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포스터 발표 : 담도,췌장 ; 중증 급성 췌장염에서의 다발성 장기부진의 예후적 중요도
윤상정 ( Sang Jeong Yoon ),최덕례 ( Duck Yeii Choi ),박성규 ( Seong Kyu Park ),김배영 ( Bae Young Kim ),이승민 ( Seung Min Lee ),조윤주 ( Yun Joo Jo ) 대한소화기학회 2002 대한소화기학회 춘계학술대회 Vol.2002 No.-
〈목적〉 급성 췌장염은 임상적으로 경증에서 중증에 이르기까지 다양하면, 동반된 합병증으로 초기에 정확한 진단과 적절한 치료가 이루어져야 한다. 저자들은 급성 췌장염 환자에서 장기부전의 발생빈도를 알아보고, 장기부전이 예후에 미치는 영향을 평가하고자 하였다. 〈방법〉 본 연구에서 1996년부터 199년까지 4년간 을지의과 대학 병원에 내원하여 급성 췌장염으로 진단 받고 치료받았던 환자 187예를 대상으로 하였다. 이들은 남자 140예, 여자 47예였고,
포스터 전시 : 담도,췌장 ; 담도계에 발생한 NET 와 Small cell carcinoma
윤상정 ( Sang Jeong Yoon ),최덕례 ( Duck Yeii Choi ),박성규 ( Seong Kyu Park ),김배영 ( Bae Young Kim ),이승민 ( Seung Min Lee ),조윤주 ( Yun Joo Jo ) 대한소화기학회 2002 대한소화기학회 춘계학술대회 Vol.2002 No.-
<증례 1> 77세 여자가 3-4일 전부터 우상복 통증과 열감이있으면서 황달이 발생하여 본원에 내원하였다. 과거력상 2년전 본원에서 간내 담석 및 농양으로 치료 받은 병력이 없었다. 내원당시 환자는 만성병색을 보였고, 혈압110/70mmHg, 맥박 78회/분, 체온 36.9℃, 호흡수 20회/분이었다. 진찰소견상 우상복 압통과 반발통이있었고, 공막은 노랗게 관찰되었고 짙은 소변색을 보였다. 검사실 소견으로 말초혈액 검사에서 WBC 28,600/㎣, HB
저분자량 헤파린으로 치료된 급성 췌장염에 동반된 간문맥 혈전증
고훈 ( Hoon Ko ),정성희 ( Sung Hee Jung ),윤상정 ( Sang Jeong Yoon ),김안나 ( An Na Kim ),전병민 ( Byung Min John ),최기영 ( Gi Young Choi ),김준형 ( Jun Hyoung Kim ) 대한내과학회 2005 대한내과학회지 Vol.69 No.5
Portal vein thrombosis is an uncommon cause for presinusoidal hypertension, which results from inherited thrombotic disorder, neoplasm, and intra-abdominal inflammation like pancreatitis. It could develop portal hypertension, culminating in variceal bleeding from esophagus or stomach. One of the medical management of portal vein thrombosis is intravenous heparinization followed by long term oral anticoagulation. Intravenous heparinization using unfractionated heparin requires aPTT monitoring for dose adjustment which is not needed for low molecular weight heparin, and has higher risk of bleeding than using low molecular weight heparin. However, the standard protocol for anticoagulation in portal vein thrombosis has not been determined yet. We experienced a case of portal vein thrombosis in acute necrotizing pancreatitis, which was successfully treated with low molecular weight heparin, as herein reported.(Korean J Med 69:541-544, 2005)
김정념,이숭환,김정식,김진호,배윤오,박성규,윤상정,한현영,이헌영 충남대학교 의과대학 의학연구소 2003 충남의대잡지 Vol.30 No.1
Omental infarction, the end result of impaired perfusion to the greater omentum, is a rare benign self-limiting clinical entity. The main clinical symptom is non-specific localized abdominal pain with a moderately raised white blood cell and erythrocyte sedimentation rate. These findings often mimic an abdominal surgical emergency. This condition is often misdiagnosed as acute appendicitis or cholecystitis. The characteristic feature of CT scan and ultrasonography provide non-invasive diagnosis in most patients with omental infarction. We report a case of patient whose CT scan showed the characteristic finding of omental infarction. The patient was improved spontaneously only with conservative care.
고프롤락틴혈증과 동반된 부신피질자극 호르몬 단독 결핍 1 예
박강서,김대수,김영승,안전옥,윤상정,장희철,김광일 대한내분비학회 1997 Endocrinology and metabolism Vol.12 No.3
Isolated ACTH deficiency is a uncommon disorder causing secondary adrenocortical insufficiency. Less than 200 cases have been reported in the world. The major clinical manifestations are hypoglycemia, weight loss, hypotension, anemia, weakness, nausea, inability to excrete water load, and hyponatremia. A 56-year-old male was admitted because of weakness, fatigue, nausea and vomiting. He was pallor and not associated with hyperpigmentation. The basal plasma ACTH and cortisol levels were 8.30 pg/ml and 0.6 ug/dl. The serum cortisol did not response to rapid ACTH stimulation test. On combined pituitary stimulation test, the cortisol did not response to insulin-induced hypoglycemia. Other anterior pituitary hormones showed normal responses except elevated prolactin level. Six months after glucocorticoid replacement therapy, the elevated basal prolactin level returned to normal. Brain MRI did not show any anatomic abnormalities of the sellar and suprasella area. We report a case of isolated ACTH deficiency accompanied by hyperprolactineia, which respond to glucocorticoid replacement therapy. (J Kor Soc Endocrinol 12:462-467, 1997)