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A Case of Acquired Brown Syndrome after Surgical Repair of a Medial Orbital Wall Fracture
Il Hun Seo,Jay Won Rhim,Young Woo Suh,Yoon Ae A. Cho 대한안과학회 2010 Korean Journal of Ophthalmology Vol.24 No.1
A case of acquired Brown syndrome caused by surgical repair of medial orbital wall fracture is reported in the present paper. A 23-year-old man presented at the hospital with right periorbital trauma. Although the patient did not complain of any diplopia, the imaging study revealed a blow-out fracture of the medial orbital wall. Surgical repair with a calvarial bone autograft was performed at the department of plastic surgery. The patient was referred to the ophthalmologic department due to diplopia that newly developed after surgery. The prism cover test at distant fixation showed hypotropia of the right eye, which was 4 prism diopters (PD) in primary gaze, 20 PD in left gaze, while orthophoric in right gaze. Eye movement of the right eye was markedly limited on elevation in adduction with normal elevation in abduction with intorsion in the right eye present. Forced duction test of the right eye showed restricted elevation in adduction. Computerized tomography scan of the orbits showed the right superior oblique muscle was entrapped between the autografted bone fragment and posterior margin of the fracture. When repairing medial orbital wall fracture that causes Brown syndrome, surgeons should always be careful of entrapment of the superior oblique muscle if the implant is inserted without identifying the superior and posterior margin of the orbital fracture site.
추가적 내경계막 제거술과 액체가스교환술을 통한 지속성 황반원공의 재수술 3예
임원일(Won Il Rhim),구희찬(Hee Chan Ku),이은구(Eun Koo Lee) 대한검안학회 2009 Annals of optometry and contact lens Vol.8 No.1
목적: 유리체절제술 후 폐쇄되지 않고 지속된 황반원공 환자에게 추가적 내경계막 제거술과 액체가 스교환술을 통한 재수술 결과를 보고하고자 한다. 대상과 방법: 2기 황반원공 1안, 3기 황반원공 1안, 4기 황반원공 1안에서 유리체절제술과 내경계막제거술 및 액체가스교환술을 시행하였다. 1차 수술 후 황반원공이 폐쇄되지 않아 재수술을 시행하였다. 망막 혈관궁까지 내경계막을 추가로 넓게 제거하였으며 수술 종료 시 액체가스교환술을 시행하였다. 결과: 재수술 후 3안 모두 황반원공의 폐쇄가 이루어졌으며 시력의 호전을 보였다. 결론: 일차 수술 후 폐쇄되지 않은 황반원공 환자에서 추가적 내경계막 제거술과 액체가스교환술을 이용한 재수술을 통하여 해부학적 성공을 이루었기에 보고하는 바이다. Purpose: To report the result of retreatment using additional internal limiting membrane removal and intraocular gas tamponade for persistent macular holes after primary surgery. Methods: Three cases of macular hole (stage 1, stage 2, stage 3) were not closed despite primary macular hole surgery using internal limiting membrane removal and intraocular gas tamponade. Retreatment for the persistent macular hole consisted of additional broad internal limiting membrane removal reaching to the major retinal vascular arcade and intraocular gas tamponade. Results: All three cases showed macular hole closure and visual improvement after retreatment. Conclusions: We report the successful results of retreatment using additional broad internal limiting membrane removal and intraocular gas tamponade for persistent macular holes after primary surgery.
임원일(Won Il Rhim),구희찬(Hee Chan Ku),이은구(Eun Koo Lee) 대한검안학회 2010 Annals of optometry and contact lens Vol.9 No.1
목적: 백내장 수술 후 발생하는 급성 녹농균 안내염의 치료 방법에 대하여 고찰하고자 한다. 대상과 방법: 백내장 수술 후 급성 녹농균 안내염이 발생한 4안의 의무기록을 후향적으로 분석하였다. 결과: 같은 날 백내장 수술을 받은 3안에서 수술 후 1일째 녹농균 안내염이 발생하였으며, 수술 받은 순서대로 심한 양상을 보였다. 안저 검사가 불가능하였던 첫번째 환자는 유리체절제술과 유리체내 항생제 주입술을 시행하였으며 시력이 0.02로 호전되었다. 두번째, 세번째 환자는 유리체내 항생제 주입술만을 시행하였고 각각 시력이 0.9로 회복되었다. 다른 1안은 백내장 수술 후 2일째 녹농균 안내염이 발생하여 유리체절제술과 유리체내 항생제 주입술을 시행하고, 4주 후 증식유리체망막병증에 의한 망막박리와 저안압증이 발생하여 2차 유리체절제술을 시행하였다. 그 결과 안구를 유지할 수 있었으며 시력이 안전수동으로 호전되었다. 결론: 백내장 수술 후 급속한 진행을 보이는 급성 감염성 안내염은 발견 즉시 적절한 치료가 이루어져야 한다. Purpose: To discuss the treatment of acute postoperative endophthalmitis caused by Pseudomonas after cataract surgery. Methods: Retrospective review of four cases of acute postoperative Pseudomonas endophthalmitis after cataract surgery. Results: Acute postoperative endophthalmitis caused by Pseudomonas aeruginosa occurred in 3 cases who underwent cataract surgery on the same day at the same clinic. The first patient who had severe intraocular inflammation blocking fundus examination recovered visual acuity to 0.02 after vitrectomy and intravitreal antibiotics injection. The second and third patients recovered visual acuity to 0.9 after intravitreal antibiotics injection. In another patient who also had acute postoperative endophthalmitis caused by Pseudomonas aeruginosa. We performed vitrectomy and intravitreal antibiotics injection. In this case, secondary vitrectomy was performed for progressive retinal detachment and hypotony caused by proliferative vitreoretinopathy 4 weeks later. The eyeball was saved and the patient recovered visual acuity to hand movement. Conclusions: Immediate treatment is essential to preserve the eyeball and vision in cases of acute, rapid progressive postoperative endophthalmitis.