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안용호 한국생명과학회 1994 생명과학회지 Vol.4 No.4
The glucose transport across the mammalian plasma membranes is carried out by members of two distinct gene families, $Na^+$/glucose to transporter (SGLT) and glucose transporters (GLUTs). The energy requiring SGLT utilizes the sodium gradient to transport glucose and galactose against the concentration gradient. The energy independent transport (Facilitative transport) of glucose down the concentration gradient is mediated by the members of GLUTs. The facilitated transport of glucose is saturable, sterospecific and bidirectional across the membrane. To date, 6 kinds of isoforms of facilitative glucose transporters are found. These proteins are expressed in a tissue and cell specific manner, and shows distinct properties that reflect their specific functional roles.
안용호,최연규,송병기,한병성 한국전기전자재료학회 2001 전기전자재료학회논문지 Vol.14 No.12
This paper was researched the effect of slurry stability on the mechanical and electrical property of the porcelain insulator with various raw materials such as feldspar, quartz, clay and l7wt% alumina. The slurry was fabricated after ball milling the mixed raw materials. Green compacts were made by the extrusion and were sintered at 1300$\^{C}$ for 60min in the tunnel kiln. All of the specimens were densified 96% of the theoretical density. The 3-point flexural strength($\sigma$$\_$B/) of the specimen stabilized slurry pH 7.8 was 1650 k9/㎠ and the vickers hardness(Hv) and the fracture toughness(K$\_$IC/) were 27.5 GPa and 2.2 MPa$.$m$\^$$\sfrac{1}{2}$/, respectively. The mechanical properties of the specimen stabilized slurry PH 9.3 were 1716 kg/㎠($\sigma$$\_$B/), 27.6 GPa(Hv) and 3.0 MPa$.$m$\^$$\sfrac{1}{2}$/(K$\_$IC/), respectively. The dielectric strength was increased from 8.3kV/mm to 13.2kV/mm as the increase of the slurry pH from 7.8 to 9.3. Therefore the physical properties of the specimen stabilized slurry pH 9.3 were improved.
안용호 대한당뇨병학회 2018 Diabetes and Metabolism Journal Vol.42 No.6
My professional journey to understand the glucose homeostasis began in the 1990s, starting from cloning of the promoter region of glucose transporter type 2 (GLUT2) gene that led us to establish research foundation of my group. When I was a graduate student, I simply thought that hyperglycemia, a typical clinical manifestation of type 2 diabetes mellitus (T2DM), could be caused by a defect in the glucose transport system in the body. Thus, if a molecular mechanism controlling glucose transport system could be understood, treatment of T2DM could be possible. In the early 70s, hyperglycemia was thought to develop primarily due to a defect in the muscle and adipose tissue; thus, muscle/adipose tissue type glucose transporter (GLUT4) became a major research interest in the diabetology. However, glucose utilization occurs not only in muscle/adipose tissue but also in liver and brain. Thus, I was interested in the hepatic glucose transport system, where glucose storage and release are the most actively occurring.