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이식 : 높은 Panel Reactive Antibody를 가진 신이식 환자에서 흐름 세포측정 교차반응과 항체 추적 시스템의 비교
황현석 ( Hyeon Seok Hwang ),형복진 ( Bok Jin Hyoung ),이소영 ( So Young Lee ),전연주 ( Youn Joo Jeon ),윤혜은 ( Hye Eun Yoon ),김진영 ( Jin Young Kim ),최범순 ( Bum Soon Choi ),양철우 ( Chul Woo Yang ),김용수 ( Yong Soo Kim ),김 대한신장학회 2008 춘계학술대회 초록집 Vol.28 No.1
폐암과 병발한 류마티스 관절염 환자에서 항암요법에 따른 임상경과
최선욱 ( Son Ook Choi ),정재규 ( Jae Gu Jung ),형복진 ( Bok Jin Hyoung ),박상미 ( Sang Mi Park ),정현정 ( Hyun Jung Jung ),박보형 ( Bo Hyoung Park ),심병용 ( Byoung Yong Shim ),김완욱 ( Wan Uk Kim ),김훈교 ( Hoon Kyo Kim ) 대한류마티스학회 2006 대한류마티스학회지 Vol.13 No.2
A 54-year-old male was admitted due to lung cancer and polyarthralgia involving wrist, hand, shoulder, and ankle joints. Five months ago, he had been diagnosed as adenocarcinoma of the lung, and treated with three cycles of chemotherapy using gemcitabine and cisplatin. In the course of chemotherapy, he had complained symmetrical polyarthralgia of hand and shoulder joints, resembling rheumatoid arthritis (RA). After treatment with chemotherapeutic agents, he still had severe polyarthritis refractory to anti-rheumatic drugs, including prednisolone, hydroxylchloroquine, and methotrexate, and thus referred to our hospital. We changed the previous anti-cancer regimens to cisplatin plus docetaxel, a semisyntheic taxane molecule, which is known to suppress experimental polyarthritis. With additional three cycles of cisplatin plus docetaxel, RA disease activity as well as polyarthralgia was nearly completely resolved, and the extent of lung cancer was not aggravated. Although RA patients have an increased risk of malignancy, the outbreak of RA was very rare in lung cancer patients. Here we report a case of coincident lung cancer and rheumatoid arthritis, which was successfully treated by docetaxel plus cisplatin chemotherapy.
송준창 ( Joon Chang Song ),황현석 ( Hyeon Seok Hwang ),형복진 ( Bok Jin Hyoung ),이소영 ( So Young Lee ),전연주 ( Yeon Joo Jeon ),장세나 ( Se Na Chang ),윤혜은 ( Hye Eun Yoon ),최범순 ( Bum Soon Choi ),김용수 ( Yong Soo Kim ),양 대한신장학회 2009 Kidney Research and Clinical Practice Vol.28 No.1
After renal transplantation, we are more likely to encounter hyperkalemia rather than hypokalemia. We report a case of kidney transplantation recipient with hypokalemia and hypertension secondary to primary aldosteronism. A 48 year-old woman was presented with fatigue and weight loss that had lasted for 3 months. She was diagnosed as autosomal dominant polycystic kidney disease that ultimately progressed to end-stage renal disease. She was operated for renal transplantation before 6 months. She had hypokalemia and hypertension at that time. The ratio of plasma aldosterone over plasma renin activity was 851.7. The computed tomography (CT) revealed 2.4×1.7 cm sized adrenal mass on the right side. The pre-transplantation CT also showed that there had been adrenal mass in the same location even before the transplantation. Right adrenalectomy was performed. After she got discharged, she was again presented with nausea and vomiting. She developed hyperkalemia and was diagnosed as hyporeninemic hypoaldosteronism. She was prescribed with fludrocortisones and recovered from the disease, and resumed the state of normokalemia and normotension.