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정병선(Byung-Sun Chung),강근옥(Kun-Og Kang),이정근(Jung-Kun Lee) 한국식품영양과학회 1984 한국식품영양과학회지 Vol.13 No.1
韓國人의 맛에 대한 感度와 食習慣을 알아보기 위해 몇가지 실험을 행하여 다음과 같은 結論을 얻었다.<br/> 1. 짠맛의 最少感味量濃度는 0.016 %, 단맛 0.245~0.249 %, 신맛 0.004~0.008 %, 쓴맛 0.008~0.012 %였으며 매운맛은 170,000~600,000 S. H. U. 였다.<br/> 2. 0.375 % 肉水(60℃)를 가장 맛좋게 하는 짠맛 농도는 0.3 %였고 2 % coffee(60℃)에서는 6 % 설탕 농도가 그리고 100 % orange juice에서는 산당 비 19.3(16.8 brix/0.8736 citric acid)을 가장 좋아하였다.<br/> 3. 76개 국의 平均 Brix는 3.83(2.3~6.4)%, 산도는 9.73(5.4~16)ml 0.1N NaOH, pH는 6.25(5.2~7.0) 그리고 ostwald 점도는 1.34(1.06~1.95)이었다.<br/> 4. 76개 국의 평균 염분농도는 1.127(0.904~1.448)% 였고 콩나물국, 시금치된장국의 매운정도는 12,500~47,500 S. H. U. 로 oleoresin capsicum 약 1.990~5.911(mg%)의 매운맛에 해당되었으며 김치국, 매운탕은 그 이상으로 무척 매웠다.<br/> 5. 嗜好飮食中 찌개류의 嗜好가 가장 높았으며 子女의 食習慣 형성에 특히 아버지의 영향이 크고 어릴 때부터 짜고 맵게 먹고있음이 나타났다. In order to provide basic data for food processors and to improve eating habits and healthy diets, four primary tastes and hot taste threshold of Koreans, physio-chemical properties of foods and food preference were investigated.<br/> The results of this study were as follows;<br/> 1. The recognition threshold concentration of four primary tastes was 0.016 % of salt, 0.245~0.249 % of sweet, 0.004~0.008 % of sour and 0.008~0.012 % of bitter. Threshold concentration of hot taste was 170,000~600,000 Scoville Heat Unit(S. H. U. ).<br/> 2. The most acceptable tastes were 0.3 % saltiness in 0.375 % broth(at 60℃), 6 % sweetness in 2 % instant coffee(at 60℃) and 19.3 sugar-acid ratio(16.8 brix/0.8736 % citric acid) in 100 % valencia orange juice(at 20℃).<br/> 3. The salt concentrations of soup were 1.127 % in average. S. H. U. (scale of hot taste) of soybean sprout soup and spinach bean paste soup ranged between 12,500 and 47,500 equivalent to oleoresin capsicum content of 1.990 ~ 5.911(mg %).<br/> 4. “Jigae” (Korean style stew) was the most favorable food and it was revealed that the father influences the formation of his children’s eating habits.
기술보고 : 해안 천연가스 인수기지에 대한 가속도 계측 기반의 지진경보 시스템 구축
선창국 ( Chang Guk Sun ),정병선 ( Byung Sun Jung ),김준호 ( Joon Ho Kim ),홍성경 ( Seong Kyeong Hong ),김기석 ( Ki Seog Kim ) 대한지질공학회 2010 지질공학 Vol.20 No.3
국내 해안 지역에 위치한 3곳의 천연가스 인수기지의 향후 발생 지진에 대한 대책 수립의 일환으로 기지 영역 내 자유장부지들 4개소와 저장 탱크 지지 기초 말뚝 1개에 대한 지진 가속도 계측 시스템을 구축하였다. 천연가스 인수기지에서의 계측 시스템의 성공적 설치와 운영을 위하여 특별히 몇 가지 부가 장치들이 고안 적용되었다. 계측된 자료로부터 산출되는 최대지반가속도 기반 의사 결정이 가능한 종합적 지진경보 소프트웨어 시스템을 개별 기지 영역 뿐만 아니라 중앙관리부서에서의 지진 신속 대응 목적으로 개발하였다. 또한, 전국 최대지반가속도 자료 연계 프레임을 중앙 통합 지진경보 시스템 내에 탑재하여 다양한 향후 확대 활용이 가능토록 구현하였다. 이 연구에서 개발한 천연가스 인수기지에 대한 가속도 계측 기반의 지진경보 시스템은 지반 조건 변화가 심할 수 있는 해안 지역의 시설 영역에 대한 프레임으로 매우 유용하게 이용될 수 있을 것이다. As part of preparing for future earthquakes near three LNG receiving terminals located in coastal regions of Korea, acceleration monitoring systems were installed at four free field sites and on a pile foundation beneath a storage tank in a receiving terminal. Several equipments and accessories were devised to successfully install and operate the monitoring system at LNG receiving terminals. Synthetic earthquake-alarming software systems designed for decisionmaking, based on peak ground acceleration computed using the measured data, were developed for rapid response during earthquakes, not only in each local terminal area but also in the central control division. In addition, a framework software linking nationwide data on peak ground accelerations was included in the integrated earthquake alarming system in the central division, for various future applications. The earthquake alarming systems developed in this research for LNG receiving terminals, based on acceleration monitoring, represent a useful framework for industrial facilities located in coastal regions, where geotechnical conditions may show marked spatial variations.
사람 암세포주 K562 , U937 및 쥐 암세포주 P388 , FM3A 에서의 항암제 다제내성 기전 및 내성극복
도현국(Hyun Kuk Doh),김재석(Jae Seok Kim),손지원(Ji Weon Son),성명식(Myung Sik Sung),김종성(Jong Seong Kim),김선희(Sun Hee Kim),정병선(Byung Sun Jung) 대한내과학회 1992 대한내과학회지 Vol.43 No.3
Background: To study the mechanism of multidrug resistnce (MDR) and overcome MDR in cancer cells, we investigated the combination effects of anticancer agents with calmodulin inhibitors (e,g trifluoroperazine (TFP) and W-7), calcium function modifiers (e.g., quinidine and verapamil) and a protein kinase-C activator (e.g., phorbol ester) in chronic myelogenous leukemic cells, histiocytic lymphoma cells and breast cancer cells. Methods: Some MDR mutant sublines which had cross-resistance to colchicine, actinomycm D, adriamycin and vinblastine were isolated from K562, U937 and FM3A cell lines. MTT analysis was used to measure the level of the cytotoxicity, The cytotoxicity and the combination effects were compared by IC50 (1nhibition concentration50) to control group. Results: In case of U937/MDR, TFP increased the combination effect to tenfold in colchicine (COL), eightfold in vinblastine (VLB) and thirteenfold in actinomycin D (ACT-D). Quinidine increased the effect to threefold in COL, fourfold in VLB and sevenfold in ACT-D. Verapamil also increased the effect to sevenfold in COL, sixfold in VLB fivefold in ACT-D. In case of K562/MDR, TFP, quinidine and verapamil each increased the combination effect to sixfold, twelvefold and thirteenfold in COL and sixfold, fivefold and fivefold in adriamycin (ADM). W-7 in concentration of 20uM increased the combination effect to six point sevenfold in ACT-D, twofold in COL and two point threefold in ADM. In the combination of COL with 0.48uM TPA, the cytotoxicity was reduced to two point eightfold in case of FM3A/MDR. TPA (12-0-tetradecanoyl phorbol 13-acetate), in concentration of 0.48uM, a phorbol ester, reduced the cytotoxicity to two point eightfold in case of FM3A/ MDR. Conclusion: When the combination effects of anticancer agents with calmodulin inhibitors (e.g TFP and W -7) and calcium function modifiers (e.g., quinidine and verapamil) were examined, the cytotoxicity changes of anticancer agents were small in drug sensitive cells. But in MDR cells, the combination effects were prominent. The reduced cytotoxicity of anticancer agents by TPA suggested that protein kinase-C might be related with the mechanism of acquiring drug resistance on cancer cells.