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Stabilization of selenium cathodes via in situ formation of protective solid electrolyte layer
Lee, Jung Tae,Kim, Hyea,Nitta, Naoki,Eom, Kwang-sup,Lee, Dong-Chan,Wu, Feixiang,Lin, Huan-Ting,Zdyrko, Bogdan,Cho, Won Il,Yushin, Gleb The Royal Society of Chemistry 2014 Journal of Materials Chemistry A Vol.2 No.44
Using PCA to detect objects in moving background
Ting Ting Li(이팅팅),Rishu Gupia(리슈),Namseok Choi(최남석),Byung-Gook Lee(이병국) 한국멀티미디어학회 2009 한국멀티미디어학회 학술발표논문집 Vol.2009 No.2
Moving object detection is a basic component of many computer vision applications. It has a critical impact on the performance of visual surveillance and intelligent transportation. This paper proposes an adaptive moving object detection approach that can deal with dynamic scenes. This approach uses PCA to describe the background model. Experimental results demonstrate that the proposed method is able to detect moving objects under various types of dynamic scenarios.
Ting Shen,Jaehwi Lee,Myung Hwan Park,Yong Gyu Lee,Ho Sik Rho,Yi-Seong Kwak,Man Hee Rhee,Yung Chul Park,Jae Youl Cho 고려인삼학회 2011 Journal of Ginseng Research Vol.35 No.2
Ginsenoside (G) Rp₁ is a ginseng saponin derivative with anti-cancer and anti-inflammatory activities. In this study, we examined the mechanism by which G-Rp₁ inhibits inflammatory responses of cells. We did this using a strategy in which DNA constructs containing cyclooxygenase (COX)-2 and inducible nitric oxide synthase (iNOS) promoters were transfected into HEK293 cells. G-Rp₁ strongly inhibited the promoter activities of COX-2 and iNOS; it also inhibited lipopolysaccharide induced upregulation of COX-2 and iNOS mRNA levels in RAW264.7 cells. In HEK293 cells G-Rp₁ did not suppress TANK binding kinase 1-, Toll-interleukin-1 receptor-domain-containing adapter-inducing interferon-b (TRIF)-, TRIF-related adaptor molecule (TRAM)-, or activation of interferon regulatory factor (IRF)-3 and nuclear factor (NF)-κB by the myeloid differentiation primary response gene (MyD88)-induced. However, G-Rp₁ strongly suppressed NF-κB activation induced by Iκβ kinase (IKK)β in HEK293 cells. Consistent with these results, G-Rp₁ substantially inhibited IKKβ-induced phosphorylation of Iκβα and p65. These results suggest that G-Rp₁ is a novel anti-inflammatory ginsenoside analog that can be used to treat IKKβ/NF-κB-mediated inflammatory diseases.
Reappraising China’s One-Child Family Policy : Do Girls and Women Suffer or Benefit?
LEE, Yuen Ting 이화여자대학교 아시아여성학센터 2013 Asian Journal of Women's Studies(AJWS) Vol.19 No.4
Earlier studies on China’s one-child family policy have emphasized the suffering of girls and women excessively, referring to it as the dark side of the coin. However, this article presents an alternative perspective on the policy through critical analyses of China’s official documents, contemporary media reports, and other literature. By presenting the historical flow and social development of the policy, it concludes by identifying five major conflicts that have ensued as a result of the policy. The first is between the protection of women’s rights by law and discrimination against women by law. The second is between the old idea of son preference and the new idea of no preference. The third is between the good upbringing of only children and their being spoilt. The fourth conflict is between the idea that reproduction endangers women’s health and reproduction improves their health. The final one is between good career prospects for women and bad ones. 不少以往有關中國大陸「一孩政策」的研究,都過份強調婦女在此政策實施後所遭受的痛苦,正所謂事情的黑暗一面。然而,本文嘗試透過當年及現今的大陸官方文獻、傳媒的報道及相關學者的研究,進行批判性的分析並提出一個反差的看法。除了展現此政策的歷史進程及社會變遷外,結論還帶出它所隱藏著的五種緊張狀態,是值得我們注意的。第一,此政策能否保障抑或歧視婦女本身的合法權益?第二,此政策能否使父母更偏愛兒子抑或使其對子女同樣痛愛?第三,獨生子女在此政策實施後是否得到良好的教育抑或被溺愛?第四,此政策能否改善抑或危害婦女的生育健康?最後,此政策為婦女帶來良好的抑或不良的就業前景?
Calvarial Defect Healing Induced by Small Molecule Smoothened Agonist
Lee, Soonchul,Shen, Jia,Pan, Hsin Chuan,Shrestha, Swati,Asatrian, Greg,Nguyen, Alan,Meyers, Carolyn,Nguyen, Vi,Lee, Min,Soo, Chia,Ting, Kang,James, Aaron W. Mary Ann Liebert 2016 Tissue engineering. Part A Vol.22 No.23
<P>Hedgehog (Hh) signaling positively regulates both endochondral and intramembranous ossification. Use of small molecules for tissue engineering applications poses several advantages. In this study, we examined whether use of an acellular scaffold treated with the small molecule Smoothened agonist (SAG) could aid in critical-size mouse calvarial defect repair. First, we verified the pro-osteogenic effect of SAG in vitro, using primary neonatal mouse calvarial cells (NMCCs). Next, a 4mm nonhealing defect was created in the mid-parietal bone of 10-week-old CD-1 mice. The scaffold consisted of a custom-fabricated poly(lactic-co-glycolic acid) disc with hydroxyapatite coating (measuring 4mm diameterx0.5mm thickness). Treatment groups included dimethylsulfoxide control (n=6), 0.5mM SAG (n=7) or 1.0mM SAG (n=7). Evaluation was performed at 4 and 8 weeks postoperative, by a combination of high-resolution microcomputed tomography, histology (H & E, Masson's Trichrome), histomorphometry, and immunohistochemistry (BSP, OCN, VEGF). In vivo results showed that SAG treatment induced a significant and dose-dependent increase in calvarial bone healing by all radiographic parameters. Histomorphometric analysis showed an increase in all parameters of bone formation with SAG treatment, but also an increase in blood vessel number and density. In summary, SAG is a pro-osteogenic, provasculogenic stimulus when applied locally in a bone defect environment.</P>
Lee, Ah Young,Wu, Ting Ting,Hwang, Bo Ra,Lee, Jaemin,Lee, Myoung-Hee,Lee, Sanghyun,Cho, Eun Ju The Korean Society of Applied Pharmacology 2016 Biomolecules & Therapeutics(구 응용약물학회지) Vol.24 No.3
Neurodegenerative diseases are often associated with oxidative damage in neuronal cells. This study was conducted to investigate the neuro-protective effect of methanolic (MeOH) extract of Perilla frutescens var. japonica and its one of the major compounds, rosmarinic acid, under oxidative stress induced by hydrogen peroxide ($H_2O_2$) in C6 glial cells. Exposure of C6 glial cells to $H_2O_2$ enhanced oxidative damage as measured by 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide and thiobarbituric acid-reactive substance assays. The MeOH extract and rosmarinic acid prevented oxidative stress by increasing cell viability and inhibiting cellular lipid peroxidation. In addition, the MeOH extract and rosmarinic acid reduced $H_2O_2-indcued$ expression of inducible nitric oxide synthase (iNOS) and cyclooxygenase-2 (COX-2) at the transcriptional level. Moreover, iNOS and COX-2 protein expression was down-regulated in $H_2O_2-indcued$ C6 glial cells treated with the MeOH extract and rosmarinic acid. These findings suggest that P. frutescens var. japonica and rosmarinic acid could prevent the progression of neurodegenerative diseases through attenuation of neuronal oxidative stress.