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광음향분광법을 이용한 CO 산화반응에서의 $Gd_{1-x}Sr_xCoO_{3-y}$ 촉매효과 연구
강희석,엄가원,황진수,여철현,최중길,Kang Hee-Seok,Oum Ka-Won,Hwang Jin-Soo,Yo Chul-Hyun,Choi Joong-Gill 대한화학회 1993 대한화학회지 Vol.37 No.1
An application of the optoacoustic detection method to investigate the catalytic effect of a perovskite, $Gd_{1-x}Sr_xCoO_{3-y}$ (x = 0.00, 0.25, 0.50, 0.75) system, in the oxidation reaction of CO is described. The optoacoustic signals Of $CO_2$ produced from the oxidation reaction were measured for differing x values in $Gd_{1-x}Sr_xCoO_{3-y}$. By monitoring optoacoustic signals with respect to the time, the concentration ratios of CO and $O_2$, and the temperature, the kinetic information on the catalytic properties of the perovskite for CO oxidation reaction can be obtained. The effect of Sr substitution in $Gd_{1-x}Sr_xCoO_{3-y}$ has been found to show the maximum catalytic effect at x = 0.25 and the substantial increase in catalytic activity at temperatures above 200$^{\circ}C$. It demonstrates that the optoacoustic detection method allows the investigation of the integrated catalytic effect not only for the oxidation reaction of CO, but also for many reactions, in general, by continuously and directly detecting the species associated with the reactions. CO 산화반응의 촉매로 사용되는 페로브스카이트 금속산화물인 $Gd_{1-x}Sr_xCoO_{3-y}$ (x = 0.00, 0.25, 0.50, 0.75) 계의 촉매효과를 조사하기 위하여 광음향 분광법을 사용하였다. 산화반응으로부터 생성된 $CO_2$의 광음향신호를 $Gd_{1-x}Sr_xCoO_{3-y}$에서 각각의 x값에 대하여 반응시간, 반응기체인 CO와 $O_2$의 농도비, 그리고 온도에 따라 측정함으로써 CO 산화반응의 $Gd_{1-x}Sr_xCoO_{3-y}$ 촉매 특성에 대한 정보를 얻었다. Gd1-xSrxCoO3-y에서 Gd가 Sr로 치환됨에 따른 촉매효과는 x=0.25일 때 최대로 나타났으며 반응 온도에 대하여는 200$^{\circ}C$ 이상에서 급격히 증가하는 것을 알 수 있었다. $CO_2$ 레이저 광음향분광법은 반응에 대한 연속적인 측정을 가능케 함으로써 CO 산화반응의 촉매효과 연구뿐만 아니라 일반적인 반응속도 연구에도 응용할 수 있는 방법임이 확인되었다.
강희석(Hee seok Kang),이대훈(Dae Hoon Lee),송영훈(Young hoon Song),유상석(Sang seok Yu) 대한기계학회 2015 대한기계학회 춘추학술대회 Vol.2015 No.11
Study for utilization of landfill gas was proposed. Portion for existing landfill gas utilization for energy source remains under 10% that is too far considering potential value of the gas. Based on the domestic landfill gas condition, reforming process by rotating arc plasma was investigated. Conversion rate of CH₄ and CO₂ determined by the amount of energy input. The main products were H₂ and CO, and representative by-product was the C₂H₂. Ratio of H₂/CO of synthesis gas range was from 1.671 to 1.699. About 2 ㎾ of energy supply is expected to result in conversion of CH₄ above 90% where specific energy density is 17.6 kJ/L<SUB>CH4</SUB>.
사람의 신경교종 세포주에서 아데노바이러스 벡터를 이용한 p16/INK4a 유전자 전달에 의한 종양성장 억제
김미숙,권희충,강희석,박인철,이창훈,김창민,이춘택,홍석일,이승훈,Kim, Mi-Suk,Kwon, Hee-Chung,Kang, Hee-Seog,Park, In-Chul,Rhee, Chang-Hun,Kim, Chang-Min,Lee, Choon-Taek,Hong, Seok-Il,Lee, Seung-Hoon 대한신경외과학회 2000 Journal of Korean neurosurgical society Vol.29 No.4
Objective : p16/INK4a, a kind of tumor suppressor genes, encodes a specific inhibitor of the cyclin D-dependent kinases CDK4 and CDK6. This prevents the association of CDK4 with cyclin D1, and subsequently inhibits phosphorylation of retinoblastoma tumor suppressor protein(pRb), thus preventing exit from the G1 phase. According to previous reports, over 50% of glioma tissue and 80% of glioma cell lines have been demonstrated inactivation of p16/INK4a gene. The purpose of this study was to determine whether recombinant adenovirus-p16 virus is a suitable candidate for gene replacement therapy in cases of glioma. Methods : Three human glioma cell lines(U251MG, U87MG and U373MG) that express mutant p16 protein were used. Replication-deficient adenovirus was utilized as an expression vector to transfer exogenous p16 cDNA into the cells ; control cells were infected with the Ad-${\beta}$-gal expressing ${\beta}$-galactosidase. To monitor gene transfer and the expression of exogenous genes, we used Western Blotting analysis. Flow cytometry studies of cellular DNA content were performed to determine the cell cycle phenotype of the glioma cells before and after treatment. Results : We showed here that restoration of p16/INK4a expression in p16 negative U87MG, U251MG and partially deleted U373MG by Ad-CMV-p16 induced growth suppression in vitro. Flow cytometric study revealed that Ad-CMV-p16 infected U87MG cells were arrested during the G0-G1 phase of the cell cycle. Expression of p16 transferred by Ad-CMV-p16 in glioma cells was highly efficient and maintained for more than seven days. Conclusions : Our results suggest that Ad-CMV-p16 gene therapy strategy is potentially useful and warrants further clinical investigation for the treatment of gliomas.