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THE <i>COSMIC INFRARED BACKGROUND EXPERIMENT</i> ( <i>CIBER</i> ): THE LOW RESOLUTION SPECTROMETER
Tsumura, K.,Arai, T.,Battle, J.,Bock, J.,Brown, S.,Cooray, A.,Hristov, V.,Keating, B.,Kim, M. G.,Lee, D. H.,Levenson, L. R.,Lykke, K.,Mason, P.,Matsumoto, T.,Matsuura, S.,Murata, K.,Nam, U. W.,Renbarg IOP Publishing 2013 The Astrophysical journal Supplement series Vol.207 No.2
Oh, K H,Yang, S W,Park, J M,Seol, J H,Iemura, S,Natsume, T,Murata, S,Tanaka, K,Jeon, Y J,Chung, C H Macmillan Publishers Limited 2011 CELL DEATH AND DIFFERENTIATION Vol.18 No.8
Apoptosis inducing factor (AIF) is a mitochondrial oxidoreductase that scavenges reactive oxygen species under normal conditions. Under certain stresses, such as exposure to N-methyl-N′-nitro-N′-nitrosoguanidine (MNNG), AIF is truncated and released from the mitochondria and translocated into the nucleus, where the truncated AIF (tAIF) induces caspase-independent cell death. However, it is unknown how cells decide to kill themselves or operate ways to survive when they encounter stresses that induce the release of tAIF. Here, we demonstrated that USP2 and CHIP contribute to the control of tAIF stability. USP2 deubiquitinated and stabilized tAIF, thus promoting AIF-mediated cell death. In contrast, CHIP ubiquitinated and destabilized tAIF, thus preventing the cell death. Consistently, CHIP-deficient cells showed an increased sensitivity to MNNG. On the other hand, knockdown of USP2 attenuated MNNG-induced cell death. Moreover, exposure to MNNG caused a dramatic decrease in CHIP level, but not that of USP2, concurrent with cell shrinkage and chromatin condensation. These findings indicate that CHIP and USP2 show antagonistic functions in the control of AIF-mediated cell death, and implicate the role of the enzymes as a switch for cells to live or die under stresses that cause tAIF release.
S. Ohnishi,K. Kondo,S. Sato,K. Ochiai,K. Takakura,C. Konno,I. Murata 한국물리학회 2011 THE JOURNAL OF THE KOREAN PHYSICAL SOCIETY Vol.59 No.23
A new collimator system was constructed to produce a new collimated DT neutron beam for new integral benchmark experiments at the first target room of the Fusion Neutronics Source facility in Japan Atomic Energy Agency. The collimator system had been designed and optimized with a neutron transport calculation code and the performance of the collimated DT neutron beam was tested with an imaging plate, activation foil and a scintillation counter. The DT neutron flux at the exit of the collimator hole was 2.22 × 10^6 cm^(-2)s^(-1), which was 239 times as large as that at the 2 cm off-centered position. It was confirmed that the new DT neutron beam had a good performance as expected.
R. Murata,T. Shirakawa,K. Umetani,E. Hiraki 전력전자학회 2019 ICPE(ISPE)논문집 Vol.2019 No.5
The high-frequency transformers used for isolated DC-DC converters tend to have large volume compared with the other components. Therefore, increasing the power density of the transformer is indispensable for increasing the power density of the DC-DC converter. Reducing the dead space of the transformer is one of the effective solutions. It is well-known that the thinly extended core structure reduces the dead space around the transformer formed by the windings protruding to the outside of the core. However, it has not been clarified how much the thinly extended core structure affects the reducing power loss of the transformer. In this paper, the effect of introducing the thinly extended core structure into the transformer is quantified by the optimization of the convertible design parameters of the transformer with systematic round-robin calculation. The result revealed the volume reduction effect by the thinly extended core was greater than the simply dead space reduction effect.
Nagatsuka, H,Murata, M,Shin, H.I,Choi, K.S,Nagai, N 경북대학교 의학연구소 1999 경북대학교병원의학연구소논문집 Vol.3 No.1
Verruciform xanthoma is an uncommon beniㅎn lesion of unknown aetiology and pathogenesis. Two cases of verruciform xanthoma are presented together with an immunohistochemical and ultra-structural study. One case co-occurred with an odontogenic keratocyst which, to our knowledge, has not been previously reported and another case occurred on masticatory mucosa with an asympto-matic, granular exophytic lesion causing a cleft-like gingival recession. In an immunohistochemical study using antibodies to macrophage (CD 68[KP1]), alpha-1-antitrypsin, vimentin, desmin, keratin, neuron-specific enolase (NSE) and S-100 protein, the characteristic foam cells of verruciform xanthoma showed strong positive staining far CD 68[KPl] and vimentin and weak positive staining for a1pha-1-antitrypsin, while the other antibodies were negative. As in previous studies, S-100 protein positive dendritic cells were detected in lesional connective tissue. In addition, the ultrastructural findings revealed characteristics of macrophages containing varying sized lipid vacuoles and degen-erating epithelial cells. These findings suppotr the concepts that the foam cells in verruciform xan-thoma are of monocyte-macrophage lineage and that the epithelial degeneration from uncertain causes may be related to the pathogenesis of the lesion. ⓒ1997 Elsevier Science Ltd