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Study on α-cluster levels in non-4n nuclei using low-energy RI beams
Yamaguchi, H,Kahl, D,Hayakawa, S,Sakaguchi, Y,Abe, K,Nakao, T,Suhara, T,Iwasa, N,Kim, A,Kim, D H,Cha, S M,Kwag, M S,Lee, J H,Lee, E J,Chae, K Y,Wakabayashi, Y,Imai, N,Kitamura, N,Lee, P,Moon, J Y,Lee, IOP Publishing 2017 Journal of physics. Conference series Vol.863 No.-
Study of 19F Levels with Ex = 6.7 - 7.7 MeV by the 15N+α Scattering Experiment
D. Kim,G. W. Kim,S. Y. Park,A. Kim,한인식,K. Abe,O. Beliuskina,S. Hayakawa,N. Imai,N. Kitamura,Y. Sakaguchi,H. Yamaguchi,S. M. Cha,K. Y. Chae,M. S. Kwag,S. W. Hong,E. J. Lee,J. H. Lee,E. K. Lee,J. Y. Moo 한국물리학회 2018 THE JOURNAL OF THE KOREAN PHYSICAL SOCIETY Vol.73 No.3
The intense γ-rays from classical novae are emitted due to the β-decay of 18F which is produced by the Hot-CNO cycle. The reaction rate of 18F(p,α)15O affects the prediction of the abundance of 18F. The main contribution to this reaction rate is from the resonances in 19Ne near and above the proton threshold. Hence, many experiments and theoretical works have been performed on the low-lying 18F+p resonances. One of the methods for investigating 19Ne parameters is studying the mirror nucleus 19F. α elastic scattering experiment was performed with a 7 MeV/u 15N beam for investigating the resonance parameters in 19F using the thick target method in inverse kinematics at the radioactive beam facility CRIB of the Center for Nuclear Study, the University of Tokyo. The excitation function of 19F was obtained for the excitation energy Ex = 6.7 - 7.7 MeV, and the parameters of the resonance states in 19F were determined using an R-matrix analysis.
Experimental investigation of a linear-chain structure in the nucleus <sup>14</sup>C
Yamaguchi, H.,Kahl, D.,Hayakawa, S.,Sakaguchi, Y.,Abe, K.,Nakao, T.,Suhara, T.,Iwasa, N.,Kim, A.,Kim, D.H.,Cha, S.M.,Kwag, M.S.,Lee, J.H.,Lee, E.J.,Chae, K.Y.,Wakabayashi, Y.,Imai, N.,Kitamura, N.,Lee North-Holland Pub. Co 2017 Physics letters. Section B Vol.766 No.-
It is a well-known fact that a cluster of nucleons can be formed in the interior of an atomic nucleus, and such clusters may occupy molecular-like orbitals, showing characteristics similar to normal molecules consisting of atoms. Chemical molecules having a linear alignment are commonly seen in nature, such as carbon dioxide. A similar linear alignment of the nuclear clusters, referred to as linear-chain cluster state (LCCS), has been studied since the 1950s, however, up to now there is no clear experimental evidence demonstrating the existence of such a state. Recently, it was proposed that an excess of neutrons may offer just such a stabilizing mechanism, revitalizing interest in the nuclear LCCS, specifically with predictions for their emergence in neutron-rich carbon isotopes. Here we present the experimental observation of α-cluster states in the radioactive <SUP>14</SUP>C nucleus. Using the Be10+α resonant scattering method with a radioactive beam, we observed a series of levels which completely agree with theoretically predicted levels having an explicit linear-chain cluster configuration. We regard this as the first strong indication of the linear-chain clustered nucleus.
Yang, S.H.,Kawachi, H.,Khan, M.A.,Lee, S.Y.,Kim, H.S.,Ha, Jong K.,Lee, W.S.,Lee, H.J.,Ki, K.S.,Kim, S.B.,Sakaguchi, S.,Maruyama, S.,Yano, H. Asian Australasian Association of Animal Productio 2008 Animal Bioscience Vol.21 No.9
Leptin is produced by adipocytes and its role in the regulation of lipid metabolism, feed intake, productive and reproductive performance of domestic animal species has been greatly stressed and extensively investigated in recent years. This study was conducted to develop a radioimmunoassay (RIA) for the estimation of plasma bovine leptin and to determine plasma leptin concentration in fattening Japanese Black cattle (Wagyu) and its crossbreds at commercial farms. Relationships of plasma leptin with plasma vitamin A and age of crossbred cattle were also determined. Recombinant bovine leptin (rbleptin) was produced by the E. coli overexpressed leptin as a GST (glutathione S-transferase)-fusion protein. Then antiserum against bovine leptin was obtained by its immunization in rabbits. Using this antiserum, a bovine specific RIA was developed and plasma leptin level was determined in 120 crossbred fattening cattle (WagyuHolstein, 50:50) at commercial farms. The plasma leptin level increased with the age of cattle and its level was greater in the crossbred heifers than in the steers. Plasma vitamin A level was negatively correlated with plasma leptin level in crossbred heifers and steers. This relationship was stronger in heifers than in steers. Plasma leptin was gradually increased with advancing age in fattening Wagyu cattle. In conclusion, development of a bovine specific RIA to estimate plasma leptin will contribute to better understanding of the role of leptin in cattle.
Lee, Sun K.,Choi, Beom K.,Kim, Young H.,Kang, Woo J.,Kim, Kwang H.,Sakaguchi, Shimon,Suh, Jae H.,Kim, Tae Y.,Kwon, Byoung S. Blackwell Publishing Ltd 2006 Immunology Vol.119 No.4
<P>Summary</P><P>The glucocorticoid-induced tumour necrosis factor receptor family related gene (GITR) has been reported to be expressed on the activated T and CD4<SUP>+</SUP>CD25<SUP>+</SUP> regulatory T cells (Treg). Signalling triggered by GITR not only neutralizes the suppressive effect of Treg cells, but also augments activation, proliferation and cytokine production of effector T cells. To test the role of GITR in 2,4,6-trinitrobenzene sulphonic acid (TNBS)-induced colitis − a murine model of mucosal inflammation − TNBS-injected Balb/c mice were treated with agonistic anti-GITR monoclonal antibody (mAb). Anti-GITR treatment increased the death rate compared to rat IgG-treated mice. Typically, death occurred within 4 days after the TNBS injection when the mice were treated with anti-GITR. The mice that survived anti-GITR treatment suffered from severe inflammation in their entire intestines. CD4<SUP>+</SUP> T-depletion protected the mice from colitis; even an anti-GITR effect was not apparent. In contrast, CD8<SUP>+</SUP> T depletion showed less protective than did CD4<SUP>+</SUP> T depletion. Stimulation of GITR enhanced the production of proinflammatory cytokines including interferon (IFN)-&ggr;, tumour necrosis factor (TNF)-&agr;, interleukin (IL)-6 and IL-12. It also enhanced the humoral response such as serum levels of IgG<SUB>2b</SUB> and IgA, which was completely dependent on CD4<SUP>+</SUP> T cells. Taken together, this study demonstrated that GITR signalling on CD4<SUP>+</SUP> T cells is involved in the development and progress of colitis by enhancing both T helper type 1 (Th1) and Th2 type responses.</P>