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Dongwoo Ham,YoungYun Cho,Mi-Suk Park,Yun-Sug Park,Sun-Young Kim,Hye-Min Seol,Yoo Mi Park,Sunok Woo,Hyojee Joung,Do-Sun Lim 한국영양학회 2020 Nutrition Research and Practice Vol.14 No.6
BACKGROUND/OBJECTIVES: The Seoul Metabolic Syndrome Management (SMESY) program is a 1-yr lifestyle modification program targeting metabolic syndrome (MetS) in Seoul residents. This study investigated the associations between adherence to dietary guidelines and MetS among the SMESY program participants. SUBJECTS/METHODS: Data of 54,385 participants aged 20-64 yrs who completed the SMESY program in 2015, had information on adherence to dietary guidelines, and were not medicated for diabetes, hypertension, or dyslipidemia were analyzed. Participants underwent MetS screening and completed a lifestyle questionnaire including adherence to 10 dietary guidelines before and after participation. Participants were classified according to the number of MetS risk factors at baseline (MetS group, ≥ 3; risk group, 1-2; healthy group, none). Adherence to dietary guidelines was determined from the number of “yes” responses regarding the fulfillment of each guideline on ≥ 5 days/week. Multiple logistic regression was used to evaluate associations between newly diagnosed MetS and changes in adherence to dietary guidelines. RESULTS: In the MetS group, MetS prevalence decreased after the SMESY program (men, -41.9%p; women, -48.7%p), and all risk factors were significantly improved (P < 0.0001). All groups exhibited improved adherence to all dietary guidelines after participation (P < 0.0001). In the MetS group with positively changed adherence scores, the MetS prevalence decreased by -44.1%p for men and -49.5%p for women, whereas the prevalence in those with negative changes decreased by -38.1%p for men and -48.6%p for women. In the risk group, those with positively changed adherence scores had significantly decreased odds ratios (ORs) for newly diagnosed MetS compared with those with negative changes (OR, 0.70; 95% confidence interval [CI], 0.61-0.80 for men; OR, 0.88; 95% CI, 0.79-0.99 for women). CONCLUSIONS: The SMESY program may effectively reduce the risk of MetS among adults with risk factors by improving adherence to dietary guidelines.
Mi Hwa Oh,Hee Sung Hwang,Boram Chung,Hyun Dong Paik,Sangha Han,Sun Moon Kang,Jun Sang Ham,Hyoun Wook Kim,Kuk Hwan Seol,Aera Jang,Gyoo Yeol Jung 한국축산식품학회 2012 한국축산식품학회지 Vol.32 No.2
This report outlines the development of a rapid, simple, and sensitive detection system for pathogenic bacteria using a capillary electrophoresis-based, single strand conformation polymorphism (CE-SSCP) combined with PCR. We demonstrate that this method, used with primers targeting the V4 region of the16S rRNA gene, is capable of the simultaneous detection of 10 microbes that could be associated with foodborne illness, caused by animal-derived foods: Salmonella enter-ica, Listeria monocytogenes, Escherichia coli O157:H7, Campylobacter jejuni, Staphylococcus aureus, Bacillus cereus, Clostridium perfringens, Yersinia enterocolitica, Vibrio parahaemolyticus, and Enterobacter sakazakii. The traditional detection techniques are time-consuming and labor-intensive, due to the necessary task of separate cultivation of each target species. As such, the CE-SSCP-PCR method, that we have developed, has the potential to diagnose pathogens rapidly, unlike the traditional technique, in order to prevent foodborne illness in a much more efficient manner.
Ligand‐activated interaction of PPARδ with c‐Myc governs the tumorigenicity of breast cancer
Ham, Sun Ah,Kim, Eunsu,Yoo, Taesik,Lee, Won Jin,Youn, Ju Ho,Choi, Mi‐,Jung,Han, Sung Gu,Lee, Chi‐,Ho,Paek, Kyung Shin,Hwang, Jung Seok,Seo, Han Geuk Alan R. Liss, Inc 2018 International journal of cancer Vol.143 No.11
<P>Peroxisome proliferator‐activated receptor (PPAR) δ is a promising therapeutic target in metabolic and inflammatory disorders. However, its role in oncogenesis is controversial, and its therapeutic potential remains to be determined. In our study, we show that ligand‐activated PPARδ forms a complex with the proto‐oncogene product c‐Myc. The interaction of PPARδ with c‐Myc affected the transcriptional activity of c‐Myc and the expression of its target genes. The PPARδ‐dependent regulation of c‐Myc activity was associated with decreased tumorigenicity in breast cancer cells. Administration of the PPARδ ligand GW501516 inhibited tumor growth in xenograft model mice bearing MDA‐MB‐231 cells stably expressing wild‐type PPARδ, but not those expressing dominant‐negative PPARδ, by interfering with c‐Myc function through protein–protein interaction. Our results indicating that PPARδ forms an antitumorigenic complex with c‐Myc in the presence of ligand suggest a potential role of PPARδ in breast cancer development.</P>
연구논문 : 국내외 농산물 인증마크에 대한 소비자 인식의 비교
박미선 ( Mi Sun Park ),이범준 ( Bum Jun Lee ),함선옥 ( Sunny Ham ),이한주 ( Han Ju Lee ) 한국식생활문화학회 2014 韓國食生活文化學會誌 Vol.29 No.6
The purpose of the study is to determine how consumers perceive certification marks for agricultural products. The study examined three agricultural product certifications, such as the certification of environment-friendly agricultural products, Good Agricultural Procuts (GAP) certification mark, and United States Department of Agriculture (USDA) certification mark. A survey was utilized for data collection from adults over the age of 19 in November, 2013. A total of 390 questionnaires were distributed, and 300 of those were obtained for an analysis of the study. The results were as follows. First, consumers`` awareness of, image of, and credibility on the agricultural products with the certification marks were not significantly different among the three certification marks except consumers’ awareness. Second, consumers’ purchase intentions for and willingness to pay for the agricultural products with the certification marks were not significantly different among the three certification marks. Third, these variables were not significantly different between buying group and non-buying group except consumers’ awareness. Lastly, buying group and non-buying group were significantly different only in gender and marital status. The findings offer implications to the food and food service industry regarding consumers’ purchase of certified agricultural products. Further, the study enforces the roles of food industry for environment.
( Youngseok Ham ),( Ji-eun An ),( Soo Min Lee ),( Sang-hoon Chung ),( Sun Hee Kim ),( Mi-jin Park ) 한국목재공학회 2021 목재공학 Vol.49 No.3
신갈나무는 국내 전역에 두루 분포되어 있는 경제, 산업적으로 활용 가치가 큰 수종이지만, 변색, 부후 등의 열화에 의한 피해가 심각하다. 이러한 이유로 신갈나무의 부후는 목재로써의 활용에 걸림돌이 되나, 부후 요인에 대한 연구는 미비한 실정이다. 본 연구에서는 신갈나무 부후에 영향하는 요인으로 곰팡이에 주목하였으며, 신갈나무의 부후 부위로부터 곰팡이를 분리, 동정하였다. 또한, 동정 된 곰팡이가 실제로 목재 열화에 영향을 미치는지 확인하기 위해 효소 활성을 평가하고, 곰팡이를 처리한 신갈나무 목재의 질량 손실을 목재 부후 실험을 통해 측정하였다. 신갈나무에서 분리된 곰팡이 5종의 genomic DNA의 ITS region을 이용한 염기서열 분석을 통해, Mucoromycota phylum에 속하는 Mucor circinelloides, Cunninghamella elegans, 그리고 Umbelopsis isabellina 3종과 Ascomycota phylum에 속하는 Ophiostoma piceae와 Aureobasidium melanogenum 2종의 곰팡이가 동정되었다. 이러한 5종의 곰팡이는 목재의 부후와 관련된 cellulase나 laccase와 같은 효소 활성이 있으며, 실제로 신갈나무의 심재와 변재의 중량을 감소시켰다. 특히, cellulase와 laccase 활성을 모두 보유한 O. piceae와 A. melanogenum는 신갈나무의 중량을 각각 6.9%와 1.5% 감소시켰다. 이러한 결과들은 본 연구에서 동정된 5종의 곰팡이가 신갈나무의 열화에 영향한다는 것을 의미하며, 신갈나무에 대한 목재 부후균으로써의 가능성을 시사한다.
Oh, Mi-Hwa,Hwang, Hee-Sung,Chung, Bo-Ram,Paik, Hyun-Dong,Han, Sang-Ha,Kang, Sun-Moon,Ham, Jun-Sang,Kim, Hyoun-Wook,Seol, Kuk-Hwan,Jang, Ae-Ra,Jung, Gyoo-Yeol Korean Society for Food Science of Animal Resource 2012 한국축산식품학회지 Vol.32 No.2
This report outlines the development of a rapid, simple, and sensitive detection system for pathogenic bacteria using a capillary electrophoresis-based, single strand conformation polymorphism (CE-SSCP) combined with PCR. We demonstrate that this method, used with primers targeting the V4 region of the16S rRNA gene, is capable of the simultaneous detection of 10 microbes that could be associated with foodborne illness, caused by animal-derived foods: Salmonella enterica, Listeria monocytogenes, Escherichia coli O157:H7, Campylobacter jejuni, Staphylococcus aureus, Bacillus cereus, Clostridium perfringens, Yersinia enterocolitica, Vibrio parahaemolyticus, and Enterobacter sakazakii. The traditional detection techniques are time-consuming and labor-intensive, due to the necessary task of separate cultivation of each target species. As such, the CE-SSCP-PCR method, that we have developed, has the potential to diagnose pathogens rapidly, unlike the traditional technique, in order to prevent foodborne illness in a much more efficient manner.
Piperlongumine Improves Lipopolysaccharide-Induced Amyloidogenesis by Suppressing NF-KappaB Pathway
Gu, Sun Mi,Lee, Hee Pom,Ham, Young Wan,Son, Dong Ju,Kim, Hoi Yeong,Oh, Ki Wan,Han, Sang-Bae,Yun, Jaesuk,Hong, Jin Tae Springer US 2018 Neuromolecular medicine Vol.20 No.3
<P>Amyloidogenesis is known to cause Alzheimer’s disease. Our previous studies have found that lipopolysaccharide (LPS) causes neuroinflammation and amyloidogenesis through activation of nuclear factor kappaB (NF-κB). Piperlongumine (PL) is an alkaloid amide found naturally in long pepper (<I>Piper longum</I>) isolates; it was reported to have inhibitory effects on NF-κB activity. We therefore investigated whether PL exhibits anti-inflammatory and anti-amyloidogenic effects by inhibiting NF-κB. A murine model of LPS-induced memory impairment was made via the intraperitoneal (i.p.) injection of LPS (0.25 mg/kg/day, i.p.). We then injected PL (1.5 or 3.0 mg/kg/day, i.p.) for 7 days in three groups of mice to observe effects on memory. We also conducted an in vitro study with astrocytes and microglial BV-2 cells, which were treated with LPS (1 µg/mL) or PL (0.5 or 1.0 or 2.5 µM). Results from our behavioral tests showed that PL inhibited LPS-induced memory. PL also prevented LPS-induced beta-amyloid (Aβ) accumulation and inhibited the activities of β- and γ-secretases. The expression of inflammatory proteins also was decreased in PL-treated mice, cultured BV-2, and primary astrocyte cells. These effects were associated with the inhibition of NF-κB activity. A docking model analysis and pull-down assay showed that PL binds to p50. Taken together, our findings suggest that PL diminishes LPS-induced amyloidogenesis and neuroinflammation by inhibiting NF-κB signaling; PL therefore demonstrates potential for the treatment of Alzheimer’s disease.</P>