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선로용량 산정과 민감도 분석의 신뢰성 향상에 관한 연구
김무룡,김한신,이창호,김봉선,김동희,홍순흠,Kim Moo-Ryong,Kim Han-Xin,Lee Chang-Ho,Kim Bong-Sun,Kim Dong-Hee,Hong Soon-Hum 대한안전경영과학회 2005 대한안전경영과학회지 Vol.7 No.4
Line capacity calculation has been used to determine optimum efficiency and safe train service for train scheduling plan and investment priority order throughout detecting bottleneck section. Because of some problems of Yamagisi and UIC methods for line capacity calculation, developing of the method of line capacity caculation and evaluation for the Korea circumstance is important. This paper deals with the reliability improvement on the integrated system of TPS(Train Performance Simulator), PES(Parameter Evaluation Simulator), LCS(Line Capacity Simulator) and simulation and sensitivity analysis for line capacity.
Ergonomic Development and Evaluation of UI Design for Optical Brain- Machine Interface (OBMI) System
Xin Cui(최신아),Hayoung Jung(정하영),Halim Kim(김하림),Yang Xiaopeng(양샤오펑),Linqing Feng(펭린칭),Shaomin Zhang(장샤오민),Mingkang Li(리밍캉),Changhao Liu(리우창하오),Jiawei Han(한지아웨이),Ruixue Wang(왕루이수에),Heecheon You(유희천) 대한인간공학회 2020 대한인간공학회 학술대회논문집 Vol.2020 No.10
The TGFβ→TAK1→LATS→YAP1 Pathway Regulates the Spatiotemporal Dynamics of YAP1
Min-Kyu Kim,Sang-Hyun Han,Tae-Geun Park,Soo-Hyun Song,Ja-Youl Lee,이유섭,Seo-Yeong Yoo,Xin-Zi Chi,김응국,Ju-Won Jang,임대식,Andre J. van Wijnen,Jung-Won Lee,배석철 한국분자세포생물학회 2023 Molecules and cells Vol.46 No.10
The Hippo kinase cascade functions as a central hub that relays input from the “outside world” of the cell and translates it into specific cellular responses by regulating the activity of Yes-associated protein 1 (YAP1). How Hippo translates input from the extracellular signals into specific intracellular responses remains unclear. Here, we show that transforming growth factor β (TGFβ)-activated TAK1 activates LATS1/2, which then phosphorylates YAP1. Phosphorylated YAP1 (p-YAP1) associates with RUNX3, but not with TEAD4, to form a TGFβ-stimulated restriction (R)-point-associated complex which activates target chromatin loci in the nucleus. Soon after, p-YAP1 is exported to the cytoplasm. Attenuation of TGFβ signaling results in re-localization of unphosphorylated YAP1 to the nucleus, where it forms a YAP1/TEAD4/SMAD3/AP1/p300 complex. The TGFβ-stimulated spatiotemporal dynamics of YAP1 are abrogated in many cancer cells. These results identify a new pathway that integrates TGFβ signals and the Hippo pathway (TGFβ→TAK1→LATS1/2→YAP1 cascade) with a novel dynamic nuclear role for p-YAP1.
Solution-Processed n-Type Graphene Doping for Cathode in Inverted Polymer Light-Emitting Diodes
Kwon, Sung-Joo,Han, Tae-Hee,Kim, Young-Hoon,Ahmed, Towfiq,Seo, Hong-Kyu,Kim, Hobeom,Kim, Dong Jin,Xu, Wentao,Hong, Byung Hee,Zhu, Jian-Xin,Lee, Tae-Woo American Chemical Society 2018 ACS APPLIED MATERIALS & INTERFACES Vol.10 No.5
<P>n-Type doping with (4-(1,3-dimethyl-2,3-dihydro-1H-benzoimidazol-2-yl)phenyl) dimethylamine (N-DMBI) reduces a work function (WF) of graphene by similar to 0.45 eV without significant reduction of optical transmittance. Solution process of N-DMBI on graphene provides effective n-type doping effect and air-stability at the same time. Although neutral N-DMBI act as an electron receptor leaving the graphene p-doped, radical N-DMBI acts as an electron donator leaving the graphene n-doped, which is demonstrated by density functional theory. We also verify the suitability of N-DMBI-doped n-type graphene for use as a cathode in inverted polymer light-emitting diodes (PLEDs) by using various analytical methods. Inverted PLEDs using a graphene cathode doped with N-DMBI radical showed dramatically improved device efficiency (similar to 3.8 cd/A) than did inverted PLEDs with pristine graphene (similar to 2.74 cd/A). N-DMBI-doped graphene can provide a practical way to produce graphene cathodes with low WF in various organic optoelectronics.</P>
Causal relationship between the loss of RUNX3 expression and gastric cancer
Qing-Lin Li,Chohei Sakakura,Kosei Ito,Xin-Zi Chi,Lee, Kwang-Youl,Lee, Chang-Woo,Han, Sang-Bae,Kim, Hwan-Mook,Kim, Wun-Jae,Atsushi Kaneda,Toshikazu Ushijima,Yoshiaki Ito,Bae, Suk-Chul 이화여자대학교 세포신호전달연구센터 2002 고사리 세포신호전달 심포지움 Vol. No.4
The human runt-related gene RUNX3/PEBP2αC, located on chromosome 1p36, is a major mediator of signals elicited by members of the transforming growth factor-β(TGF-β) superfamily. Here we show that 45-60% of gastric cancer cell lines and surgically resected specimens do not significantly express RUNX3 due to a combination of hemizygous deletion and hypermethylation of the RUNX3 promoter region. Tumorigenicity of gastric cancer cell lines in nude mice was inversely related to their level of RUNX3 expression, and one gastric tumor associated mutation(R122C), occurring within the conserved Runt domain completely abolished the tumor suppressive effect of RUNX3. The results suggest that a lack of RUNX3 function is causally related to the genesis and progression of human gastric cancer.