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안수미,윤기영,신연명,최경현,임근승,장문경,송윤미,공은희 고신대학교의과대학 2008 고신대학교 의과대학 학술지 Vol.23 No.2
Background : About 30% of patients in the hospital are under nourishment. A large portion of people are undernourished when they are admitted to the hospital and in the majority of these, undernutrition develops further while in hospital. Patient-Generated subjective global assessment(PG-SGA), a method of nutritional assessment based on clinical judgment, has been widely used to assess the nutritional status of adults for both clinical and research purposes. Modified PG-SGA can be used as a nutrition screening tool, and a comprehensive nutrition assessment is desirable for those who are malnourished. The purpose of this study is to develop a simple, reliable and valid nutrition screening program(NSP). Methods : We applied the nutrition screening program(NSP) to adult inpatients who are at risk of malnutrition using the Modified Patient-Generated subjective global assessment(PG-SGA) and objective assessment(electrical medical record data). Results : Therefore simple and beneficial NSP is developed for patients with severe malnutrition. Using NSP. nutritional information of the severe malnutrition patient should be shared with the physicians and they should be taken care of by clinical dietitians to improve their nutritional status. Conclusion : The Modified PG-SGA and nutrition screening tool are simple, reliable and valid nutrition screening program(NSP).
농촌인구에서의 대사증후군의 특징과 비만 관련 인자와의 연관성
임수 ( Im Su ),권경훈 ( Kwon Gyeong Hun ),김은주 ( Kim Eun Ju ),임덕상 ( Im Deog Sang ),임효진 ( Im Hyo Jin ),조성일 ( Jo Seong Il ),이윤용 ( Lee Yun Yong ),박경수 ( Park Gyeong Su ),이홍규 ( Lee Hong Gyu ) 한국지질동맥경화학회(구 한국지질학회) 2002 韓國脂質學會誌 Vol.12 No.4
Objective: Metabolic syndrome is a loosely defined state characterized by insulin resistance, which includes phenotypes of glucose intolerance, abdominal obesity, dyslipidemia and hypertension. However, there is no internationally agreed definition for th
탈진운동 후 (-)-Hydroxycitrate 섭취가 저장글리코겐회복능에 미치는 영향
이수천 ( Lee Su Cheon ),임창수 ( Im Chang Su ),장응찬 ( Jang Eung Chan ),임기원 ( Im Gi Won ),윤신중 ( Yun Sin Jung ),류승필 ( Lyu Seung Pil ) 한국운동영양학회 2003 Physical Activity and Nutrition (Phys Act Nutr) Vol.7 No.2
Hydroxycitric acid (HCA) is not interfere fat oxidation during moderate intensity exercise and reduce fat accumulation in the body. Depleted glycogen by exhausted exercise caused fatigue that reduced exercise performance in the continuos day or match. lf considering the effect of HCA, even so, ingestion after exhaustive exercise. Therefore, 500 mg of HCA was intubated to the Sprague-Dawley male rats that have exercise trained for 3-week and exercise on the treadmill until exhaustive. Liver and muscle (soleus, gastrocnemius and quadriceps) glycogen and blood glucose, lactate, and FFA were analyzed. Glycogen contents were reduced after exhaustive exercise then increased 24-h of recovery periods without HCA effect. Blood profiles were not affected by HCA either. These result suggested that HCA effect was showed only during exercise in rats.
대기오염과 천식증상에 의한 응급실내원과의 연관성에 관한 환자교차연구
임형준,이상윤,윤기정,주영수,강대희,조수현 대한산업의학회 2000 대한직업환경의학회지 Vol.12 No.2
Objectives : This study, using case-crossover design, search a relationship between changing levels of air pollutants and asthma attack in asthma patients. Methods : We searched the daily total number of asthmatic patients at hospital's emergency room in Seoul, Korea by investigating total 49 general hospital's emergency room records. Otherwise, We investigated medical records of asthmatic patients at one of the 49 hospitals we have searched. We investigated addresses that they are living, diagnosis, smoking history, whether respiratory infection or not, medications. We analyzed the data by 1:m conditional logistic regression used when matched case-control study is analyzed. Control periods were chosen by bidirectional paired matching technique 7, 14 days before and after case periods. Results : the relative risk of asthmatic attack by 100 ppm ozone increase between June and September were 1.348(95 % CI = 1.010∼1.619). Between November and February, the relative risk by 100 ㎍/㎥ TSP increase, 100ppb sulfur dioxide and nitrogen dioxide were respectively 1,278(1.010∼1.619), 1.237(1.019∼1.502), 1.285(1.006∼1.640). In stratified analysis, the relative risk were significant when the asthmatic patients were children aged 15 years or below, when there were respiratory infection, and when the patients took medications irregularly Conclusions : There was statistically significant association between ambient levels of air pollutants and asthmatic attack. So, we have to exert our efforts to minimize the air pollution effectively, to protect public health from air pollution.
Im, Jintaek,Jeon, Jun Ho,Cho, Min Kyung,Woo, Sang Su,Kang, Seok-Seong,Yun, Cheol-Heui,Lee, Kangseok,Chung, Dae Kyun,Han, Seung Hyun Elsevier 2009 Molecular immunology Vol.47 No.2
<P><B>Abstract</B></P><P>We investigated the mechanism for the induction of a chemokine, IL-8, by bacterial flagellins in the human alveolar type II epithelial cell line, A549. Bacterial flagellin induced expression of IL-8 mRNA and protein in dose- and time-dependent manners. IL-8 expression was inhibited by nystatin (a lipid rafts inhibitor) but not by chlorpromazine (a clathrin-coated pits inhibitor). Interestingly, Toll-like receptor 5 (TLR5) recognizing flagellins was found in the intracellular compartment of A549 but rarely on the cell surface. Flagellin-induced IL-8 expression appears to be mediated through TLR5 as determined by <I>in vitro</I> transient transfection experiment in HEK-293 cells expressing TLR5 using a reporter gene construct containing IL-8 promoter. IL-8 expression was attenuated by inhibitors for protein kinase C (PKC) and mitogen-activated protein (MAP) kinases. Furthermore, NF-κB and NF-IL6 transcription factors played an important role in the flagellin-induced IL-8 gene expression in A549 cells. Collectively, these results suggest that flagellin-induced IL-8 expression requires formation of lipid rafts, intracellular TLR activation, and subsequent activation of PKC and MAP kinases leading to the activation of the transcription factors NF-κB and NF-IL6 in human alveolar type II epithelial cells.</P>