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정명아,서유승,양진수,박준섭,윤진훈,이중건,이준승,이영규,김동희,조성범,주종은 대한신장학회 2002 Kidney Research and Clinical Practice Vol.21 No.2
Pelvic actinomycosis is a chronic granulomatous suppurative disease caused by an anaerobic grampositive organism Actinomyces israelii. It is com-monly associated with an intrauterine device(IUD) and can mimick pelvic or intra-abdominal malignant neoplasm. Ureteral obstruction leading to hydronephrosis is a rare complication of tubo-ovarian abscess. We experienced a case of hydronephrosis as a complication of pelvic actinomycotic abscess. The patient was a 46-year-old women presenting with fever and right flank pain. Leukocytosis and pyuria were present and a hydronephrosis was diagnosed by intravenous pyelography. Ultrasonography and a computerised tomography revealed a mass in right adnexum compressing the right ureter. Removal of retroperitoneal abscess and salphingo-oophorectomy were done and the diagnosis of actinomycosis was made by pathologic finding of resected mass. Postoperatively, the patient was treated with second-generation cephalosporin successfully. (Korean J Nephrol 2002;21(2):337-340)
Screening and Characterization of Microorganisms with Fibrinolytic Activity from Fermented Foods
(Myeong Ae Yu),(Gwan Sub Sim),(Seung Taek Kwon),(Jae Kwan Hwang),(Jung Kue Shin),(Ik Hyun Yeo),(Yu Rang Pyun),(Seon Joo Yoon) 한국미생물생명공학회 2002 Journal of microbiology and biotechnology Vol.12 No.4
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기관 삽관 중인 환자에서 Blind Protected Specimen Brushing의 역할
유희승 ( Yu Hui Seung ),홍지현 ( Hong Ji Hyeon ),윤장욱 ( Yun Jang Ug ),엄광석 ( Eom Gwang Seog ),이재명 ( Lee Jae Myeong ),김철홍 ( Kim Cheol Hong ),장승훈 ( Jang Seung Hun ),김동규 ( Kim Dong Gyu ),이명구 ( Lee Myeong Gu ),현인 대한결핵 및 호흡기학회 2003 Tuberculosis and Respiratory Diseases Vol.55 No.1
4-Gb/s의 속도로 동작하는 DBI 방식을 역으로 이용한 난수 특성 강화 회로
유승명(Seung-Myeong Yu),강윤하(Yunha Kang),안종찬(Jongchan An),김나현(Nahyun Kim),송준영(Junyoung Song) 대한전자공학회 2020 대한전자공학회 학술대회 Vol.2020 No.8
A scheme for improving the random number characteristic using reversed data bus inversion (RDBI) method is proposed in the 180nm CMOS technology. The RDBI designed to reduce the similarity between adjacent data and partiality of the output data. In this paper, the reversed hamming distance calculator is proposed to judge similarity and inverse data based on that judgment. The power consumption of core is 11.41 mW with 1.8―V supply at 4―Gb/s. The total area is 0.111 mm2.
Yu, Ha-Young,Lee, Sang-Min,Nam, Jae-Hoon,Lee, Seung-Joon,Fabrè,gue, Damien,Park, Myeong-heom,Tsuji, Nobuhiro,Lee, Young-Kook Elsevier 2017 Acta materialia Vol.131 No.-
<P>The objective of the present study was to elucidate the complicated interrelationship between necking, post-uniform elongation (e(pu)), strain rate sensitivity (SRS), fracture mechanism and Al concentration in Fe-18Mn-0.6C-xAl twinning-induced plasticity steels. Many tensile tests were conducted for in- and ex situ observations of necking, fracture surfaces, crack propagation and the density and size of micro-voids with the assistance of a high-speed camera and X-ray tomographic equipment. The addition of Al increased epu, SRS and reduction ratios in dimension of the neck part of tensile specimens, and also changed fracture mode from quasi-cleavage to ductile fracture at the edge part. The quasi-cleavage surface of Al-free specimen was induced by edge and side cracks occurring along grain boundary junctions and twin boundaries within the edges and side surfaces where local deformation bands meet. The ductile-fracture surface of 1.5 %Al-added specimen was formed by the coalescence of micro-voids. While the side-to-middle crack propagation occurred in Al-free and 1 %Al-added specimens due to side cracks, the middle-to-side crack propagation was observed in 1.5 %Al-added specimen. The Al-free specimen had the larger size of the 20 largest voids compared to the 1.5 %Al-added specimen despite its lower void density and local strain due to the accelerated growth of voids near the tips of side cracks. Evaluating the negligible epu of Al-free specimen by SRS is not deemed to be reasonable due to its inappreciable necking and side cracks. The improvement of epu in 1.5 %Al-added specimen is primarily due to disappearance of edge and side cracks. (C) 2017 Acta Materiali a Inc. Published by Elsevier Ltd. All rights reserved.</P>
Caveolin‐1 deficiency induces premature senescence with mitochondrial dysfunction
Yu, Dong‐,Min,Jung, Seung Hee,An, Hyoung‐,Tae,Lee, Sungsoo,Hong, Jin,Park, Jun Sub,Lee, Hyun,Lee, Hwayeon,Bahn, Myeong‐,Suk,Lee, Hyung Chul,Han, Na‐,Kyung,Ko, Jesang,Lee, Jae BLACKWELL PUBLISHING 2017 AGING CELL Vol.16 No.4
<P><B>Summary</B></P><P>Paradoxical observations have been made regarding the role of caveolin‐1 (Cav‐1) during cellular senescence. For example, caveolin‐1 deficiency prevents reactive oxygen species‐induced cellular senescence despite mitochondrial dysfunction, which leads to senescence. To resolve this paradox, we re‐addressed the role of caveolin‐1 in cellular senescence in human diploid fibroblasts, A549, HCT116, and Cav‐1<SUP><I>−/−</I></SUP> mouse embryonic fibroblasts. Cav‐1 deficiency (knockout or knockdown) induced cellular senescence via a p53‐p21‐dependent pathway, downregulating the expression level of the cardiolipin biosynthesis enzymes and then reducing the content of cardiolipin, a critical lipid for mitochondrial respiration. Our results showed that Cav‐1 deficiency decreased mitochondrial respiration, reduced the activity of oxidative phosphorylation complex I (CI), inactivated SIRT1, and decreased the NAD<SUP>+</SUP>/NADH ratio. From these results, we concluded that Cav‐1 deficiency induces premature senescence via mitochondrial dysfunction and silent information regulator 2 homologue 1 (SIRT1) inactivation.</P>
Seung-Hyun Yu,Myeong-Jun Kim,Jin-Jeon,Hoon-Ki Park,Hwan-Sik Hwang,Kye-Yeung Park 대한가정의학회 2019 Korean Journal of Family Medicine Vol.40 No.6
Background: Although the number of medical institutions running a smoking cessation clinic is on the rise, there remains a paucity of research on the long- and short-term success rates of smoking cessation programs, as well as on smoking relapse rates, before and after project implementation. This study assessed the general characteristics of patients visiting the smoking cessation clinic, success rate of smoking cessation in the short term, and risks of relapse. Methods: Medical records from March 2015 to April 2017 were analyzed and telephone surveys were conducted with 151 smokers who visited a hospital smoking cessation clinic from March 2015 to April 2017. Results: Of the 139 smokers who were eligible for follow-up, 22 (15.8%) failed to quit smoking initially. The clinic’s 6-month success rate of smoking cessation was 64.83%. Those with higher medication compliance had a lower risk of primary failure (odds ratio, 0.056; 95% confidence interval, 0.005–0.609), whereas those with higher age (hazard ratio [HR], 0.128; P=0.0252) and a greater number of visits to the clinic (HR, 0.274; P=0.0124) had a lower risk of relapsing. Conclusion: The risk of primary failure to quit was higher with low medication compliance, and that of relapsing was higher with lower age and fewer number of clinic visits. Various evaluation and analysis methods can be carried out in the future based on the accumulated data for maintenance of smoking cessation and relapse prevention.
( Myeong-eun Jegal ),( Seung-youn Jung ),( Yu-seon Han ),( Yung-jin Kim ) 생화학분자생물학회(구 한국생화학분자생물학회) 2019 BMB Reports Vol.52 No.5
Hepatitis B virus (HBV) encoding the HBV x protein (HBx) is a known causative agent of hepatocellular carcinoma (HCC). Its pathogenic activities in HCC include interference with several signaling pathways associated with cell proliferation and apoptosis. Mutant C-terminal-truncated HBx isoforms are frequently found in human HCC and have been shown to enhance proliferation and invasiveness leading to HCC malignancy. We investigated the molecular mechanism of the reduced doxorubicin cytotoxicity by C-terminal truncated HBx. Cells transfected with C-terminal truncated HBx exhibited reduced cytotoxicity to doxorubicin compared to those transfected with full-length HBx. The doxorubicin resistance of cells expressing C-terminal truncated HBx correlated with upregulation of the ATP binding cassette subfamily B member 1(ABCB1) transporter, resulting in the enhanced efflux of doxorubicin. Inhibiting the activity of ABCB1 and silencing ABCB1 expression by small interfering ribonucleic acid (siRNA) increased the cytotoxicity of doxorubicin. These results indicate that elevated ABCB1 expression induced by C-terminal truncation of HBx was responsible for doxorubicin resistance in HCC. Hence, co-treatment with an ABCB1 inhibitor and an anticancer agent may be effective for the treatment of patients with liver cancer containing the C-terminal truncated HBx. [BMB Reports 2019; 52(5): 330-335]