to maximal oxygen uptake was not different from that of nomal subjects. 4) The maximal oxygen uptake, maximal Co₂output, maximal oxygen pulse, minute ventilation, breathing reserve, heart rate reserve, dead space at maximal exercise, anaerobic thre shold and ratio of anaerobic threshold in controlled bronchial asthmatics were not different form those of normal subjects, Conclusion: The exercise capacity is decreased in chronic obstructive pulmonary disease. But, there is no difference between nomal controls and controlled asthmatic patients in exercise capacity. This result suggest that exercise capacity of controlled asthmatic patients is not decreased.