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효율적(效率的)인 자원순환사회 형성을 위한 자원재활용(資源再活用) 정책 고찰(考察)
류수호,Ryu, Su-Ho 한국자원리싸이클링학회 2009 資源 리싸이클링 Vol.18 No.2
녹색성장 정책의 핵심 단위 과제인 "온실가스 저감"과 "자원순환형 사회" 구축을 위해서는 폐기물 에너지회수와 물질재활용을 상호 보완적이고 균형적으로 실천하는 것이 중요하다고 할 것이다. 이에 본 연구는 과거 우리나라의 외국의 관련정책, "제4차 자원재활용 기본계획"과 "폐기물에너지화 종합대책"을 살펴보고 바람직한 자원재활용정책방향을 모색해 보았다. 그 결과 우리나라의 폐기물관리정책은 선진외국과 큰 차이가 없었으나 독일은 에너지회수를 중요하게 고려하고 고도 전처리를 의무화 하고 있는 점이 달랐으며, 우리나라의 여건은 과거보다 재찰용이 더욱 어려워질 것으로 판단되었다. "제4차 자원재활용 기본계획"에서는 필름류가 실질적이고 지속가능한 재활용이 되지 않고 있는 점을 알 수 있었으며, "폐기물에너지화" 종합대책에서는 RDF/RPF 에너지 회수가 일반소각 발전과 물질재활용에 비하여 효율성이 낮음을 알수 있었다. 이러한 문제를 해결하기 위해서는 에너지 및 폐열 회수는 에너지 회수(Recovery)로 개념 정립을 하며 전 과정평가(LCA: Life Cycle Assessment) 체계를 구축하는 등 제도적인 정비와 함께 필름류 합성수지를 용해하여 분자합성수지를 추출하는 기술 등의 기술개발도 필요함을 알수 있었다. To accomplish the greenhouse gas reduction which is over core unit project of the "Green growth" policy and "Resource circulation society", it is important to maintain proper balance and complement between energy recovery from waste and material recycling. This research(study) examined the related policies on the past of korea and foreign country, and also "The 4th resource recycling master plan" and "Energy recovery from waste plan" to provide advisable direction for resource recycling policy. The results of the research(study) showed that there were no significant difference between korea and developed foreign countries waste management policies. But in German policy, energy recovery from waste and pre-treatment are importantly considered and highly required for permission. Under current circumstance in korea, recycling will be more difficult than in the past. According to "The 4th resource recycling master plan", film type of synthetic resin was not sustainable recycled material in substance."Energy recovery from waste plan", proved that the energy recovery from RDF/RPF have lower efficiency than regular incineration generation and substance recycling. To solve these problems, the energy and remainder heat recovery must be generalized to "Energy recovery" concept and institutional improvement such as LCA(Life Cycle Assessment) system are need to support it. And also technology development to extract synthetic polymer by dissolved film type of synthetic resin must be provided.
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Barringtonia augusta Kurz 추출물의 항염증 및 항산화 효능 평가
류수호(Soo Ho Ryu),김민정(Min Jeong Kim),Tran The Bach,정성근(Sung Keun Jung) 한국식품과학회 2021 한국식품과학회지 Vol.53 No.2
본 연구에서는 LPS로 유도된 RAW 264.7 대식세포에서 Barringtonia augusta Kurz 추출물의 항염증, 항산화 효능을 확인하였다. BKE는 LPS에 의한 NO와 ROS의 생성을 유의적으로 억제하였고, iNOS 발현 또한 억제하였다. 특히 IKK 매개 NF-κB pathway에서, IKK의 인산화 억제를 통하여, IκBα의 감소와 NF-κB의 인산화를 억제하여, NF-κB의 세포질에서 핵으로의 이동을 유의적으로 억제하였다. 이 논문은 BKE가 NO와 ROS의 생성과 iNOS의 발현을 IKK매개 NF-κB 인산화 억제를 통해 항염증 및 항산화 효과를 나타냄을 보여주었다. 이러한 결과를 바탕으로 BKE는 항염증, 항산화 기능성 식품 또는 의약품 소재로써 활용가치가 높을 것으로 기대된다. Barringtonia augusta Kurz is a species of the genus Barringtonia. Although several studies have analyzed the biological activity of B. racemosa Roxb and B. acutangula, the anti-inflammatory and antioxidant effects of B. augusta extract (BKE) remain unclear. Therefore, in this study, we investigated the anti-inflammatory and antioxidant effects of BKE using lipopolysaccharide (LPS) and RAW 264.7. BKE suppressed LPS-induced nitric oxide (NO) and inducible NO synthase expression without affecting RAW 264.7 cell viability. Additionally, BKE showed 2,2-Diphenyl-1-picrylhydrazyl and 2,2-azino-bis(3-ethylbenzothiazoline-6-sulfonic acid) radical scavenging capacities and inhibited LPS-induced reactive oxygen species production in RAW 264.7 cells. BKE also suppressed LPS-induced phosphorylation of IκB kinase and nuclear factor kappa-B (NF-κB) and p65 translocation from the cytosol to the nucleus in RAW 264.7 cells. These results suggest that BKE is a possible novel material that exerts beneficial antioxidant and anti-inflammatory effects through the inhibition of NF-κB signaling pathways.
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Renal Protective Effects of Sparstolonin B in a Mouse Model of Sepsis
김채영,류수호,김나연,이원화,배종섭 한국생물공학회 2022 Biotechnology and Bioprocess Engineering Vol.27 No.2
Sparstolonin B (SsnB) exists in the tubers of Scirpus yagara and Sparganium stoloniferum, and it is known to modulate inflammatory mediators. Here, we investigated whether SsnB could reduce the renal functional damage in a cecal ligation and puncture (CLP)-induced septic mice. The effect of SsnB was measured via assessment of blood urea nitrogen (BUN), serum creatinine, total glutathione, lipid peroxidation, catalase activity, glutathione peroxidase activity, and superoxide dismutase activity. Our findings showed that SsnB treatment in mice with CLP-induced renal damage elevated the BUN and creatinine levels in plasma and protein levels in the urine. In contrast, the excessive production of nitric acid and induction of nitric oxide synthase were decreased. Moreover, SsnB inhibited the activation of nuclear factor-kappa B, reduced the plasma levels of tumor necrosis factor-α and interleukin-6, and thus reduced lethality. SsnB also increased lipid peroxidation and restored the levels of superoxide dismutase, glutathione peroxidase, and catalase in kidney tissues, thereby enhancing the antioxidant defense system. Conclusively, our results indicate that SsnB can protect mice from sepsis-induced renal injury.
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김채영,류수호,최혁재,박동호,배종섭 한국생물공학회 2022 Biotechnology and Bioprocess Engineering Vol.27 No.6
Particulate matter, with an aerodynamic diameter equal to or less than 2.5 μm (PM2.5), is an air pollutant that causes serious pulmonary injury when inhaled. Sparstolonin B (SsnB) from the Chinese herb, Sparganium stoloniferum inhibits the expression of inflammatory cytokines and is involved in survival pathways. We investigated the protective effects of SsnB against PM2.5-induced lung damage. PM2.5 was pretreated intranasally and 30 minutes later, SsnB was injected via a vein in the tail of mouse. The effects of SsnB on PM2.5-induced lung damages, barrier disruptive responses, and pulmonary inflammation and the underlying mechanism of SsnB were investigated. SsnB significantly reduced pathological lung injury, the lung wet/dry weight ratio, and the levels of permeability. It also considerably attenuated PM2.5-induced myeloperoxidase activity in lung tissue, reduced PM2.5-induced inflammatory cytokine levels, and suppressed PM2.5-induced lymphocytes in bronchial alveolar fluid. Moreover, SsnB increased the phosphorylation of the mammalian target of rapamycin (mTOR) and significantly inhibited the expression of PM2.5-stimulated toll-like receptor 2, 4 (TLR2, 4), MyD88, and autophagy-related proteins, LC3II and Beclin 1. We concluded that SsnB regulates both the TLR2, 4-MyD88 and mTOR-autophagy pathways, therefore SsnB can be used as a potential therapeutic agent for preventing PM2.5-induced pulmonary damage.
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