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- 저자 : 조경삼 ( K. S. Cho ) (검색결과 5 건)
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급성(急性) 신부전(腎不全)이 동반(同伴)된 근양사(筋壤死)에 있어서의 $^{99m}Tc$-MDP 골주사(骨走査)
문희범,한진석,김삼용,조보연,이정상,고창순,조경삼,Moon, H.B.,Han, J.S.,Kim, S.Y.,Cho, B.Y.,Lee, J.S.,Koh, C.S.,Cho, K.S. 대한핵의학회 1980 핵의학 분자영상 Vol.14 No.2
We studied four patients with muscle necrosis associated with acute renal failure to evaluate the diagnostic value of the bone scan in this disease. The illness followed carbon monoxide poisoning in two patients, acute physical exertion in one and contaminated intramuscular injection in the other. Whole-body rectilinear bone scans using technetium 99m-methyldiphosphonate were done. In all patients, increased muscle labelling at the regions of suspected muscle injury was showed, and in one, it was after normalization of serum muscle enzyme levels. In one patient, the bone scan was rechecked 8 months later and showed no residual abnormality. Above all, the site and precise extent of muscle injury could be detected and the degree of muscle labelling seemed to correlate with the severy of muscle injury. These findings suggest that isotope scanning may be useful in the diagnosis of patients with acute muscle necrosis.
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그레이브스병 환자의 Suppressor Induction Factor ( SIF ) 의 활성도
양인명(I . M . Yang),윤휘중(H . J . Yoon),조경삼(K . S . Cho),김진우(J . W . Kim),김광원(K . W . Kim),최영길(Y . K . Choi) 대한내과학회 1986 대한내과학회지 Vol.31 No.1
N/A Autologous mixed lymphocyte culture reaction (AMLCR) has been known to represent the generation of suppressor T cell without direct evidence that the proliferating cell is suppressor T cell itself. Our previous study had demonstrated that the proliferative response of T cell during AMLCR in the patients with Graves' disease is not different from that in normal control, The recent studies showed that suppressor induction factor(SIF) which induce suppressor T cell is produced during the AMLCR. We observed the SIF activity in the patients with Graves disease by measuring the suppression of normal allogeneic MLCR by the normal T cell incubated with the supernatant of AVLCR, and also observed the proliferative response of T cell incubated with the supernatant of AMLCR proliferate to determine whether the suppressor T cell proliferate or not during AMLCR. T cells incubated with the supernatant of AMLCR suppressed the allogeneic MLCR prominently in normal controls(55±11%), and the supernatant of patients' AMLC induced the suppressor T cells which successfully suppressed the allogeneic MLCR(50±9%). T cells incubated with the supernatant of AMLCR did not proliferate. These data indicate that the proliferating cells during AMLCR are the self-antigen recognizing T cells which secrets SIF, a kind of lymphokines, to induce the suppressor T cells, It is suggested that the process of self antigen recognization is normal in Graves' disease.
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각종 갑상선질환에서 혈청 myoglobin치에 관한 연구
정순일,조보연,이문호,장연복,고창순,조경삼,김삼용 대한핵의학회 1980 핵의학 분자영상 Vol.14 No.2
각종 갑상선질환에서 혈청 myoglobin치의 변화를 관찰하고 방사면역측정법으로 혈청 myoglobin치를 측정한 결과 다음과 같은 결과를 얻었다. 1) 치료전 갑상선기능항진증 환자의 혈청 myoglobin 평균치는 39.1±20.3 ng/ml로 정상대조군의 50.9±17.0 ng/ml보다 낮았으나 통계적으로 유의한 차이는 없었다. 2) 갑상선기능저하증 환자의 혈청 myoglobin치는 28∼156 ng/ml로 5예중 3예에서 증가되어 있었다. 3) 혈청 myoglobin치와 혈청 T₃, T₄치와는 유의한 상관관계가 없었다. 4) 갑상선중독성 근병증의 진단과 그 정도 및 치료후 경과관찰의 지표로서 유용할 가치가 있다고 사료된다. To evaluate the change of serum myoglobin in the thyroid disease, we measured serum myoglobin by radioimmunoassay in 24 normal subjects, 34 patients with hyperthyroidism (20 untreated, 14 treated) and 5 patients with hypothyroidism. The mean values of serum myoglobin in 24 normal control, 20 untreated hyperthyroidism and 14 treated hyperthyroidism were 50.9±17.0 ng/ml, 39.1±203 ng/ml and 51.0±18.3 ng/ml respectively. The serum myoglobin level of untreated hyperthyroidism seemed to be lower than that of normal control without statistical significance. The serum myoglobin levels in 5 patients with hypothyroidism ranged 25-156 ng/ml, and 3 patients showed increased serum myoglobin level. We observed one case with severe degree of thyrotoxic myopathy whose myoglobin level was markedly elevated initially and was decreased to normal range after treatment. This results suggests that the determination of serum myoglobin might be useful in the evaluation of thyrotoxic myopathy.
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이문호,홍기석,고창순,이권전,조경삼,김병국,김노경 대한핵의학회 1977 핵의학 분자영상 Vol.11 No.1
$quot;1965년부터 1977년까지 사이에 서울대학교 의과대학 부속병원 내과에서 진성다혈증으로 진단받았던 8명의 환자에 대하여 임상적 고찰을 시행하여 다음과 같은 결과를 얻었다. 1) 성별로는 남녀 각각 4명이었으며 진단시의 평균연령은 54세였고 50대가 3명으로 가장 많았다. 2) 자각증상은 안면홍조 두통, 소양증, 및 현훈의 순이었고, 타각증상으로는 안면홍조, 비장종대(6명), 간장종대(4명)의 순이었다. 3) 검사실적소견상 적혈구용적은 55±10ml/kg로 전례에서 증가되었고, 동맥혈산소포화도도 전례에서 92% 이상이었으며, 말초혈액내 백혈구수는 5명에서, 혈소판은 2명에서 그리고 leukocyte alkaline phosphatase치는 전례에서 증가되어 있었다. 4) 골수천자소견상 골수세포증식은 5례(63%)에서 관찰되었고, 거대핵세포의 증식은 4례에서 있었으며, 골수철분은 전례에서 소실되었다. 골수생검을 실시한 2례중 1례에서만 골수섬유화를 정시하고 있었다. 5) 병발된 질환으로는 고혈압이 2례, 복부대동맥과 좌측신동맥의 혈전증이 1례 있었다. 6) 치료는 사혈만을 실시한 예가 2례 busulfan을 이용한 화학요법을 시행한 경우가 1례이었으며 나머지 5례에서는 32P를 투여하였다. 7) 평균 29.9개월간 $quot; Eight patients with polycythemia vera were diagnosed from 1965 through 1977 among the patients of Seoul National University Hospital. In our series there were 4 men and 4 women with average age of 54 years at the time of diagnosis. Symptoms of facial plethora, headache, pruritus and dizziness were the most frequent manifestations of disease in order of frequency. At inital examination 8 of 8 patients had facial plethora, 6 of 8 patients splenomegaly and 4 if 8 patients hepatomegaly. The laboratory data revealed mean Red cell volume of 55+10ml/kg which was elevated in all cases. Leukocytosis was observed in 5 of 8 patients and thrombocytosis 2 of 8 patients. Leukocyte alkaline phosphatase was increased in all cases. Bone marrow aspiration disclosed typical panmyelosis in 5 of 8 patients. In one patient there was diffuse myelofibrosis on bone marrow biopsy. The combined diseases included 2 patient with hypertension and 1 patient with thrombosis of aorta and left renal artery. The patients were treated with phlebotoany alone in 2 patients, chemotherapy with busulfan in 1 patient and 32P in 5 patients with favorable results. During the mean 29.9 months follow up period, there were development of iron deficiency in 3 patients, hyperuricemia in 2 patients and thrombosis in 1 patient. The occurrence of acute leukemia of myelofibrosis was not observed in the course of disease.
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