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관동맥 질환 환자에서 Lipoprotein ( a ) 농도와 당내인성과 관련성
두영철(Young Cheoul Doo),최조영(Jo Young Choi),장명국(Myung Kuk Jang),홍성훈(Sung Hun Hong),장명준(Myeong Jun Chang),고순희(Soon Hee Koh),한규록(Kyoo Rok Han),오동진(Dong Jin Oh),유규형(Kyu Hyung Ryu),고영박(Young Bahk Koh),이영(You 대한내과학회 1996 대한내과학회지 Vol.51 No.4
Objectives: A raised Lp (a) lipoprotein concentration is associated with coronary artery disease and impaired glucose intolerance has also been shown to be predictive of coronary artery clisease in some studies. It has been suggested that there is a significant association between impaired glucose tolerance and increased circulating Lp (a) lipoprotein concentration. The object of this study is to determine whether glucose intolerance and raised Lp (a) concentration are associated in subjects with coronary artery disease. Methods: The study group comprised 60 patients with coronary artery disease (M:20, mean age 56+/-13 year) and 70 control subjects without coronary artery disease (M:15, mean age 58+/-10 year). We compared the clinical variables, lipid profile including Lp (a), fasting glucose, and fasting insulin in subjects with coronary artery disease with impaired glucose tolerance and normal glucose tolerance, and in controls. Results: 1) Nine of 60 patients (15%) with coronary artery disease had glucose intolerance. There were no significant difference in the incidence of cardiovascular risk factors, body mass index, left ventricular mass index, the levels of lipid including Lp (a), and the levels of fasting glucose, insulin, and C-peptide except in the incidence of smoking (48% in patients with coronary artery disease vs 24% in normal control, p<0.05) between patients with coronary artery disease and normal controls. 2) Between coronary artery disease patients group with and without glucose intolerance, and normal controls, there were no difference in the level of Lp (a) concentration. Conclusion: The level of fasting glucose and concentration of Lp (a) were no difference in between patients with coronary artery disease and normal controls. There was no difference in concentration of Lp (a) in patients of coronary artery disease with and without glucose intolerance, and so suggest that raised Lp (a) lipoprotein concentration are not responsible for the association between impaired glucose tolerance and coronary artery disease.
증례 / Vancomycin 에 의한 지연성 과민반응 1예 : 색전증을 동반한 심내막염 환자
김지수(Jee Soo Kim),박대균(Dae Gyun Park),고영박(Young Bahk Koh) 대한내과학회 1999 대한내과학회지 Vol.56 No.3
Vancomycin, one of glycopeptide antibiotics, has been used in recent years with the emergence of methicillin- resistant staphylococcus aureus (MRSA), coagulase negative staphylococci (CNS) as important hospital pathogens. A 75 years male patient receiving vancomycin 1g intravenously as twice daily dose for treatment of bacterial endocarditis, suffered from high fever, generalized diffuse erythematous maculopapular eruption, itching and eosinophilia, during course of 16th day of vancomycin therapy for treatment of bacterial endocarditis. This delayed hypersensitivity reaction was resolved with discontinuation of the drug and treatment with antihistamine. Awareness of vancomycin associated delayed hypersensitivity reactions is necessary during the treatment in patients with long-term infusion of vancomyin despite of delayed cutaneous reaction and fever associated with vancomycin therapy is not common.
박순옥,이영,홍경표,오동진,임종윤,고영박,유규형 대한내과학회 1990 대한내과학회지 Vol.39 No.4
It is known that mitral valve closure begins as a consequence of atrial contraction and is completed with ventricular contraction. If the atrial contraction is not supervened immediately by ventricular contraction, the mitral valve closure by atrial contraction is incomplete. At that time, diastolic mitral regurgitation can occur. We observed diastolic mitral regurgitation with Doppler echocardiography in 2 cases of 1st-degree atrioventricular (AV) block and in another case with 2nd-degree AV block (Wenckeback type). It means that diastolic mitral regurgitation can occur when the PR interval is prolonged. We conclude that mitral valve closure by atrial contraction is ineffective and is completed by ventricular contraction.