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Ju Hyoung Lee,Kyeong Min Jo,Tae Oh Kim,Jong Ha Park,Seung hyun Park,Jae Won Jung,So Chong Hur,양성연 대한소화기내시경학회 2016 Clinical Endoscopy Vol.49 No.6
Brunner’s gland hamartomas are small benign lesions that are most commonly found in the bulb of the duodenum. They are veryuncommon, and most are found incidentally during upper gastrointestinal series or esophagogastroduodenoscopy. The lesions tendto be asymptomatic, but patients may present with symptoms of duodenal obstruction or hemorrhage secondary to ulceration. Histologically, a Brunner's gland hamartoma consists of the components of Brunner's gland cells, as well as glandular, adipose andmuscle cells. In this study, we report the case of a 30-year-old man who presented with upper gastrointestinal bleeding and obstructivesymptoms due to a giant Brunner's gland hamartoma in the duodenal bulb. The hamartoma was successfully removed by endoscopicresection. No significant complications were observed. Microscopically, the lesion was found to be entirely composed of variableBrunner's glands and adipocytes.
Seung-Ju Ahn,Jae-Kyoung Choi,Young Mi Joo,Min-A Lee,Pyung-Rak Choi,Yeong-Mi Lee,Myong-Shin Kim,So-Young Kim,Eun-Hee Jeon,Byung-In Min,Chong-Rak Kim 대한의생명과학회 2004 Biomedical Science Letters Vol.10 No.3
Lung cancer is a leading cause of cancer death worldwide; however, despite major advances in cancer treatment during the past two decades, the prognostic outcome of lung cancer patients has improved only minimally. This is largely due to the inadequacy of the traditional screening approach of diagnosis in lung cancer, which detects only well-established overt cancers and fails to identify precursor lesions in premalignant conditions of the bronchial tree. In recent years this situation has fundamentally changed with the identification of molecular abnormalities characteristic of premalignant changes; these concern tumour suppressor genes, loss of heterozygosity at crucial sites and activation of oncogenes. Basic knowledge at the molecular level has extremely important clinical implications with regard to early diagnosis, risk assessment and prevention, and therapeutic targets. In this study we used a "cap-finder" subtractive hybridization method, "long distance" polymerase chain reaction (PCR), streptavidin magnetic beads mediated subtraction, and spin column chromatography to detect differential expression genes of human small cell lung carcinoma. We have now isolated ninety two genes that expressed differentially in the human small cell lung carcinoma cells and analyzed of 12 clones with sequencing, nine cDNAs include tapasin (NGS-17) mRNA, BC200 alpha scRNA, chromosome 12q24 PAC RPCI3-462E2, protein phosphatase 1 (PPPICA), translocation protein 1 (TLOC1), ribosomal protein S24 (RPS24) mRNA, protein phosphatase (PPEF2), cathepsin Z, MDM2 gene and three novel genes. They may be oncogenesis-related proteins.
삼배체 염색체 이상을 보인 급성 림프구성 백혈병 1 예
전종구(Chong Ku Jun),차주영(Ju Young Cha),오형모(Hyung Mo Oh),신요식(Yo Shik Shin),김윤권(Yun Kwon Kim),김소연(So Yon Kim),김영중(Young Jung Kim),박병익(Byung Yik Park),조민구(Min Koo Cho),이권전(Gwon JUn Lee),이경인(Kyung In Lee),이은 대한내과학회 2001 대한내과학회지 Vol.61 No.2
In addition to age, white cell count and immunophenotype, karyotype has been reported to be one of the important prognostic factors in acute lymphocytic leukemias. Furthermore 70 percent of patients with acute B lymphocytic leukemia presented chromosomal abnormalities, which is known to have a close relationship with the prognosis. Among the abnormalities, triploid is rare and known to have the worse prognosis. Structural chromosomal abnormality of the 11q23 band is more common in childhood acute lymphocytic leukemia and has been rarely reported in adult lymphocytic leukemia. We present a case of a 29 year - old male patient with acute lymphocytic leukemia, who had triploid and chromosomal translocation including 11q23 band along with the review of related literature.(Korean J Med 61:190-194, 2001)
Jang, Moon Ju,Kim, Hye-Sun,Lee, Hye-Gyn,Kim, Gi Jin,Jeon, Hwang Gyun,Shin, Hyun-Soo,Chang, Sei-Kyung,Hur, Gin-Hyung,Chong, So Young,Oh, Doyeun,Chung, Hyung-Min S. Karger AG 2013 Acta haematologica Vol.129 No.4
<P>Abstract</P><P><B><I>Background and Aims:</I></B> Immunomodulatory properties of mesenchymal stem cells (MSCs) have been applied to reduce the incidence of graft-versus-host disease (GVHD) after hematopoietic stem cell transplantation (HSCT). Among the various sources of MSCs that have immunomodulatory effects in vitro, only placenta-derived MSCs (PD-MSCs) have not been evaluated in an in vivo model of GVHD. In this study, we investigated the immunomodulatory properties of PD-MSCs in vitro and evaluated their clinical potential for controlling GVHD in an animal model. <B><I>Methods:</I></B> A GVHD animal model was established by transplanting C57BL/6 donor bone marrow cells and spleen cells into lethally irradiated BALB/c recipient mice. To control GVHD, human PD-MSCs were transplanted into recipient mice (5 × 10<SUP>5</SUP> or 1 × 10<SUP>6</SUP> cells). <B><I>Results:</I></B> PD-MSCs suppressed mitogen-stimulated T cell proliferation in vitro in a dose-dependent manner. Moreover, PD-MSCs inhibited cytokine secretion (interleukin-12, tumor necrosis factor-α and interferon-γ) of activated T cells. In vivo, the survival rate in the PD-MSC group (transplanted with 1 × 10<SUP>6</SUP> cells) was higher than that in the control group and histological scores were low in the PD-MSC group. <B><I>Conclusion:</I></B> We present the first evidence that human PD-MSCs can efficiently control GVHD in an HSCT in vivo model.</P><P>Copyright © 2012 S. Karger AG, Basel</P>
Jang, Moon Ju,Jeon, Young Joo,Min, Kyung Tae,Oh, Jisu,Chong, So Young,Park, Seonyang,Yun-Choi, Hye Sook,Oh, Doyeun,Kim, Nam Keun Raven Press 2012 Clinical and applied thrombosis/hemostasis Vol.18 No.4
<P>Although it was thought that platelets did not play a significant role in the pathogenesis of venous thromboembolism (VTE), several studies demonstrated that a marked activation of platelets occurs in patients with VTE. We carried out a case-control study to investigate the effect of the T744C P2RY12 polymorphism on the risk of VTE in the Korean population. We enrolled 154 consecutive patients with VTE and 415 healthy controls. Genotype frequencies for patients with TT, TC, and CC were 71.4%, 24.7%, and 3.9% and in the controls, 68.2%, 30.1%, and 1.7%, respectively. T744C P2RY12 polymorphism did not significantly affect the risk of VTE. Our study shows that T744C P2RY12 polymorphism did not significantly affect the risk of VTE in the Korean population.</P>