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홍은경(Eun Kyoung Hong),장명준(Myeong Jun Chang),강경우(Kyeong Woo Kang),전용철(Yong Cheol Jeon),이명석(Myung Seok Lee),김우중(Woo Joong Kim),심정원(Jung Weon Shim),이영철(Young Chul Rhee),정수영(Soo Yeong Jeong) 대한소화기학회 1996 대한소화기학회지 Vol.28 No.4
Xanthogranulomatous cholecystitis is an uncommon focal or diffuse destructive inflammatory lesion of the gallbladder. It is characterized macroscopically by yellowish tumor-like mass in the wall of the gallbladder, and microscopically characterized in the early stages by a large number of foamy histiocytes and acute inflammatory cells, and in the )ater stages by increasing fibrosis. It has been usually mistaken for gallbladder cancer and often seen with stones. A case is presented in which a 59-year-old woman with clinical gallbladder cholesterol polyp was found at surgery to have a edematous and thickened wall of gallbladder, multiple black pigmented stones and 3 x 0.5cm sized yellowish mass on gallbladder bed which was confirmed later as xanthogranulomatous cholecystitis, pathologically. (Korean J Gastroenterol 1996;28:582 - 586)
관동맥 질환 환자에서 Lipoprotein ( a ) 농도와 당내인성과 관련성
두영철(Young Cheoul Doo),최조영(Jo Young Choi),장명국(Myung Kuk Jang),홍성훈(Sung Hun Hong),장명준(Myeong Jun Chang),고순희(Soon Hee Koh),한규록(Kyoo Rok Han),오동진(Dong Jin Oh),유규형(Kyu Hyung Ryu),고영박(Young Bahk Koh),이영(You 대한내과학회 1996 대한내과학회지 Vol.51 No.4
N/A Objectives: A raised Lp (a) lipoprotein concentration is associated with coronary artery disease and impaired glucose intolerance has also been shown to be predictive of coronary artery clisease in some studies. It has been suggested that there is a significant association between impaired glucose tolerance and increased circulating Lp (a) lipoprotein concentration. The object of this study is to determine whether glucose intolerance and raised Lp (a) concentration are associated in subjects with coronary artery disease. Methods: The study group comprised 60 patients with coronary artery disease (M:20, mean age 56+/-13 year) and 70 control subjects without coronary artery disease (M:15, mean age 58+/-10 year). We compared the clinical variables, lipid profile including Lp (a), fasting glucose, and fasting insulin in subjects with coronary artery disease with impaired glucose tolerance and normal glucose tolerance, and in controls. Results: 1) Nine of 60 patients (15%) with coronary artery disease had glucose intolerance. There were no significant difference in the incidence of cardiovascular risk factors, body mass index, left ventricular mass index, the levels of lipid including Lp (a), and the levels of fasting glucose, insulin, and C-peptide except in the incidence of smoking (48% in patients with coronary artery disease vs 24% in normal control, p<0.05) between patients with coronary artery disease and normal controls. 2) Between coronary artery disease patients group with and without glucose intolerance, and normal controls, there were no difference in the level of Lp (a) concentration. Conclusion: The level of fasting glucose and concentration of Lp (a) were no difference in between patients with coronary artery disease and normal controls. There was no difference in concentration of Lp (a) in patients of coronary artery disease with and without glucose intolerance, and so suggest that raised Lp (a) lipoprotein concentration are not responsible for the association between impaired glucose tolerance and coronary artery disease.