http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
송태효 ( Song Tae Hyo ) 한국불어불문학회 2003 불어불문학연구 Vol.0 No.56
La proprie´te´ en tant que ce qui est impossible. C`est dans ces termes proble´matiques, non seulement en raison de notre syste`me de pense´e lie´ a` l`inaptitude de mettre en corre´lation la question de la proprie´te´ et celle de la logique, mais encore en vertu du champ d`investigation e´piste´mologique sous-jacent, que Proudhon a` la fois aborde et conclut ces re´flexions sur le proble`me de la proprie´te´. De ce fait ce proble`me majeur dans la pense´e de Proudhon ne nous autorise pas a` l`aborder par le biais pre´tendu socio-politique, et ceci a` l`encontre de l`approche de la dialectique mate´rialiste qui nous est en effet coutumie`re. Car il rele`ve de la me´taphysique elle-me^me que Proudhon tentait de populariser. Si d`un co^te´ l`approche me´taphysique de la question de la proprie´te´ constitue la rigueur implaccable et la force irre´sitible de la pense´e de Proudhon, de l`autre elle soule`ve pour nous la difficulte´ de la lecture de Proudhon du fait qu`elle est lie´e a` la the´ologie aussi bien qu`a` la logique. C`est pourquoi Proudhon a tout naturellement mis en rapport la proprie´te´ et le langage qui est le proble`me par excellence a` la fois de la the´ologe et de la logique. Ainsi Proudhon se conduit a` penser ces deux proble`mes a` partir de leur point commun qui est la conscience de la nuit, conscince qui s`ouvre comme malgre´ soi par le mot me^me d`impossible`.
剔出白鼠精管에서 Norepinephrine의 筋收縮效果에 미치는 Diphenhydramine의 影響
송태효,곽기철,곽용근,조규박,김용기 의과학연구소 1991 全北醫大論文集 Vol.14 No.4
Influences of H1-antihistaminics on the contractile response of isolated rat vas deferens to norepinephrine were studied in order to clarify the mechanisms of stimulant action of the drugs. 1. Lagre doses (10^-5M) of diphenhydramine and chlorpheniramine potentiated the contractile action of norepinephrine, but small deses (10^-7M) of them and the lage dose (10^-5M) of methdilazine suppressed the contractile action of norepinephrine. 2. Large dose (10^-5M) of diphenhydramine potentiated the contractile action of phenylephrine and isoproterenol. 3. The potentiating effect of diphenhydramine on contractile action of norepinephrine was partially inhibited by cocaine (3×10^-6M) or berapamil (10^-6M), and was completely blocked by KT362 (10^-4M). 4. Large dose of diphenhydramine inhibited the uptake of ^3H-norepinephrine, and increased the efflux of ^3H-norepinephrine. 5. Large dose of diphenhydramine increased the ^45Ca uptake. From the above results, it indicates that diphenhydramine has both potentiating and inhibiting effects on the contractile action of norepinephrine. And also, it is suggested that the potentiation of norepinephrine action by diphenhydramine is due to inhibition of amine pump as well as to increases of intracellular calcium both through increased Ca2+influx and release of calcium from intracellular storage site.
개구리 心臟에서 Ouabain 效果에 미치는 Serotonin의 影響
이태준,송태효,이태준 의과학연구소 1989 全北醫大論文集 Vol.13 No.2
The influence of 5-hdroxytryptamine and / or cyproheptadine on the action of ouabain was investigated in the isolated frog heart or in the whole fros. 1. LD_50 of ouabain was 2.88㎍/10g in control group. 2. Low doses(100 or 300 ㎍/10g) of 5-hydroxytryptamine increased LD_50 of ouabain, but high dose of the drug decreased LD_50 of ouabain. However, the effect of 5-hydroxytryptamine was blocked by cyproheptadine. 3. Ouabain induced dose-dependent biphasic response ; at low doses(10^-7-10^-6M) ouabain decreased contractile force of myocardium, while high doses of the drug(above 3X10^-6M) increased oncractile force as well as decrease of heart rate. 4. 5-hydroxytryptamine inhibited the negative inotrophic and bradycardiac effects of ouabain, but cyproheptadine did not affect the effects of 5-hydroxytryptamine. From the above results, it is suggested that there are two dose-related ouabain-sensitive sites in frog heart, and bradycardiac effect of ouabain is different from that of extracardiac regions.
實驗的 高血壓 白鼠의 心脈管系 機能調節에 있어서 中樞 Opiate System의 役割
김기원,송태효,조규박 의과학연구소 1989 全北醫大論文集 Vol.13 No.2
The possible involvement of central opiate system in the control of cardiovascular function and in the antihypertensive action of clonidine has been examined in unanesthetized rats with sham-operated or 2-kidney, 1-clip(2K1C) renal hypertension. In botn groups of rats, intraventricular clonidine(3-30㎍/kg) produced hypotension and bradycardia. Hypotensive action of clonidine was more potent in the hypertensive ratis than in the normotensive sham-operated rats. Yohimbine(30㎍/kg, i.v.t.) inhibited the hypotension and bradycardia produced by clonidine. Naloxone(50㎍/kg, i.v.t.) inhibited the action of clonidine in 2K1C hypertensive rats but not influenced in the sham-operated rats. Intraventricular morphine(10-100㎍/kg) also educed blood pressure and heart rate in both groups of rats. But these effects were not affected by yohimbine, but antagonized by naloxone(50㎍/kg, i.v.t.). Chronic treatment of 2K1C rats with clonidine(3×20㎍/kg, p.o., for 14days from 1 day after 2K1C operation) suppressed the development of hypertension and maintained the blood pressrue in normal level and this efect of clonidine was abolished by naloxone(2㎍/kg, i.p.). In the 2K1C hypertensive rats, immunoreactive β-endorphin content was significantly decreased, but maximum binding(Bmax) of [³H]-naloxone was significantly increase in brain of 2K1C hypertensive rats. However, Kd value was not changed. These results suggest that the opioidergic component might be involved in the antihypertensive action of clonidine only in hypertensive and that central opiate system might play important roles in pathophysiology of development and maintenance of hypertension.