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Kang Zhi‐Wei,Liu Fang‐Hua,Xu Yong‐Yu,Cheng Jia‐Hui,Lin Xiao‐Li,Jing Xiang‐Feng,Tian Hong‐Gang,Liu Tong‐Xian 한국곤충학회 2021 Entomological Research Vol.51 No.1
Odorant‐degrading enzymes (ODEs) have been found in insect antennae and play a critical role in signal chemical degradation once the message is conveyed. Significant progress has been made in characterizing ODEs in a variety of pests but very little is known in their natural enemies. We have carried out an antennae‐ and sex‐specific transcriptome of Aphidius gifuensis, a natural enemy of aphid, to identify the candidate ODEs. Based on the antennae‐ and sex‐specific transcriptome, a total of 100 putative ODEs were identified including one aldehyde oxidase (AOX), four alcohol dehydrogenases (ADs), eight UDP‐glucuronosyltransferases (UGTs), 45 cytochrome P450 (P450s), nine glutathione S‐transferases (GSTs) and 40 carboxylesterases (CCEs or CXEs). Additionally, we used RT‐qPCR to determine the expression profiles of these genes in tissues of both sexes. Based on the phylogenic analysis and tissue‐expression patterns, AgifEstE4, AgifCXE3, AgifCCE4, AgifCCE7, and AgifCCE18 were suggested as key ODEs in A. gifuensis. In addition, the female or male specifically enriched genes, such as AgifCCE17, AgifEstB1, AgifCYP18a1, AgifUGT2C2, were also considered to involve in the chemosensory processing in A. gifuensis. This study not only identified the candidate ODEs in A. gifuensis but also provided source for further exploration of the molecular mechanisms of chemical signal transductions in A. gifuensis, as well as other hymenopteran species.
Wei Qiang Jia,Cho Kwang Min,Jeung Joo Lee,Kee Woong Park 한국잡초학회 2021 한국잡초학회 별책(학술대회 초록집) Vol.41 No.1
Schoenoplectus juncoides is a noxious weed in paddy field of South Korea. The excessive and unreasonable use of ALS-inhibiting herbicide caused the evolution of herbicide-resistant S juncoides. As a part of the management of ALS-inhibiting herbicide resistance, early detection of the ALS-inhibiting herbicide resistance in S juncoides is necessary. In this study, we evaluated three methods of rapidly detecting ALS-resistance in S juncoides, including the cutting root test, cutting shoot test, and resistance in-season quick (RISO) test. Three herbicide-resistant populations of S juncotdes (GR, 217R, and WR) and one susceptible population of S juncoides (HS) collected from the Chungnam and Jeonbuk provinces of South Korea served as experimental materials for the evaluation of three resistance test methods established by previous studies. In the three test methods, the GR and 217R were confirmed as resistant to flucetosulfuron and susceptible to penoxsulam. The WR was highly resistant to flucetosulfuron and penoxsulam. All the three resistance test methods have been successfully applied for identifying the ALS-inhibiting herbicide-resistant S juncoides in-season. Especially, the result of cutting roots test demonstrated that the GR, 217R, and WR biotypes were highly resistant to flucetosulfuron with the R/S ratios of 34, 100, and 1852, respectively. The WR biotype was highly resistant to penoxsulam with the R/S ratio of 912, whereas the GR and 217R biotypes were susceptible to penoxsulam. These in-season quick tests are likely to contribute to the use of effective herbicide in crops leading to decreased costs of labour and inputs in paddy field, and increased profits.
Jia-Lin Song,Wei Zheng,Wei Chen,Yun Qian,Yuan-Ming Ouyang,Cun-Yi Fan 생화학분자생물학회 2017 Experimental and molecular medicine Vol.49 No.-
Our study aims to explore the effects of lentivirus-mediated microRNA-124 (miR-124) gene-modified bone marrow mesenchymal stem cell (BMSC) transplantation on the repair of spinal cord injury (SCI) in rats. BMSCs were isolated from the bone marrow of rats. The target gene miR-124 was identified using a luciferase-reporter gene assay. Seventy-two rats were selected for construction of the SCI model, and the rats were randomly divided into the blank group, sham group, SCI group, negative control (NC) group, overexpressed miR-124 group and si-PDXK group. The mRNA expression of miR-124 and the mRNA and protein expression of pyridoxal kinase (PDXK) were detected by quantitative real-time polymerase chain reaction and western blotting. The locomotor capacity of the rats was evaluated using the Basso, Beattie and Bresnahan (BBB) scale. Brdu, neuron-specific enolase (NSE), neurofilament (NF) and microtubule-associated protein 2 (MAP2) were detected using immunohistochemistry. The expression levels of thyrotropin-releasing hormone (TRH), prostacyclin (PGI2) and gangliosides (GM) were measured using an enzyme-linked immunosorbent assay. PDXK was identified as the target gene of miR-124. The overexpressed miR-124 group exhibited higher miR-124 expression than the SCI, NC and si-PDXK groups. Compared with the SCI and NC groups, the PDXK expression was downregulated in the overexpressed miR-124 and si-PDXK groups, and the BBB scores were significantly increased 7, 21 and 35 days after transplantation. The double-labeled positive cell densities (Brdu+NSE/NF/MAP2) and the expression levels of TRH, PGI2 and GM in the overexpressed miR-124 group were significantly higher than those in the NC and SCI groups. These results indicated that miR-124 targeted PDXK to accelerate the differentiation of BMSCs into neurocytes and promote SCI repair.