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안면부 종창을 동반한 Henoch-Scho¨nlein 자반증 1례
이재휘,서무규,임문환,박성기,김태우,이창연 동국대학교 경주대학 1991 東國論集 Vol.10 No.-
We experienced a case of Henoch-Scho‥nlein purpura with are manifestation of facial swelling in a 7-year-old boy.
비만 여성을 대상으로 한 해조류 추출물의 국부처치와 운동 병행을 통한 지방분해 효과의 임상적 검정
서태수,이인숙,변재철,김규호,박승한,김유영,김상국,김현정,김상기,신동철,성미영,박종석,김종기,박언휘 한국스포츠리서치 2003 한국 스포츠 리서치 Vol.14 No.3
This study was carried out to test the body fat breakdown during topical seaweed extract treatment and in combination with the aerobic exercise. Of 20 female obesity subjects, ten treated a cream types of seaweed extract(control group) and ten treated a placebo cream(placebo group). Topical treatment of each sample was designed for 4 weeks, and treated with 2 times a day about 10ml on the abdomen and the thigh, respectively. All subjects were participated aerobic dance half hour a day and four times a week. After 4 weeks of topical treatment and exercise, they were measured body fat, skinfold thickness, body circumference. Then venous blood samples were taken and analyzed blood cells, serum enzymes, and serum lipids. There were reduction of body weight, % of body fat, antebrachium, brachium, chest, waist, thigh circumference and abdomen thickness between pre and post treatment in both group. Especially, body weight and % of body fat significantly reduced in the subjects treating the seaweed extract than those treating the placebo. There were significant decrement in total cholesterol and LDL whereas increment in HDL and TBIL in the control group, but no significant difference in the placebo group. In conclusion, there were 7.6% of body fat reduction during 4 weeks of aerobic exercise and 21% of body fat reducement in combination of topical seaweed treatment and exercise, respectively. These results suggest that the topical treatment of seaweed extract should be very effective for fat breakdown. Especially, the body fat breakdown maximized in the combination with the topical seaweed extracts treatment and the aerobic exercise.
GPS-X 시뮬레이션을 이용한 2단탈질 공정에서 외부탄소원 적용성 평가
정창화,심유섭,김태형,박철휘 대한상하수도학회 2004 상하수도학회지 Vol.18 No.1
The purpose of this study was to evaluate adaptability of external carbon source using GPS-X program in pilot plant composed with 2-stage denitrification process. The result from analysis of pilot plant operation and GPS-X simulation showed that effluent concentration could be simulated similarly by modifying operation conditions, such as DO concentration, C/N ratio and other calibrated parameter. In order to satisfy the standard of the effluent water quality on T-N of 20㎎/L, it required approximately 3.1 of C/N ratio and 50% of nitrogen removal efficiency when influent T-N is 36.9㎎/L. To maintain the stable water quality of the receiving water, the effluent T-N concentration should be less than 10-15㎎/L and the appropriate C/N ratio to remove nitrogen was 4.27-6.82. The analysis of sensitivity to kinetic coefficient and reaction constant showed that Y_(H) and μ_(mAUT) were most sensitive to nitrate and ammonia nitrogen, relatively and sensitivity coefficient of their were 1.32, 1.98. It was concluded that as Y_(H) decreased and μ_(mAUT) increased, the reaction rates of denitrification and nitrification increased and the removal efficiencies of NO₃^(-)-N and NH₄^(+)-N improved.
Park, Seung Min,Park, Chan Woo,Lee, Tae-Kyeong,Cho, Jeong Hwi,Park, Joon Ha,Lee, Jae-Chul,Chen, Bai Hui,Shin, Bich-Na,Ahn, Ji Hyeon,Tae, Hyun-Jin,Shin, Myoung Cheol,Ohk, Taek Geun,Cho, Jun Hwi,Won, Mo Medknow PublicationsMedia Pvt Ltd 2016 Neural regeneration research Vol.11 No.7
<P>Ischemic preconditioning (IPC) is a condition of sublethal transient global ischemia and exhibits neuroprotective effects against subsequent lethal ischemic insult. We, in this study, examined the neuroprotective effects of IPC and its effects on immunoreactive changes of antioxidant enzymes including superoxide dismutase (SOD) 1 and SOD2, catalase (CAT) and glutathione peroxidase (GPX) in the gerbil hippocampal CA1 region after transient forebrain ischemia. Pyramidal neurons of the stratum pyramidale (SP) in the hippocampal CA1 region of animals died 5 days after lethal transient ischemia without IPC (8.6% (ratio of remanent neurons) of the sham-operated group); however, IPC prevented the pyramidal neurons from subsequent lethal ischemic injury (92.3% (ratio of remanent neurons) of the sham-operated group). SOD1, SOD2, CAT and GPX immunoreactivities in the sham-operated animals were easily detected in pyramidal neurons in the stratum pyramidale (SP) of the hippocampal CA1 region, while all of these immunoreactivities were rarely detected in the stratum pyramidale at 5 days after lethal transient ischemia without IPC. Meanwhile, their immunoreactivities in the sham-operated animals with IPC were similar to (SOD1, SOD2 and CAT) or higher (GPX) than those in the sham-operated animals without IPC. Furthermore, their immunoreactivities in the stratum pyramidale of the ischemia-operated animals with IPC were steadily maintained after lethal ischemia/reperfusion. Results of western blot analysis for SOD1, SOD2, CAT and GPX were similar to immunohistochemical data. In conclusion, IPC maintained or increased the expression of antioxidant enzymes in the stratum pyramidale of the hippocampal CA1 region after subsequent lethal transient forebrain ischemia and IPC exhibited neuroprotective effects in the hippocampal CA1 region against transient forebrain ischemia.</P>
Ceramide and Sphingosine 1-Phosphate in Liver Diseases
Park, Woo-Jae,Song, Jae-Hwi,Kim, Goon-Tae,Park, Tae-Sik Korean Society for Molecular and Cellular Biology 2020 Molecules and cells Vol.43 No.5
The liver is an important organ in the regulation of glucose and lipid metabolism. It is responsible for systemic energy homeostasis. When energy need exceeds the storage capacity in the liver, fatty acids are shunted into nonoxidative sphingolipid biosynthesis, which increases the level of cellular ceramides. Accumulation of ceramides alters substrate utilization from glucose to lipids, activates triglyceride storage, and results in the development of both insulin resistance and hepatosteatosis, increasing the likelihood of major metabolic diseases. Another sphingolipid metabolite, sphingosine 1-phosphate (S1P) is a bioactive signaling molecule that acts via S1P-specific G protein coupled receptors. It regulates many cellular and physiological events. Since an increase in plasma S1P is associated with obesity, it seems reasonable that recent studies have provided evidence that S1P is linked to lipid pathophysiology, including hepatosteatosis and fibrosis. Herein, we review recent findings on ceramides and S1P in obesity-mediated liver diseases and the therapeutic potential of these sphingolipid metabolites.