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이만영(Man Young Lee),승기배(Ki Bae Seung),김종진(Jong Jim Kim),노태호(Tae Ho Rho),채장성(Jang Seong Chae),김종상(Jong Sang Kim),홍순조(Soon Jo Hong),최규보(Kyu Bo Chol) 대한내과학회 1990 대한내과학회지 Vol.39 No.6
N/A To evaluate the clinical differences between Q wave myocardial infarction and non-Q wave myocardial infarction, the records of 336 patients with first myocardial infarction were reviewed. According to the presence or absence of Q waves on electrocardiogram, the patients were divided into two groups: a Q wave myocardial infarction group and a non-Q wave myocardial infarction group. The results were as follows: 1) According to standard electrocardiographic criteria, among 336 patients 271 patients (80.6%) had Q wave myocardial infarctions, and 65 patients (19.4%) had non-Q wave myocaridal infarctions. 2) The average age and male-to-female ratio were similar in the two groups. There were no significant differencres between the two groups in serum cholesterol levels and in incidences of a history of hypertension and diabetes mellitus. 3) Peak cardiac enzyme levels of CPK and LDH were significantly higher in the Q wave myocardial infarction group than in the non-Q-wave myocardial infarctions group. 4) When the complications of arrythmia, congestive heart failure and hospital mortality were compaired, incidences of AV block and congestive heart failure were significantly higher in the Q wave myocardial infarction group, but there was no difference in hospital mortality between the two groups. 5) Incidences of recurrent angina, congestive heart failure, reinfarction, death, and cause of death were not different statistically between the two groups, even though there was a tendency to have more recurrent angina and reinfarction in the non-Q wave myocardial infarction group and more congestive heart failure in the Q wave myocardial infarction group.
박지원(Ji Won Park),이인석(In Seok Lee),이현승(Hyun Seung Lee),유기동(Ki Dong Yoo),전은정(Eun Jung Jun),박인수(In Soo Park),정욱성(Wook Sung Chung),노태호(Tae Ho Rho),이만영(Man Young Lee),채장성(Jang Seong Chae),김재형(Jae Hyung Kim 대한내과학회 1999 대한내과학회지 Vol.56 No.1
Swallowing syncope is a rare syndrome of a sudden and temporary loss of consciousness on swallowing. 66- year-old man was admitted due to dysphagia and syncope. He had no history of cardiac or gastrointestinal problem. In manometry, there was increased pressure of lower esophageal sphincter consistent to secondary achalasia and 24-hour Holter monitoring showed sinus arrest and atrioventricular block while swallowing. Head-up tilt test and provocation test with ballooning tube were shown mixed pattern of cardioinhibitory and vasodepressor response. Propranolol was given but not effective. Permanent pacemaker was implanted and there was no more syncopal episode. For evaluation of dysphagia symptom gastrofiberscopy was done and we found stomach carcinoma at the gastric cardia portion that infiltrated to the lower end of esophagus. Partial esophagogastrectomy with anastomosis was done and then the cardiac arrhythmias were disappeared. In the microscopic finding of gastroesophageal portion, it revealed tumor cells infiltrated to vagus nerve located in esophageal submucosa. We propose that swallowing syncope is induced mainly by esophageal abnormality with or without cardiac abnormality and stomach carcinoma metastasized to esophagus is an etiology of swallowing syncope. We report a case of swallowing syncope due to metastatic esophageal carcinoma involving vagal nerve.