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석탄의 발열량 범주별 CO2 배출량 분석을 통한 석탄발전의 온실가스 감축 방안 연구
정현록 한국기후변화학회 2022 한국기후변화학회지 Vol.13 No.4
In this paper, we sought to reduce greenhouse gas emissions from coal power plants by analyzing CO2 emissions by coal calorific value. To this end, the analysis data of coal introduced to three coal power plants in Korea from 2014 to 2021 was used, and the CO2 emissions for high-calorific coal, medium-calorific coal, and low-calorific coal were analyzed. The CO2 emission factors were 95,264 kgCO2/TJ for high-calorific coal, 97,096 kgCO2/TJ for medium-calorific coal, and 100,027 kgCO2/TJ for low-calorific coal. CO2 emission intensity was 891.33 kgCO2/MWh for high-calorific coal, 908.47 kgCO2/MWh for medium-calorific coal, and 935.89 kgCO2/MWh for low-calorific coal. Therefore, CO2 emissions can be reduced by increasing the proportion of high-calorific coal compared to low-calorific coal in coal power generation. However, some issues need to be addressed before expanding the proportion of high-calorific coal. First, as particulate matter emissions are higher for high-calorific coal, the performance of air pollution prevention facilities must be improved. Second, because the price of high-calorific coal is higher than that of low-calorific coal, policy changes regarding the individual consumption tax imposed on coal are needed to improve the price competitiveness of high-calorific coal.
Genuine traditional Korean medicine, BaekJeol-Tang for the treatment of rheumatoid arthritis
한나라,심우문,설무창,김민철,이창희,김동원,이세훈,이호철,류중민,남봉수,김종옥,문성오,장현록,김영석,이인,양진영,황규선,천창선,정현석 셀메드 세포교정의약학회 2013 셀메드 (CellMed) Vol.3 No.2
Inflammation in rheumatoid arthritis is characterized by immune cell infiltration and cytokine secretion. In particular, mast cells and their cytokines play an important role in the pathogenesis of rheumatoid arthritis. Korean medicine, BaekJeol-Tang (BT) was designed by traditional Korean medicine theory. We already reported therapeutic effect of BT in rheumatoid arthritis. Here, we report the specific underlying mechanism of BT in activated human mast cells, HMC-1 cells. In addition, we report for the first time that BT significantly inhibited the production and mRNA expression of proinflammatory cytokines including thymic stromal lymphopoietin, interleukin (IL)-1β, IL-6, IL-8, and tumor necrosis factor-α in activated HMC-1 cells. BT also decreased the activation of mitogen-activated protein kinases, nuclear factor-κB, and caspapase-1. Taken together, these results indicate that BT has potential as a regulator of inflammatory reactions for the treatment of arthritis such as osteoarthritis and rheumatoid arthritis.