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MIVB 용접용 개폐형 자속발생기에 의한 자원밀도분포의 수치해석
구진모,김재웅,Ku Jin-Mo,Kim Jae-Woong 대한용접접합학회 2004 대한용접·접합학회지 Vol.22 No.6
MlAB(magnetically impelled arc butt) welding is a sort of pressure welding method by melting two pipe sections with high speed rotating arc and upsetting two pipes in the axial direction. The electro-magnetic force, the driving force of the arc rotation, is generated by interaction of arc current and magnetic field induced from the magnetic flux generator in the welding system. In this study, an openable coil system for the generation of magnetic flux and a 3-dimensional numerical model for analyzing the electro-magnetic field were proposed. Through the fundamental numerical analyses, a magnetic concentrator was adopted for smoothing the magnetic flux density distribution in the circumferential direction. And then a series of numerical analysis were performed for investigating the effect of system parameters on the magnetic flux density distribution in the interested welding area.. Numerical quantitative analyses showed that magnetic flux density distribution generated from the proposed coil system is mainly dependent on the exciting current in the coil and the position of coil or concentrator from the pipe outer surface. And the gap between pipe ends and arc current are also considered as important factors on arc rotating behavior.
광대역 이동 액세스 시스템에서의 실시간 및 비실시간 통합 서비스 지원을 위한 적응적 임계값 기반 패킷 스케줄링 기법
구진모,김성경,김태완,김재훈,강충구,Ku, Jin-Mo,Kim, Sung-Kyung,Kim, Tae-Wan,Kim, Jae-Hoon,Kang, Chung-G. 한국통신학회 2007 韓國通信學會論文誌 Vol.32 No.3A
실시간과 비실시간 서비스 클래스의 각 QoS요구 사항을 만족하면서 전체 시스템 용량을 최대화하기 위하여 DTPQ스케줄링 알고리즘이 제안된 바 있다. 기존 DTPQ방식은 개별 서비스 클래스의 QoS를 만족하면서 전체 시스템의 평균 수율을 극대화하기 위해 실시간 서비스의 HOL 패킷의 지연 시간이 정해진 임계값을 넘는 경우에만 서비스하는 접근으로서, 이를 구현하기 위해서는 주어진 환경에서 적절한 지연 임계값을 설정할 수 있어야 한다. 본 논문에서는 실시간 사용자의 채널 상태와 지연 시간에 따라 지연 임계값을 적응적으로 적용하는 A-DTPQ 기반의 스케줄링 기법을 제안한다. 이는 기존의 DTPQ알고리즘과 달리 트래픽 부하 및 채널 특성 등에 따라 지연 임계값을 적응적으로 결정하는 방식이다. 시스템 레벨 시뮬레이션을 통해 제안 방식의 고정된 최적 임계값을 가지는 DTPQ방식보다 성능이 향상시킬 수 있음을 확인하였으며, 향후 연구에서는 다양한 서비스 클래스에 대해 이와 같은 개념을 일반화할 수 있을 것이다. The Delay Threshold-based Priority Queueing (DTPQ) scheme has been shown useful for scheduling both real-time (RT) and non-real-time (NRT) service traffic in mobile broadband wireless access (MBWA) systems. The overall system capacity can be maximized subject to their QoS requirement by the DTPQ scheme, which takes the urgency of the RT service into account only when their head-of-line (HOL) packet delays exceed a given delay threshold. In practice, the optimum delay threshold must be configured under the varying service scenarios and a corresponding traffic load, e.g., the number of RT and NRTusers in the system. In this paper, we propose an adaptive version of DTPQ scheme, which updates the delay threshold by taking the urgency and channel conditions of RT service users into account. By evaluating the proposed approach in an orthogonal frequency division multiple access/time division duplex (OFDM/TDD)-based broadband mobile access system, it has been found that our adaptive scheme significantly improves the system capacity as compared to the existing DTPQ scheme with a fixed delay threshold.
TRIP-16 화합물의 베타세포 당지질독성 해독 기전 분석
박선미(Sun mi Park),정대연(Dae youn Jeong),구진모(Jin mo Ku),황윤정(Yoon Jung Hwang),최성이(Sung E Choi),강엽(Yup Kang),정귀완(Kwi wan Jeong) 대한약학회 2016 약학회지 Vol.60 No.6
Gradual deterioration of pancreatic beta cells is a conundrum still to be resolved in the diabetes field. The toxicity manifested by high level of glucose and saturated fatty acid (SFA), which is collectively termed as glucolipotoxicity, has been accepted as a predominant factor of beta cell dysfunction or failure. In this study, we examined the effect of TRIP (Triazolo[ 3,4-a]phthalazine)-16, a compound derivatized from TRIP which had been identified in a high throughput screening to discover chemicals protecting INS-1 cells from glucolipotoxicity. We also investigated its mode of action to reduce glucolipotoxicity, especially regarding modulation of lipid metabolism. From a series of cell-based experiments, we observed that TRIP-16 reduced fatty acid-induced triglyceride (TG) accumulation, whereas it increased oxidation rate of glucose or palmitate. The compound also reduced reactive oxygen species generated by palmitate and upregulated expression levels of carnitine palmitoyltransferase-1a (CPT-1a), peroxisome proliferator-activated receptor gamma coactivator 1-alpha (PGC- 1α) and uncoupling protein 2 (UCP-2), implicating that it can facilitate mitochondrial energy metabolism to oxidize palmitate. In terms of beta cell functioning, TRIP-16 augmented glucose-stimulated insulin secretion (GSIS) per se. Taken together, our data strongly suggest that TRIP-16 can be a potential drug candidate for prevention of type 2 diabetes through beta cell protection.