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- 저자 : 김호중(Ho J oong Kim) (검색결과 3 건)
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김완중(Wan Jung Kim),김중남(Jung Nam Kim),김호중(Ho Jung Kim),문철웅(Chul Oong Moon),이승일(Soung Il Lee),장경식(Kyoung Sig Chang),홍순표(Soon Pyo Hong) 대한내과학회 1990 대한내과학회지 Vol.39 No.4
N/A Heart rate variability is a noninvasive index of the neural, especially parasympathetic, activity of the heart. Autonomic neuropathy is frequently observed in diabetic patients, and a 5-year survival rate of this kind of patient is low. To evaluate autonomic neuropathy in diabetic patients, 35 diabetic patients (proliferative retinopathy: 14, neurogenic bladder:8, postural hypotenion:6) measured for RR intervals and Valsalva ratios compared with 77 persons of the normal control group. RR variations (standard deviation: SD, coefficient of variation: CV, max-min, max/min) during deep respiration and Valsalva ratios (Vmax-Vmin, Vmax/Vmin) during Valsalva maneuver were significantly lower in the diabetic patients than in the normal control group (p<0.001, respectively). RR variations and Valsalva ratios decreased progressively with an increase of age in the control group and in diabetic patients (p<0.001, respectively). RR variations and Valsalva ratios were significantly lower in the diabetic patients with retinopathy, neurogenic bladder, and postural hypotension compared to diabetic patients without such conditions (P value <0.01, <0.05, <0.05, respectively). It is concluded that diabetic autonomic neuropathy can be indicated by the measurement of RR variations and Valsalva ratios.
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Hepatorenal Syndrome에 대한 Prostaglandin E1 유사화합물인 Misoprostol의 치료 효과
정철헌(Chul Hun Jung),김지훈(Ji Hoon Kim),이오영(Oh Young Lee),김정호(Jeong Ho Kim),김호중(Ho J oong Kim),함준수(Joon Soon Hahm),박경남(Kyung Nam Park),기춘석(Choon Suhk Kee) 대한소화기학회 1994 대한소화기학회지 Vol.26 No.3
N/A Hepatorenal syndrome is a functional renal impairrnent occuring mainly, but not exclusive- ly, in decompensated cirrhosis. Its pronosis is very poor. We studied to evaluate the effect of misoprostol (prostaglandin E, analogue) in patients with hepatorenal syndrome. We observed the therapeutic effect of oral misoprostol(0.4mg, four times per day) for 12 days in six pa- t.ients with hepatorenal syndrome who had oliguria and renal functional deterioration despite adequate supp)ement of fluid. The mean urinary output obtained over the 4 days preceding misoprostol administration was 263, 328, 275, 232, 265 and 356 ml per 24 hours, respectively, in the six patients, despite adequate volume expansion by plasma albumin and fresh frozen plasma. Diuresis increased t,o 906, 900, 625, 1085, 1495 and 1038 ml per 24hours, respectively, on day 1 12 after onset of therapy. In addition of diuresis, serum creatinine levels were 3.5, 2.6, 2.7, 4.5, 4.2 and 3.6mg/ dl before and changed to 2.0,1.9, 2.2, 5.0, 2.1 and 2.9mg/dl during treatment. Therefore, recov- ery of renal function in five patients were evident. Spot urine sodium concentration increased from average 30mEq/L to 40mEq/L in six cases before and during the treatment of misoprostol. In the present study, the oral administration of high dose of misoprostol in patients with hepatorenal syndrome seems to produce marked diuresis with recovery of renal failure, sug gesting again the role of prostaglandin in the pathogenesis of hepatorenal syndrome. There- fore, until further improvements in the management of hepatorenal syndrome in the future, administration of synthetic prostaglandin E,(misoprostol) may constitute an acceptable thera peutic intervention.(Korean J Gastroenterol 1994; 26: 498 505)
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문철웅,정종훈,박천국,이승일,배학연,장경식,김만우,정춘해,홍순표,이병래,김호중 朝鮮大學校 附設 醫學硏究所 1993 The Medical Journal of Chosun University Vol.18 No.1
Renal ischemia is one of the most common causes of acute renal failure. Four factors related to the pathogenesis of acute renal failure are vasoconstriction, decreased glomerular filtration rate, tubular back leak of filtrate, and intratubular obstruction. The cellular response to renal ischemic insults include decreased content of adenosine trihosphate, lipid peroxidation induced membrane degradation, alteration in cellular pH, and calcium or phospholipase induced mitochondrial dysfunction. Much attention has been given to the role of increased cellular calcium as a pathogenetic contributor to cell injury during ischemia. Author studied the protective effects of calcium antagonists on cellular injury during renal ischemia in rat. To investigate the protective role of these agents, author measured the amount of malondialdehyde(MDA) and the enzyme activities of free radical scarvengers-superoxide dismutase(SOD), catalase and glutathione peroxidase from non-pretreated group and calcium antagonists pretreated groups in control, ischemia and reflow subgroups. The results were summerized as follows: 1) The amount of MDA in non-pretreated group was higher in the reflow compared with the control(<p<0.01). But, in all pretreated groups, there was no statistically difference in the amount of MDA. 2) The SOD activity in non-pretreated group was lower in both the ischemia and the reflow compared with the control (P<0.05). But, in both verapamil and trifluoperazine-pretreated groups, there was no statistically difference in the SOD activity. 3) Both catalase and glutathione peroxidase activities in non-pretreated group were lower in both the ischemia and the reflow compared with the control (P<0.05). But in all pretreated groups, there was no statically difference in both catalase and glutathione peroxidase activities. These results suggest that free radical mediated ischemic injury by renal artery clamp in rat can be protected by intraperitoneal pretreatment with calcium antagonists. As trifluoperazine has a protective effect in renal ischemia, the calcium activated calmodulin dependent enzyme may play a role in renal ischemic injury.
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