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MOLECULAR LINE OBSERVATIONS TOWARD THE COMPACT HII REGIONS IN W58
MINH Y. C.,ROH D. G.,KIM H. R. The Korean Astronomical Society 1994 Journal of The Korean Astronomical Society Vol.27 No.1
The 3mm transitions to CO, $^{13}CO,\;CS,\;HCO^+$, and HCN have been observed toward the compact HII regions in W58 using the 14m Daeduk Radio Telescope (DRT). Some of the observed lines show high-velocity wings resulted from outflowing materials of the compact HII regions. We derive the beam averaged column densities of the observed species and compare their relative abundances. The $HCO^+$ abundance appears to be smaller by about an order of magnitude than those of 'typical' quiet molecular clouds. CS may be a good reference molecule in comparing relative abundances in different physical conditions.
Pham, Loc‐,Duyen D.,Hayakawa, Kazuhide,Seo, Ji Hae,Nguyen, Minh‐,Nguyet,Som, Angel T.,Lee, Brian J.,Guo, Shuzhen,Kim, Kyu‐,Won,Lo, Eng H.,Arai, Ken Wiley Subscription Services, Inc., A Wiley Company 2012 GLIA Vol.60 No.6
<P><B>Abstract</B></P><P>After stroke and brain injury, cortical gray matter recovery involves mechanisms of neurovascular matrix remodeling. In white matter, however, the mechanisms of recovery remain unclear. In this study, we demonstrate that oligodendrocytes secrete matrix metalloproteinase‐9 (MMP‐9), which accelerates the angiogenic response after white matter injury. In primary oligodendrocyte cultures, treatment with the proinflammatory cytokine interleukin‐1β (IL‐1β) induced an upregulation and secretion of MMP‐9. Conditioned media from IL‐1β‐stimulated oligodendrocytes significantly amplified matrigel tube formation in brain endothelial cells, indicating that MMP‐9 from oligodendrocytes can promote angiogenesis <I>in vitro</I>. Next, we asked whether similar signals and substrates operate after white matter injury <I>in vivo</I>. Focal white matter injury and demyelination was induced in mice via stereotactic injection of lysophosphatidylcholine into corpus callosum. Western blot analysis showed that IL‐1β expression was increased in damaged white matter. Immunostaining demonstrated MMP‐9 signals in myelin‐associated oligodendrocytic basic protein‐positive oligodendrocytes. Treatment with an IL‐1β‐neutralizing antibody suppressed the MMP‐9 response in oligodendrocytes. Finally, we confirmed that the broad spectrum MMP inhibitor GM6001 inhibited angiogenesis around the injury area in this white matter injury model. In gray matter, a neurovascular niche promotes cortical recovery after brain injury. Our study suggests that an analogous oligovascular niche may mediate recovery in white matter. © 2012 Wiley Periodicals, Inc.</P>