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법ㆍ제도적 변화에 따른 국민임대주택 부대복리시설 설치기준 및 적용현황 분석
강선주(Kang, Sunju),정윤남(Jeong, Yun-Nam),김세용(Kim, Seiyong) 한국도시설계학회 2012 도시설계 : 한국도시설계학회지 Vol.13 No.3
본 연구는 국내 국민임대주택 관련 법제상의 부대복리시설 설치기준 변화와 시기별 특성을 고찰하고, 설계 및 계획단계에서의 적용현황을 분석함으로써 관련 법제와의 연계성 및 실효성을 검토하였으며, 이를 위해 한국토지주택공사에서 최근 10년 동안 계획한 국민임대주택을 대상으로 부대복리시설 적용현황을 분석하였다. 연구 결과, 국민임대주택 부대복리시설의 설치기준 및 특성에 따라 세 시기로 구분할 수 있었으며, 정책적ㆍ시대적 흐름에 따른 변화 배경에 따라 시설 항목별 기준의 변화과정 및 특성을 파악할 수 있었다. 또한 설치현황 분석 결과, 최소규정을 만족하는 것 외에 관련 법제와의 유기적인 연계 및 거주자 특성에 대한 반영과 능동적 대응이 부족한 것으로 나타났다. 본 연구는 국민임대주택 부대복리시설 관련 법ㆍ제도의 변천 과정에 대한 고찰을 통해 시기별 특성 및 정책적 변화를 파악하고, 적용현황을 함께 분석함으로써 국민임대주택 부대복리시설의 설치기준에 대한 실증적인 현황 및 문제점을 고찰하였다는 점에서 의의를 가진다. This research considered the transitions in standards and time-period characteristics of national rental housing service facilities by examining the connectivity and effectiveness of policy reforms through an analysis of their applications in the design and planning phase. It analyzed service facilities planned for national rental housing by the Korea Land and Housing Corporation (LH) over the past ten years. Through time series analysis, three distinct phrases were drawn according to shifts in standards and applications of national rental housing service facilities. It was concluded that the changes in processes and of standards characteristics followed the political and periodical flows of the era. The analysis also found that there was insufficient input from the residents in implementing the policy changes, only barely satisfying the bare minimum legal code.
Cho, Minji,Choi, Hojoon,Nam, Seung Won,Kim, Sunju Korean Society of Environmental Biology 2021 환경생물 : 환경생물학회지 Vol.39 No.2
Unarmored dinoflagellates, in the family Kareniaceae, include harmful or toxic bloom-forming species, which are associated with massive fish kills and mortalities of marine organisms worldwide. The occurrence and distribution of the toxigenic species in the family Kareniaceae were investigated in the brackish and coastal waters of Korea between July 2018 and October 2020. During the survey, we collected seven newly recorded species; Karenia papilionacea, Karlodinium digitatum, Karl. veneficum, Karl. zhouanum, Takayama acrotrocha, T. helix, and T. tasmanica. A total of fifteen strains of the seven taxa were successfully established as clonal cultures and examined using LM, SEM, and molecular phylogeny inferred from LSU rDNA sequences. Herein, we present the taxonomic information, morphological features, and molecular phylogenetic positions of the unrecorded dinoflagellate species collected from Korean coastal waters.
Jeon, Boo Seong,Nam, Seung Won,Kim, Sunju,Park, Myung Gil The Korean Society of Phycology 2018 ALGAE Vol.33 No.1
Members of the family Parviluciferaceae (Alveolata, Perkinsozoa) are the well-known dinoflagellate parasitoids along with Amoebophrya ceratii species complex and parasitic chytrid Dinomyces arenysensis and contain six species across three genera (i.e., Parvilucifera infectans, P. sinerae, P. rostrata, and P. corolla, Dinovorax pyriformis, and Snorkelia prorocentri) so far. Among Parvilucifera species, the two species, P. infectans and P. sinerae, are very similar or almost identical each other morphologically and genetically, thereby make it difficult to distinguish between the two. The only main difference between the two species known so far is the number of sporangium wall (i.e., 2 layers in P. infectans vs. 3 layers in P. sinerae). During sampling in Masan bay, Korea during the spring season of 2015, the dinoflagellate Akashiwo sanguinea cells infected by the parasite Parvilucifera were observed and this host-parasite system was established in culture. Using this culture, its morphological and ultrastructural features with special emphasis on the variation in the number of sporangium wall over developmental times, were investigated. In addition, the sequences of rDNA regions and ${\beta}-tubulin$ genes were determined. The result clearly demonstrated that the trophocyte at 36 h was covered with 4 layers, and then outer layer of the sporocyte gradually degraded over time, resulting in wall structure consisting of two layers, with even processes being detached from 7-day-old sporangium with smooth surface, indicating that the difference in the number of layers seems not to be an appropriate ultrastructural character for distinguishing P. infectans and P. sinerae. While pairwise comparison of the large subunit rDNA sequences showed 100% identity among P. infectans / P. sinerae species complex, genetic differences were found in the small subunit (SSU) rDNA sequences but the differences were relatively small (11-13 nucleotides) compared with those (190-272 nucleotides) found among the rest of Parvilucifera species (P. rostrata and P. corolla). Those small differences in SSU rDNA sequences of P. infectans / P. sinerae species complex may reflect the variations within inter- strains of the same species from different geographical areas. Taken together, all morphological, ultrastructural, and molecular data from the present study suggest that they are the same species.
Kang, Seok-Min,Lim, Soyeon,Song, Heesang,Chang, Woochul,Lee, Sunju,Bae, Sang-mee,Chung, Ji Hyung,Lee, Hakbae,Kim, Ho-Gyoung,Yoon, Deok-Hyo,Kim, Tae Woong,Jang, Yangsoo,Sung, Jae-Mo,Chung, Nam-Sik,Hwan Elsevier 2006 european journal of pharmacology Vol.535 No.1
<P><B>Abstract</B></P><P>Myocardial oxidative stress and Ca<SUP>2+</SUP> overload induced by ischemia-reperfusion may be involved in the development and progression of myocardial dysfunction in heart failure. Xanthine oxidase, which is capable of producing reactive oxygen species, is considered as a culprit regarding ischemia-reperfusion injury of cardiomyocytes. Even though inhibition of xanthine oxidase by allopurinol in failing hearts improves cardiac performance, the regulatory mechanisms are not known in detail. We therefore hypothesized that allopurinol may prevent the xanthine oxidase-induced reactive oxygen species production and Ca<SUP>2+</SUP> overload, leading to decreased calcium-responsive signaling in myocardial dysfunction. Allopurinol reversed the increased xanthine oxidase activity in ischemia-reperfusion injury of neonatal rat hearts. Hypoxia-reoxygenation injury, which simulates ischemia-reperfusion injury, of neonatal rat cardiomyocytes resulted in activation of xanthine oxidase relative to that of the control, indicating that intracellular xanthine oxidase exists in neonatal rat cardiomyocytes and that hypoxia-reoxygenation induces xanthine oxidase activity. Allopurinol (10 μM) treatment suppressed xanthine oxidase activity induced by hypoxia-reoxygenation injury and the production of reactive oxygen species. Allopurinol also decreased the concentration of intracellular Ca<SUP>2+</SUP> increased by enhanced xanthine oxidase activity. Enhanced xanthine oxidase activity resulted in decreased expression of protein kinase C and sarcoendoplasmic reticulum calcium ATPase and increased the phosphorylation of extracellular signal-regulated protein kinase and p38 kinase. Xanthine oxidase activity was increased in both ischemia-reperfusion-injured rat hearts and hypoxia-reoxygenation-injured cardiomyocytes, leading to reactive oxygen species production and intracellular Ca<SUP>2+</SUP> overload through mechanisms involving p38 kinase and extracellular signal-regulated protein kinase (ERK) via sarcoendoplasmic reticulum calcium ATPase (SERCA) and protein kinase C (PKC). Xanthine oxidase inhibition with allopurinol modulates reactive oxygen species production and intracellular Ca<SUP>2+</SUP> overload in hypoxia-reoxygenation-injured neonatal rat cardiomyocytes.</P>