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외상성 뇌손상 환자에 있어서 S100β의 혈중 농도와 뇌손상의 정도 및 예후의 관계
김오현,이강현,윤갑준,박경혜,장용수,김현,황성오,Kim, Oh Hyun,Lee, Kang Hyun,Yoon, Kap Jun,Park, Kyung Hye,Jang, Yong Su,Kim, Hyun,Hwang, Sung Oh 대한외상학회 2007 大韓外傷學會誌 Vol.20 No.2
Purpose: $S100{\beta}$, a marker of traumatic brain injury (TBI), has been increasingly focused upon during recent years. $S100{\beta}$, is easily measured not only in cerebrospinal fluid (CSF) but also in serum. After TBI, serum S 10019, has been found to be increased at an early stage. The purpose of this study was to evaluate the clinical correlations between serum $S100{\beta}$, and neurologic outcome, and severity in traumatic brain injury. Methods: From August 2006 to October 2006, we made a protocol and studied prospectively 42 patients who visited the emergency room with TBI. Venous blood samples for $S100{\beta}$, protein were taken within six hours after TBI and vital signs, as well as the Glasgow Coma Scale (GCS), were recorded. The final diagnosis and the severity were evaluated using the Abbreviated Injury Score (AIS), and the prognosis of the patients was evaluated using the Glasgow Outcome Score (GOS). Results: Thirty-eight patients showed a favorable prognosis (discharge, recovery, transfer), and four showed an unfavorable prognosis. Serum $S100{\beta}$, was higher in patients with an unfavorable prognosis than in patients with a favorable prognosis, and a significant difference existed between the two groups ($0.74{\pm}1.50\;{\mu}g/L$ vs $7.62{\pm}6.53\;{\mu}g/L$ P=0.002). A negative correlation existed between serum $S100{\beta}$, and the Revised Traumatic Score (R2=-0.34, P=0.03), and a positive correlation existed between serum $S100{\beta}$, and the Injury Severity Score (R2=0.33, P=0.03). Furthermore, the correlation between serum $S100{\beta}$, and the initial GCS and the GCS 24 hours after admission to the ER were negative (R2=-0.62, P<0.001; R2=-0.47, P=0.005). Regarding the GOS, the mean serum concentration of $S100{\beta}$, was $7.62\;{\ss}{\partial}/L$ (SD=${\pm}6.53$) in the expired patients, $1.15\;{\mu}g/L$ in the mildly disable patient, and $0.727\;{\mu}g/L$ (SD=${\pm}0.73$) in the recovered patients. These differences are statistically significant (p<0.001). Conclusion: In traumatic brain injury, a higher level of serum concentration of $S100{\beta}$, has a poor prognosis for neurologic outcome.
이종영(Jong Young Lee),박경란(Kyung Ran Park),윤갑준(Kap Jun Yoon),홍인수(In Soo Hong) 대한두경부종양학회 1995 대한두경부 종양학회지 Vol.11 No.2
The effects of radiation therapy on salivary function of parotid gland were studied 20 male patients by radiation for tumors of head and neck. Saliva samples were collected before, during the radiotherapy and follow up. Parotid salivary function was quantitatively evaluated using a sialometry, sialochemistry, and scintigraphy. Salivary flow rates decreased in all individuals after irradiation. Glands than were partially irradiated were more likely to have some residual function than fully irradiated glands. Sodium and chloride contents were increased in parotid saliva of radiation patients where as potassium content was not changed definitively, pH was not changed during radiotherapy, but increased during follow up period. Scintigraphic stimulatory response rates showed similar result as flow rates. These results showed that exclusion of partial parotid gland from radiation is necessary to prevent severe xerostomia.
외상성 뇌손상 환자에 있어서 S100β의 혈중 농도와 뇌손상의 정도 및 예후의 관계
김오현 ( Oh Hyun Kim ),이강현 ( Kang Hyun Lee ),윤갑준 ( Kap Jun Yoon ),박경혜 ( Kyung Hye Park ),장용수 ( Yong Su Jang ),김현 ( Hyun Kim ),황성오 ( Sung Oh Hwang ) 대한외상학회 2007 大韓外傷學會誌 Vol.20 No.2
Purpose: S100β, a marker of traumatic brain injury (TBI), has been increasingly focused upon during recent years. S100β, is easily measured not only in cerebrospinal fluid (CSF) but also in serum. After TBI, serum S100β, has been found to be increased at an early stage. The purpose of this study was to evaluate the clinical correlations between serum S100β, and neurologic outcome, and severity in traumatic brain injury. Methods: From August 2006 to October 2006, we made a protocol and studied prospectively 42 patients who visited the emergency room with TBI. Venous blood samples for S100β, protein were taken within six hours after TBI and vital signs, as well as the Glasgow Coma Scale (GCS), were recorded. The final diagnosis and the severity were evaluated using the Abbreviated Injury Score (AIS), and the prognosis of the patients was evaluated using the Glasgow Outcome Score (GOS). Results: Thirty-eight patients showed a favorable prognosis (discharge, recovery, transfer), and four showed an unfavorable prognosis. Serum S100β, was higher in patients with an unfavorable prognosis than in patients with a favorable prognosis, and a significant difference existed between the two groups (0.74±1.50 μg/L vs 7.62±6.53 μg/L P=0.002). A negative correlation existed between serum S100β, and the Revised Traumatic Score (R2=-0.34, P=0.03), and a positive correlation existed between serum S100β, and the Injury Severity Score (R2=0.33, P=0.03). Furthermore, the correlations between serum S100β, and the initial GCS and the GCS 24 hours after admission to the ER were negative (R2=-0.62, P<0.001; R2=-0.47, P=0.005). Regarding the GOS, the mean serum concentration of S100β, was 7.62 ß∂/L (SD=±6.53) in the expired patients, 1.15 μg/L in the mildly disable patient, and 0.727 μg/L (SD=±0.73) in the recovered patients. These differences are statistically significant (p<0.001). Conclusion: In traumatic brain injury, a higher level of serum concentration of S100β, has a poor prognosis for neurologic outcome. (J Korean Soc Traumatol 2007;20:138-143)
간장 ( 肝臟 ) 및 담도 ( 膽道 ) : 알콜성 간질환에 있어서 혈청 Procollagen Type III Peptide 및 Laminin의 측정
권상옥(Sang Ok Kwon),이광훈(Kwang Hoon Lee),장우익(Woo Ik Jang),이동기(Dong Ki Lee),김호근(Ho Guen Kim),김현수(Hyun Soo Kim),윤갑준(Kap Jun Yoon) 대한소화기학회 1991 대한소화기학회지 Vol.23 No.1
N/A To evaluate if serum procollagen type III peptide (P-III-P) and laminin levels reflect the extent of liver fibrosis or inflammation, we have studied 67 patients with histologically proven alcoholic liver disease and 12 non-alcoholic control with normal liver function test. The result showed that P-III-P values were significantly elevated in the patients with alcoholic hepatitis (1.87 +- 2.01 U/ml), alcoholic chronic active hepatitis (1.16 +- 1.20) and alcoholic liver cirrhosis (1.26 +- 1.50) compared to fatty liver (90.76 +- 0.49), alcoholic hepatic fibrosis (0.68 +- 0.60) and healthy controls (0.67+- 0.34 U/ml) (p<0.05). When the cut-off value for P-III-P is set at 1.25 U/ml (mean of the fatty liver+2 SD), nearly all (94.1%) of the case with elevated values above the cut-off were alcoholic hepatitis, alcoholic chronic active hepatitis and liver cirrhosis. Furthermore, a close correlation (r= 0.60) was found between the serum P-III-P and laminin (p<0.001). Although P-III-P and laminin did not appear to be particularly useful for differentiating various types of alcoholic liver disease, their concentrations seemed to be related to a degree of hepatic inflammation. And it is evident from our finding that an unexpected increase in serum P-III-P and laminin values could indicate the presence of alcoholic hepatitis, alcoholic chronic active hepatitis and alcoholic liver cirrhosis.