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김국환,안영준,양은석,박상기,박영봉,문경래 朝鮮大學校 附設 醫學硏究所 1997 The Medical Journal of Chosun University Vol.22 No.2
Hypernatremia(serum sodium>150meq/L) is caused by conditions that produce an excessive gain of sodium or result in an excessive loss of body fluid that is greater than the loss of sodium. The prognosis for hypernatremic dehydration is poorer than isotonic or hypotonic dehydration, because severe hyperosmolarity may result in cerebral damage( seizures, cerebral hemorrhages, thromboses, and subdural effusions). This cerebral injury may result in a permanent neurologic deficit. We experienced two cases of hypernatremic dehydration. Theses cases may have resulted from faulty preparation of infant formulas, as with the use of condensed milk powder. The first case experienced seizures during treatment, but survived, yet the second case died in spite of intensive care.
경련환아의 뇌척수액내의 당과 혈당치에 미치는 인자에 대한 연구
김국환,문경래,양은석,박진,박동호,조남수,이경운,박상기 대한응급의학회 1997 대한응급의학회지 Vol.8 No.4
Background: In febrile convulsions, glucose concentrations are known to increase both in the blood and cerebrospinal fluid(CSF). The reason behind this increase is, however, not completely understood. We have studied the effects of convulsion and fever on the CSF and blood concentrations in four different groups of children: convulsive and nonconvulsive children, with and without fever. Methods: This study was retrospectively undertaken to review medical records of 167 children who had lumbar puncture at Chosun University Hospital during 2 years from January 1994 to December 1996. The blood samples for glucose determinations were taken just before the lumbar puncture. The effects of several variables (body temperature, duration of fever, convulsions) were analysed on glucose concentrations in the CSF and blood between the groups. Results: 1) The concentration of glucose in the CSF was significantly higher in convulsive children with(90.9±24.5mg/dl) and without fever(105±29.7mg/dl) than in non-convulsive children with (68.4±12.3mg/dl)and without fever(67.5±13.3mg/dl). 2) The concentration of glucose in the blood was not significant in the different groups. 3) There was no correlation between the body temperature and the concentration of glucose in the CSF(r=0.09, p>0.05). 4) A negative correlation between the duration of the fever and the concentration of glucose in the CSF was found in febrile children(r=0.26, p<0.05). Conclusions: Our results show that an increase in the CSF glucose concentration in children may be influenced more by a stress reaction, evoked by the seizure than increased body temperature.