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정헌섭(Jung, Heon-Seob),민병권(Min, Byung-Kwon),이성훈(Lee, Seong-Hoon),우희수(Woo, Hee-Soo),박현호(Park, Hyun-Ho) 한국소음진동공학회 2012 한국소음진동공학회 논문집 Vol.22 No.9
We considered an approach in terms of materials for improvement of insulation performance of vehicle rubber hoses. Ethylene propylene rubber(EPDM) for heater hoses in cooling system and acrylic rubber(AR) for intercooler hose in intake system were chosen for mixing for the vibration and noise performance. We modified EPDM and AR through changing compound of base polymer, reinforcement fillers and additives. Dynamic mechanical analysis(DMA) was used to measure viscoelastic properties such as shear modulus and loss factor($tan{\delta}$). Vehicle acceleration test was also conducted to observe indoor changes in insulation performance of hoses.
Lee, Heon-Jeong,Lee, Min-Soo,Kang, Rhee-Hun,Kim, Hyun,Kim, Soon-Duck,Kee, Baik-Seok,Kim, Young Hoon,Kim, Yong-Ku,Kim, Jung Bum,Yeon, Byung Kil,Oh, Kang Seob,Oh, Byung-Hoon,Yoon, Jin-Sang,Lee, Chul,Jun Wiley Subscription Services, Inc., A Wiley Company 2005 Depression and Anxiety Vol.21 No.3
<P>Posttraumatic stress disorder (PTSD) is a prevalent anxiety disorder marked by behavioral, physiologic, and hormonal alterations. The etiology of PTSD is unknown, although exposure to a traumatic event constitutes a necessary, but not sufficient, factor. Serotonergic dysfunction has been implicated in PTSD. The present study examined the possible association between the serotonin-transporter-linked polymorphic region (SERTPR) and PTSD. The genotype and allele frequencies of the SERTPR were analyzed in 100 PTSD patients and 197 unrelated healthy controls using a case–control design. The frequency of the s/s genotype was significantly higher in PTSD patients than in normal controls. These findings suggest that the SERTPR s/s genotype is one of the genetic factors for the susceptibility to PTSD. Further investigations are required into the influence of gene polymorphisms on the biological mechanisms of PTSD, its clinical expression, and its response to treatment. Depression and Anxiety 00:1–5, 2005. © 2005 Wiley-Liss, Inc.</P>
천연고무 컴파운딩의 초탄성 재료 모델링 및 차량용 부싱 성능의 유한요소해석 연구
조중근(Jung Keun Cho),김라성(Ra Sung Kim),박인경(In-Kyung Park),김예찬(Ye Chan Kim),황민주(Min Ju Hwang),서종환(Jonghwan Suhr),정헌섭(Heon Seob Jung),강재욱(Jae Wook Kang),남재도(Jae-Do Nam) 한국고분자학회 2018 폴리머 Vol.42 No.6
본 연구에서는 차량용 고무 부싱의 설계기준이 되는 강성(stiffness)을 정확히 예측하기 위해 천연고무 컴파운드의 응력-인장 특성을 반영한 Ogden 3차 초탄성 모델이 고무 재료의 거동을 가장 정확하게 예측한다는 것을 규명하였다. 스웨이징 공정에 의한 4.6%의 변형 결과를 재료의 응력-변형률에 보정(calibration)하여 초탄성 모델에 적용한 결과, 스웨이징 효과를 고려하지 않은 예측 대비 약 45% 정확도가 향상되었으며, 실제 시험 결과와 99.6%의 정확도를 보였다. 본 연구는 정확한 고무소재 물성의 모델링을 통하여 부싱의 특성을 성공적으로 예측할 수 있다는 것을 확인하였고, 이를 통해 차량용 부싱의 설계, 공정 및 성능 평가 단계를 획기적으로 단축할 수 있는 방법을 제안하였다. The design process in automobile industry requires to predict the stiffness of rubber busing materials. The behavior of rubber compound can be simulated using the strain energy density function. It is needed to consider the change of the material properties because the compression deformation caused by the swaging process of the busing. In this study, numerical simulations of the stress-strain curve including the swaging process were carried out using the finite element method and compared with experimental data. The Ogden 3rd model of strain energy density functions predicted the behavior of the busing with natural rubber compound. The stress-strain curves of the rubber busing was calibrated using that the initial compression of the swaging process was the 4.6% strain. Compared to the stiffness of the bushing without swaging effect, the stiffness was improved by 45% and had 99.6% accuracy with the actual test results.
Endotoxin Is Not Essential for the Development of Cockroach Induced Allergic Airway Inflammation
Shin, Yoo Seob,Sohn, Jung-Ho,Kim, Joo-Young,Lee, Jae Hyun,Cho, Sang-Heon,Hong, Soo-Jong,Lee, Joo-Shil,Hong, Chein-Soo,Park, Jung-Won Yonsei University College of Medicine 2012 Yonsei medical journal Vol.53 No.3
<P><B>Purpose</B></P><P>Cockroach (CR) is an important inhalant allergen and can induce allergic asthma. However, the mechanism by which CR induces airway allergic inflammation and the role of endotoxin in CR extract are not clearly understood in regards to the development of airway inflammation. In this study, we evaluated whether endotoxin is essential to the development of CR induced airway allergic inflammation in mice.</P><P><B>Materials and Methods</B></P><P>Airway allergic inflammation was induced by intranasal administration of either CR extract, CR with additional endotoxin, or endotoxin depleted CR extract, respectively, in BALB/c wild type mice. CR induced inflammation was also evaluated with toll like receptor-4 (TLR-4) mutant (C3H/HeJ) and wild type (C3H/HeN) mice.</P><P><B>Results</B></P><P>Intranasal administration of CR extracts significantly induced airway hyperresponsiveness (AHR), eosinophilic and neutrophilic airway inflammation, as well as goblet cell hyperplasia in a dose-dependent manner. The addition of endotoxin along with CR allergen attenuated eosinophilic inflammation, interleukin (IL)-13 level, and goblet cell hyperplasia of respiratory epithelium; however, it did not affect the development of AHR. Endotoxin depletion in CR extract did not attenuate eosinophilic inflammation and lymphocytosis in BAL fluid, AHR and IL-13 expression in the lungs compared to CR alone. The attenuation of AHR, eosinophilic inflammation, and goblet cell hyperplasia induced by CR extract alone was not different between TLR-4 mutant and the wild type mice. In addition, heat inactivated CR extract administration induced attenuated AHR and eosinophilic inflammation.</P><P><B>Conclusion</B></P><P>Endotoxin in CR extracts may not be essential to the development of airway inflammation.</P>