http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
Kang, Beum-Kwan,Kim, Joo-Hyeong,Lee, Kyeong-Hee,Lim, Sang-Cheol,Ji, Jae-Jun,Lee, Jong-Won,Kim, Heung-Tae The Korean Society of Plant Pathology 2009 Plant Pathology Journal Vol.25 No.2
The survival of Colletotrichum acutatum was investigated in soil, infected fruits, and infected fruit debris incorporated into soil at several temperatures with different soil moisture levels. Samples were examined at 2-week intervals for 18 weeks to determine the survival of the pathogen based on the number of colony forming unit (CFU) of C. acutatum recovered on a semi-selective medium. C. acutatum conidia survived in both sterile and non-sterile soil at 4 and $10^{\circ}C$ for 18 weeks. If infected pepper fruits were completely dried, C. acutatum survived for 18 weeks at temperature from 4 to $20^{\circ}C$. Soil temperature and moisture affected the survival of C. acutatum in infected fruit debris incorporated into soil after air-drying. The effect of soil moisture on survival was weaker at low temperatures than at high temperatures. For up to 16 weeks, conidia were recovered from fruit debris in soil that had been kept at 4 to $20^{\circ}C$ and below 6% soil moisture. Conidia were recovered from fields until approximately 6 months after pepper fruits were harvested. Using PCR with species-specific primers and a pathogenicity test, we identified conidia recovered from soil and infected fruit from both the laboratory and field as C. acutatum and as the primary inoculum causing pepper anthracnose.
Addison씨 병 1예 : 저나트륨혈증성 뇌증을 동반한
강성구,이광우,김영인,손호영,차봉연,강무일,홍관수,김범생,안석주 대한내분비학회 1989 Endocrinology and metabolism Vol.4 No.1
Addison's disease is a rare disorder resulting from chronic deficiency of adrenal cortical hormone. Clinical manifestations are general weakness, weight loss, hyperpigmentation, hypovolemia with hyponatremia and hyperkalemia. The 42-year-old man has experienced slowly progressive weakness, weight loss and generalized pigmentation, especially sun exposed area, extensor surface and his nail bed since 1 year ago. On the time of admission, his main clinical manifestatons were bradykinesia, ataxia, tremor, slurred speech, mental confusion which was diagnosed and managed as Parkinsons disease at another hospital. His all neurologic symptoms are completely resolved with the replacement of adrenocortical hormones and the correction of hypovolemia. Here we experienced one case of Addisons disease with the typical signs of encephalopathy combined with severe hyponatremia. (J Kor Soc Endocrinol 4:66-71, 1989)
위부 갑상선 기능저하증 3예 - Type Ia 및 II
이영길,강성구,이광우,손호영,강무일,윤건호,손현식,유순집,홍관수,박영부,김범생 대한내분비학회 1991 Endocrinology and metabolism Vol.6 No.2
Pseudohypoparathyroidism (PHP) is a rare clinical state of hypoparathyroidism that results from target tissue resistance to parathyroid hormone, associated with a secondary, hypocalcemia-induced increase in parathyroid gland function. Classically PHP have clinical (tetany and seizures) and biochemical (hypocalcemia and hyperphosphatemia) features of hypoparathyroidism and AHO (Albright's hereditary osteodystrophy), characterized by several distinctive skeletal and developmental defects. But other variants were reported according to clinical and biochemical manifestation. The biochemical hallmark of PHP is PTH resistance. In some patients with PHP, the guanyl nucleotide regulatory protein (G-protein) is decreased by 50 percent in red blood cells in such patients. This defect appears to produce resistance to several other hormones that apparently exert their actions by stimulating the increased production of cellular cyclic AMP. Pseudohypoparathyroidism is classified to Type Ia, Type Ib and Type Ⅱ according to clinical and biochemical manifestations. We experienced recently three pseudohypoparathyroidism patients admitted to St. Mary's Hospital. One was classic PHP (type Ia), manifestated by tetany, hypocalcemia, hyperphosphatemia and AHO.The others were PHP without AHO (PHP type Ⅱ). Here we confirmed these patients to specific PHP subtype with the urinary cAMP response to PTH (J Kor Soc Endocrinol 6:170~178, 1991).
Yun-Sik Kim,Ji Young Min,Beum Kwan Kang,Ngyeun Van Bach,Woo Bong Choi,박은우,김흥태 한국식물병리학회 2007 Plant Pathology Journal Vol.23 No.3
The anthracnose disease on pepper fruits in Korea was caused by Colletotrichum acutatum as well as C. gloeosporioides. Since C. acutatum showed less sensitivity to benomyl, it was analyzed whether the less sensitivity was given by the same mechanism for the fungicide resistance of C. gloeosporioides. The isolates of C. acutatum were less sensitive to the three benzimidazole fungicides tested, benomyl, carbendazim, and thiophanate- methyl. However, the of C. acutatum isolates were different from the resistant isolates of C. gloeosporioides in their response to diethofencarb, one of N-phenylcarbamates; the former was still less sensitive to diethofencarb than the latter. The differences in the resistance mechanisms in two species were conspicuous in sequence analysis of the tub2 genes. The genes from C. acutatum did not show any non-synonymous base substitutions at the regions known to be correlated with the benzimidazole-resistance. All of these data may indicate that the less sensitivity of C. acutatum to benomyl is based on different mechanism(s) from that of C. gloeosporioides.
Kim, Yun-Sik,Min, Ji-Young,Kang, Beum-Kwan,Bach, Ngyeun-Van,Choi, Woo-Bong,Park, Eun-Woo,Kim, Heung-Tae The Korean Society of Plant Pathology 2007 Plant Pathology Journal Vol.23 No.3
The anthracnose disease on pepper fruits in Korea was caused by Colletotrichum acutatum as well as C. gloeosporioides. Since C. acutatum showed less sensitivity to benomyl, it was analyzed whether the less sensitivity was given by the same mechanism for the fungicide resistance of C. gloeosporioides. The isolates of C. acutatum were less sensitive to the three benzimidazole fungicides tested, benomyl, carbendazim, and thiophanate-methyl. However, the of C. acutatum isolates were different from the resistant isolates of C. gloeosporioides in their response to diethofencarb, one of N-phenyl-carbamates; the former was still less sensitive to diethofencarb than the latter. The differences in the resistance mechanisms in two species were conspicuous in sequence analysis of the tub2 genes. The genes from C. acutatum did not show any non-synonymous base substitutions at the regions known to be correlated with the benzimidazole-resistance. All of these data may indicate that the less sensitivity of C. acutatum to benomyl is based on different mechanism(s) from that of C. gloeosporioides.
Park, Jong-Kyu,Kim, Gyoung-Hee,Min, Gyung-Mi,Park, Hee-Jin,Hur, Jae-Seoun,Kang, Beum-Kwan,Kim, Heung-Tae,Choi, Woo-Bong,Koh, Young-Jin The Korean Society of Plant Pathology 2006 Plant Pathology Journal Vol.22 No.1
Anthracnose severely occurred on begonia (Begonia semperflorens Link.) nurseries in Gyeongju, Gyeongbuk in July, 2004. More than $80\%$ of begonia seedlings were diseased in the greenhouse surveyed and diseased leaves per plant were $12.1\%$ in average. Yellowish spots occurred on the leaves of begonia as initial symptoms, and they coalesced irregularly to form large brown pleomorphic lesions. Severely infected leaves were defoliated, resulting in abnormal growth of the entire plant. Colletotrichum sp. was repeatedly isolated from the diseased plants and was identified as Colletotrichum acutatum on the basis of the mycological characteristics on potato dextrose agar and RAPD analysis. Pathogenicity of the fungus was also confirmed by artificial inoculation on healthy plants. The optimum temperature for mycelial growth of C. acutatum was around $25^{\circ}C$. The fungus was sensitive to azoxystrobin, bitertanol, diethofencarb-carbendazim, difenoconazole and tebuconazole. This is the first report on the anthracnose of begonia caused by C. acutatum in Korea.
Jong Kyu Park,최우봉,김경희,고영진,김흥태,허재선,Gyung Mi Min,Hee Jin Park,Beum Kwan Kang 한국식물병리학회 2006 Plant Pathology Journal Vol.22 No.1
Anthracnose severely occurred on begonia (Begonia semperflorens Link.) nurseries in Gyeongju, Gyeongbuk in July, 2004. More than 80% of begonia seedlings were diseased in the greenhouse surveyed and diseased leaves per plant were 12.1% in average. Yellowish spots occurred on the leaves of begonia as initial symptoms, and they coalesced irregularly to form large brown pleomorphic lesions. Severely infected leaves were defoliated, resulting in abnormal growth of the entire plant. Colletotrichum sp. was repeatedly isolated from the diseased plants and was identified as Colletotrichum acutatum on the basis of the mycological characteristics on potato dextrose agar and RAPD analysis. Pathogenicity of the fungus was also confirmed by artificial inoculation on healthy plants. The optimum temperature for mycelial growth of C. acutatum was around 25oC. The fungus was sensitive to azoxystrobin, bitertanol, diethofencarb-carbendazim, difenoconazole and tebuconazole. This is the first report on the anthracnose of begonia caused by C. acutatum in Korea.