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( Sang Mi Chung ),( Ju Whan Choi ),( Young Seok Lee ),( Jong Hyun Choi ),( Jee Youn Oh ),( Kyung Hoon Min ),( Gyu Young Hur ),( Sung Yong Lee ),( Jae Jeong Shim ),( Kyung Ho Kang ) 대한결핵 및 호흡기학회 2019 Tuberculosis and Respiratory Diseases Vol.82 No.1
Background: Bronchoscopy is a useful diagnostic and therapeutic tool. However, the clinical use of high-flow nasal cannula (HFNC) in adults with acute respiratory failure for diagnostic and invasive procedures has not been well evaluated. We present our experiences of well-tolerated diagnostic bronchoscopy as well as cases of improved saturation in hypoxaemic patients after a therapeutic bronchoscopic procedure. Methods: We retrospectively reviewed data of hypoxaemic patients who had undergone bronchoscopy for diagnostic or therapeutic purposes from October 2015 to February 2017. Results: Ten patients (44-75 years of age) were enrolled. The clinical purposes of bronchoscopy were for diagnosis in seven patients and for intervention in three patients. For the diagnoses, we performed bronchoalveolar lavage in six patients. One patient underwent endobronchial ultrasonography with transbronchial needle aspiration of a lymph node to investigate tumour involvement. Patients who underwent bronchoscopy for therapeutic interventions had endobronchial mass or blood clot removal with cryotherapy for bleeding control. The mean saturation (SpO<sub>2</sub>) of prebronchoscopy in room air was 84.1%. The lowest and highest mean saturation with HFNC during the procedure was 95% and 99.4, respectively. The mean saturation in room air post-bronchoscopy was 87.4%, which was 3.3% higher than the mean room air SpO<sub>2</sub> pre-bronchoscopy. Seven patients with diagnostic bronchoscopy had no hypoxic event. Three patients with interventional bronchoscopy showed improvement in saturation after the procedure. Bronchoscopy was well tolerated in all 10 cases. Conclusion: This study suggests that the use of HFNC in hypoxaemic patients during diagnostic and therapeutic bronchoscopy procedures has clinical effectiveness.
[Fe II] 1.64 μm features of Jets and Outflows from Young Stellar Objects in the Carina Nebula
Jong-Ho Shinn,Tae-Soo Pyo,Jae-Joon Lee,Ho-Gyu Lee,Hyun-Jeong Kim,Bon-Chul Koo,Hwankyung Sung,Moo-Young Chun,A.-Ran Lyo,Dae-Sik Moon,Jaemann Kyeong,Byeong-Gon Park,Hyeonoh Hur,Dae-Sik Moon 한국천문학회 2014 天文學會報 Vol.39 No.1
( Sung Woo Cho ),( Jung Hoon Cha ),( Na Ri Park ),( Won Hee Hur ),( Pill Soo Sung ),( Jong Young Choi ),( Seung Kew Yoon ),( Si Hyun Bae ),( Hee Chul Nam ) 대한간학회 2020 춘·추계 학술대회 (KASL) Vol.2020 No.1
Aims: Non-alcoholic fatty liver disease (NAFLD) is a metabolic- related disorder ranging from simple steatosis to more severe forms, but the exact mechanism of progression remains unknown. MicroRNAs(miR), a class of small noncoding RNAs, are implicated in controlling a variety of biological processes. The aim of this study is to investigate the regulatory and protective role of miR-22-3p in NAFLD progression. Methods: Both in vitro and in vivo models of NAFLD were generated by treating HepG2 and Huh-7 cells with palmitic acid (PA) and by feeding mice a high-fat diet (HFD), respectively. HE and Oil Red O staining were used to examine liver tissue morphology and lipid deposition, respectively. qRT-PCR (quantitative real time polymerase chain reaction) was used for investigate expression of miR, SIRT1, and proteins involved in lipogenesis Results: HFD-mice hepatic tissues and PA-treated HepG2 and Huh-7 cells presented excess lipid production. Both in vitro and in vivo NAFLD model displayed decreased miR-22-3p and SIRT1 expression as evidenced by qRT-PCR. Overexpression of miR-22-3p induced downregulation of FAS, PPAR gamma and SREBP-1c via upregulation of SIRT1 expression. Reduction of hepatic lipid accumulation was observed by Oil red O staining. Conclusions: In this study, miR-22-3p had a role in ameliorating hepatic lipogenesis by regulation of SIRT1 signal pathway in NAFLD model. The overexpressed miR-22-3p protects hepatocytes from lipid metabolism and suppresses hepatic lipogenesis, suggesting as a potential target for the therapeutic strategy of NAFLD.
A Case of IgG4-Related Disease Presenting as Massive Pleural Effusion and Thrombophlebitis
( Jong Hyun Choi ),( Jae Kyeom Sim ),( Jee Youn Oh ),( Eun Joo Lee ),( Gyu Young Hur ),( Seung Heon Lee ),( Sung Yong Lee ),( Je Hyeong Kim ),( Sang Yeub Lee ),( Chol Shin ),( Jae Jeong Shim ),( Kwang 대한결핵 및 호흡기학회 2014 Tuberculosis and Respiratory Diseases Vol.76 No.4
Immunoglobulin (Ig) G4-related disease is a recently recognized systemic fibroinflammatory condition characterized by a lymphoplasmacytic infiltrate rich in IgG4-positive plasma cells with elevated circulating levels of IgG4. The disease can either be localized to one or two organs, or present as diffuse multi-organ disease. Furthermore, lesions in different organs can present simultaneously or metachronously. In the pulmonary manefestations, lesions associated with IgG4-related disease have been described in the lung parenchyma, airways and pleura, as well as the mediastinum. We report a case of IgG4-related disease presenting as massive pleural effusion and thrombophlebitis.
Hur, Won-Seok,Choi, Sang-Sik,Lee, Mi-Kyoung,Lee, Dong-Kyu,Lee, Jae-Jin,Kim, Kyong-Jong The Korean Pain Society 2011 The Korean Journal of Pain Vol.24 No.4
Kummell's disease is a spinal disorder characterized by delayed post-traumatic collapse of a vertebral body with avascular necrosis. Although definitive treatment for Kummell's disease has not been established, it has been reported that percutaneous vertebroplasty or kyphoplasty has shown good results. However, these procedures are not recommended for severely collapsed vertebral bodies because of the risk of cement leakage or technical difficulties. Authors report a rare case of spontaneous reduction in vertebral height by the insertion of a working cannula into the vertebral body in Kummell's disease.
Jong-Hee Lee,Yeon-Jae Hur,Do-Yeon Kwak,Jun-Hyun Cho,Yeong-Nam Yoon,Bong-Choon Lee,Ji-Yun Lee,Sang-Yeong Kim,Yeong-Bo Sohn,Un-Sang Yeo,Yu-Chun Song,Choon-Woo Lee,Min-Hee Nam,Jae-Keun Sohn 한국육종학회 2012 한국육종학회 심포지엄 Vol.2012 No.07
The green rice leafhopper (GRH), Nephotettix cincticeps Uhler, is one of the most serious insect pests affecting cultivated rice (Oryza sativa L.) in temperate regions of East Asia. To understand the genetic basis of the GRH resistance, a F2 population derived from across between a highly resistant variety,Cheongnam and a susceptible variety, Junambyeo was analyzed by genetic analysis and association mapping. GRH resistance was evaluated using the F2 populations. The results showed that a single dominant gene in Cheongnam. DNA from 22 F2 individuals being either resistant or susceptible were pooled to produce bulk resistant and bulk susceptible DNA samples. Parents and bulks were screened with 192 SSR markers and twolinked SSRmarker, RM6082 and RM20145 were identified.Subsequent mapping in the original mapping population showed that thelocusis flanked by the SSR markers, RM20130 and RM20152 on chromosome 6. To physically map this locus, the-linked markers were landed on the artificial chromosome clones of the reference cv., Nipponbare, released by the International Rice Genome Sequencing Project. The DNA markers found to be closely linked to Grh3 would be useful for marker-assisted selection for the improvement of resistance to GRH in rice.
Insect Tolerance Rehmannia glutinosa Cultivar, “Chunggang”
Jong Won Han(한종원),Mok Hur(허목),Sung Cheol Koo(구성철),Mei Lan Jin,Woo Moon Lee(이우문),Jae Ki Chang(장재기),Su Dong Kim(김수동),Hyo Jin Kim(김효진),Young Jin Seo(서영진),Jeong Seok Ha(하정석),Yeon Bok Kim(김연복),Sang Hoon Le 한국약용작물학회 2018 한국약용작물학회 학술대회논문집 Vol.2018 No.2