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손병호,이병헌,장지호,Sohn, Byung-Ho,Lee, Byung-Hyun,Jang, Ji-Ho 한국벤처창업학회 2006 벤처창업연구 Vol.1 No.1
Industry-academy Collaboration alms to produce synergy-empowerment through co-opt between university and business within R&D and human resource management. The lack of indigenous R&D capabilities makes Korean small businesses not have sustainable competitive advantages. While the Korean government has increased the budgetary supports on small businesses since the late 1990s to enhance technological capabilities of small businesses, the competitiveness gap between large and small firms has been widened. The alternative for the government support is considered as active industry-academy collaboration. After exploring various policy measures for the collaboration with classifying them and pointing out their problems from a technological innovation perspective, this study shows the more expected roles of the collaboration to be a catalyst coordinating the R&D activities among the local small businesses, local universities, and public research institutes. Based upon the analyses, this study suggests several policy alternatives to facilitate the technological innovations of small businesses in the regional innovation clusters.
복부수술 전 젖소초유의 경구투여가 내독소혈증과 TNF-α에 미치는 영향
서현일(Hyun Il Seo),전우규(Woo Kyu Jeon),강진호(Jin Ho Kang),임시영(Si Young Lim),이승세(Seung Sei Lee),손병호(Byung Ho Sohn),류창학(Chang Hak Ryu),신준호(Jun Ho Shin),김흥대(Hung Dai Kim),한원곤(Won Kon Han),오태윤(Tae Yun Oh),우희연(H 대한외과학회 2006 Annals of Surgical Treatment and Research(ASRT) Vol.70 No.6
쥐의 대동맥 동종이식 후 발생하는 내막 과형성의 형태학적 분석
김형태,손창용,조원현,손병호,박관규 대한혈관외과학회 1997 Vascular Specialist International Vol.13 No.2
Intimal hyperplasia, an abnormal migration and proliferation of vascular smooth muscle cells with associated deposition of extracellular connective tissue matrix, is a chronic structual changes occuring in denuded arteries, arterialized vein and prosthetic bypass graft. This is one of the most important cause of vascular graft failure within the first year after operation. Certain growth factors, particularly basic fibroblast growth factor, transforming growth factor-p, and platelet-derived growth factor, are believed to be the cause of the smooth muscle cell proliferation and migration. This smooth muscle cell proliferation and collagen deposition eventually produce intimal thickening with subsequent stenosis or occlusion of the vascular lumen. In order to evaluate the serial changes of injured vessel wall, aortic patch allograft was done in rat, and studied the morphological finding at 1 day, 1, 2, 6, and 8 weeks after graft. The results were summerized as follows; (1) During the early phase after graft, no significant wall changes were seen in the region of the anastomotic site except the presence of acute inflarnmatory cells with platelet aggregation and thrombus formation. (2) The intimal thickening was apparent by 1 week and was predominantly composed of smooth muscle cells. At the 2 weeks after graft, endothelial cells were partially regenerated to cover the patch graft, and intimal hyperplasia was composed of a mixture of smooth muscle cells and extracellular matrix, mostly collagen. (3) Six weeks after graft, prominent features were production and deposition of collagen rather than proliferation of smooth muscle cells. Reendothelialization over the thickened intima was seen at 8 weeks and the propagation of intimal hyperplasia to adjacent intima of normal vessel was also noted. In conclusion, intimal hyperplasia after vascular injury seemed to be a progressive response of the proliferation and migration of smooth muscle cells and this result might be used for further study about the suppression of intimal hyperplasia.