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Shuang Wang,Qian Yang,Zhi-Hua Liu,Lei Sun,Dan Wei,Jun-Zheng Zhang,Jin-Zhu Song,Yun Wang,Jia Song,Jin-Xia Fan,Xian-Xin Meng,Wei Zhang 한국미생물학회 2011 The journal of microbiology Vol.49 No.1
A moderately halophilic bacterial strain 15-13^T, which was isolated from soda meadow saline soil in Daqing City, Heilongjiang Province, China, was subjected to a polyphasic taxonomic study. The cells of strain 15-13^T were found to be Gram-negative, rod-shaped, and motile. The required growth conditions for strain 15-13^T were: 1-23% NaCl (optimum, 7%), 10-50°C (optimum, 35°C), and pH 7.0-11.0 (optimum, pH 9.5). The predominant cellular fatty acids were C18:1 ω7c (60.48%) and C16:0 (13.96%). The DNA G+C content was 67.6 mol%. Phylogenetic analysis based on 16S rRNA gene sequence comparisons indicated that strain 15-13^T clustered within a branch comprising species of the genus Halomonas. The closest phylogenetic neighbor of strain 15-13^T was Halomonas pantelleriensis DSM 9661^T (98.9% 16S rRNA gene sequence similarity). The level of DNA-DNA relatedness between the novel isolated strain and H. pantelleriensis DSM 9661^T was 33.8%. On the basis of the phenotypic and phylogenetic data, strain 15-13^T represents a novel species of the genus Halomonas, for which the name Halomonas alkalitolerans sp. nov. is proposed. The type strain for this novel species is 15-13^T (=CGMCC 1.9129^T =NBRC 106539^T).
Galectin-9 Acts as a Prognostic Factor with Antimetastatic Potential in Hepatocellular Carcinoma
Zhang, Zhao-Yang,Dong, Jia-Hong,Chen, Yong-Wei,Wang, Xian-Qiang,Li, Chong-Hui,Wang, Jian,Wang, Guo-Qiang,Li, Hai-Lin,Wang, Xue-Dong Asian Pacific Journal of Cancer Prevention 2012 Asian Pacific journal of cancer prevention Vol.13 No.6
Considerable research has been conducted concerning galectin-9 and carcinomas, but little information is available about any relation with the hepatocellular carcinoma. In this study, we employed a small interfering RNA (siRNA) targeting galectin-9 to down-regulate the expression in HepG2 cells. As a result, after galectin-9 expression was reduced, cell aggregation was suppressed, while other behaviour such as the proliferation, adhesion and invasion to ECM, cell-endothelial adhesion and transendothelial invasion of the cells were markedly enhanced. When tumors of 200 patients with hepatocellular carcinoma were tested for galectin-9 expression by immunohistochemistry, binding levels demonstrated intimate correlations with the histopathologic grade, lymph node metastasis, vascular invasion and intrahepatic metastasis (P<0.05). Moreover, survival analysis indicated that patients with galectin-9 expression had much longer survival time than those with negative lesions, and the Log-rank test indicated that this difference was statistical significant (P<0.0001). The Cox proportional hazards model suggested that negative galectin-9 expression in hepatocellular carcinoma represented a significant risk factor for patient survival. We propose that galectin-9 might be a new prognostic factor with antimetastatic potential in patients with hepatocellular carcinoma.
Wang Jing-Xuan,Yang Yong,Xian Wei,Li Qiu-Ying 한국강구조학회 2020 International Journal of Steel Structures Vol.20 No.5
When vertical load-bearing members of building structures are subjected to accidental loadings, such as an earthquake, fi re or explosion, the remaining structure chiefl y relies on the beam mechanism and catenary mechanism of the steel beam as the main resistance capacity, to prevent local or large-scale progressive collapse. This paper presents a three-dimensional fi nite element model of concrete-fi lled square steel tubular (CFST) column to steel beam joint with bolted–welded hybrid connection in a middle-column-removal scenario using ABAQUS software. The multi-scale modeling method was employed to calculate the collapse resistance and mechanism under vertical load. The vertical displacement and bearing capacity curve of the middle failure column was calculated and analyzed. The collapse mechanism and bearing capacity contribution of the steel beam tying in diff erent directions are also discussed. Results indicate that the failure mechanism of the process includes the beam mechanism, the translation mechanism, the catenary mechanism and the failure stage. After the short-span steel beam had been destroyed, the long-span steel beam continued to provide collapse resistance capacity. To improve progressive collapse resistance, the CFST column to steel beam joint with welded haunch is proposed in this paper. Comparing the anti-collapse bearing capacity and failure characteristics of the joint with the welded haunch and common joint indicates that the anti-collapse capacity of the joint with welded haunch can be improved, and the research results can be referenced for engineering progressive collapse design.
Wang, X L,Dou, S X,Ren, Zhi-An,Yi, Wei,Li, Zheng-Cai,Zhao, Zhong-Xian,Lee, Sung-IK IOP Pub 2009 Journal of physics, an Institute of Physics journa Vol.21 No.20
<P>We measured the initial <I>M</I>–<I>H</I> curves for a sample of the newly discovered superconductor NdFeAsO<SUB>0.82</SUB>Fe<SUB>0.18</SUB>, which had a critical temperature, <I>T</I><SUB>c</SUB>, of 51 K and was fabricated at the high pressure of 6 GPa. The lower critical field, <I>H</I><SUB>c1</SUB>, was extracted from the deviation point of the Meissner linearity in the <I>M</I>–<I>H</I> curves, which show linear temperature dependence in the low temperature region down to 5 K. The <I>H</I><SUB>c1</SUB>(<I>T</I>) indicates no s-wave superconductivity, but rather an unconventional superconductivity with a nodal gap structure. Furthermore, the linearity of <I>H</I><SUB>c1</SUB> at low temperature does not hold at high temperature, but shows other characteristics, indicating that this superconductor might have multi-gap features. Based on the low temperature nodal gap structure, we estimate that the maximum gap magnitude Δ<SUB>0</SUB> = (1.6 ± 0.2) <I>k</I><SUB>B</SUB><I>T</I><SUB>c</SUB>.</P>
Xian-Feng Gong,Min-Wei Wang,Shin-Ichi Tashiro,Satoshi Onodera,Takashi Ikejima 대한약학회 2005 Archives of Pharmacal Research Vol.28 No.1
Pseudolaric acid B is a major compound found in the bark of Pseudolarix kaempferi Gordon. In our study, pseudolaric acid B inhibited growth of human melanoma cells, A375-S2 in a timeand dose-dependent manner. A375-S2 cells treated with pseudolaric acid B showed typical characteristics of apoptosis including morphologic changes, DNA fragmentation, sub-diploid peak in flow cytometry, cleavage of poly-ADP ribose polymerase (PARP) and degradation of inhibitor of caspase-activated DNase (ICAD). P53 protein expression was upregulated while cells were arrested at the G2/M phase of the cell cycle. There was a decrease in the expression of anti-apoptotic Bcl-2 and Bcl-xL proteins, whereas pro-apoptotic Bax was increased. The two classical caspase substrates, PARP and ICAD, were both decreased in a time-dependent manner, indicating the activation of downstream caspases.
Xian Zhang,Xiaofei Fan,Yu Xue,Yantao Wang,Wei Cai 제어·로봇·시스템학회 2017 International Journal of Control, Automation, and Vol.15 No.2
This paper is concerned with the problem of the robust exponential passive filter design for uncertainneutral-type neural networks with time-varying mixed delays. Our aim is to design a Luenberger-type filter forestimating information about the neuron states, which is required in some applied areas. By constructing an appropriateLyapunov-Krasovskii functional and using the Wirtinger-based integral inequality to estimate its derivative,a delay-range-dependent and delay-rate-dependent criterion is presented to ensure the augmented filtering dynamicsystem to be robustly exponentially stable and passive with an expected dissipation. Since the criterion is presentedin the form of linear matrix inequalities with nonlinear constraints, a cone complementarity linearization algorithmis proposed to determine the filter gain from solution to the nonlinear problem. Finally, a numerical example isgiven to demonstrate the effectiveness of the proposed method.
Wei, Ling,Wang, Xing-Wu,Sun, Ju-Jie,Lv, Li-Yan,Xie, Li,Song, Xian-Rang Asian Pacific Journal of Cancer Prevention 2015 Asian Pacific journal of cancer prevention Vol.16 No.3
Mediator 19 (Med19) is a component of the mediator complex which is a coactivator for DNA-binding factors that activate transcription via RNA polymerase II. Accumulating evidence has shown that Med19 plays important roles in cancer cell proliferation and tumorigenesis. The involvement of Med19 in sensitivity to the chemotherapeutic agent cisplatin was here investigated. We employed RNA interference to reduce Med19 expression in human non-small cell lung cancer (NSCLC) cell lines and analyzed their phenotypic changes. The results showed that after Med19 siRNA transfection, expression of Med19 mRNA and protein was dramatically reduced (p<0.05). Meanwhile, impaired growth potential, arrested cell cycle at G0/G1 phase and enhanced sensitivity to cisplatin were exhibited. Apoptosis and caspase-3 activity were increased when cells were exposed to Med19 siRNA and/or cisplatin. The present findings suggest that Med19 facilitates tumorigenic properties of NSCLC cells and knockdown of Med19 may be a rational therapeutic tool for lung cancer cisplatin sensitization.
Mu, Xian-Min,Shi, Wei,Sun, Li-Xin,Li, Han,Wang, Yu-Rong,Jiang, Zhen-Zhou,Zhang, Lu-Yong Asian Pacific Journal of Cancer Prevention 2012 Asian Pacific journal of cancer prevention Vol.13 No.4
Background/Aim: Pristimerin isolated from Celastrus and Maytenus spp can inhibit proteasome activity. However, whether pristimerin can modulate cancer metastasis is unknown. Methods: The impacts of pristimerin on the purified and intracellular chymotrypsin proteasomal activity, the levels of regulator of G protein signaling 4 (RGS 4) expression and breast cancer cell lamellipodia formation, and the migration and invasion were determined by enzymatic, Western blot, immunofluorescent, and transwell assays, respectively. Results: We found that pristimerin inhibited human chymotrypsin proteasomal activity in MDA-MB-231 cells in a dose-dependent manner. Pristimerin also inhibited breast cancer cell lamellipodia formation, migration, and invasion in vitro by up-regulating RGS4 expression. Thus, knockdown of RGS4 attenuated pristimerin-mediated inhibition of breast cancer cell migration and invasion. Furthermore, pristimerin inhibited growth and invasion of implanted breast tumors in mice. Conclusion: Pristmerin inhibits proteasomal activity and increases the levels of RGS4, inhibiting the migration and invasion of breast cancer cells.