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이가영(Gayoung Lee ),김우선(Denis Woo-Seon Kim) 비판사회학회 2017 경제와 사회 Vol.- No.116
이 글은 국제자원활동이 참여 청년들에게 끼친 재(탈)영토화된 삶과 정체성의 변화를 연구한다. 청년들의 국제자원활동에 대한 기존 연구는 참여자의 동기나 만족도, 프로그램의 효율성에 집중되어 있기에, 활동으로 말미암은 정체성의 변화는 기존 연구가 간과하고 있는 주제이다. 국제자원활동은 한국과 소임 국가를 가로지르고 연결하는 초국적 사회적 장에서 전개되기에, 이들은 외국으로의 이주와 활동과 귀환을 통해서 문화 혼종과 탈영토화/재영토화를 체험하게 된다. 이 연구는 자원활동 후 귀환한 청년들의 심층면접을 통해서 그들의 재영토화과정을 한국형, 현지형, 세계시민형, 유목형으로 유형화하였다. 이 논의는 귀환 청년들이 갖는 정체성의 다양한 유형을 보임으로써, 동질적인 한국적 정체성의 유지라는 가정에 대해 의문을 제기하고, 내부로부터 진행되는 주관적 세계화에 대한 관심을 촉구한다. The article examines the impact of the young Korean volunteers’ experience in the international development upon their identity formation. Their experience entails not only the departure from and the return to their home country, but also the life and work in the transnational field. Hence, they have to face cultural hybridization and de(re)-territorialization. This article explores how this experience influences upon the identity formation and thereby identifies the four different paths of returnees’ identity as ‘Korean,’ ‘the assigned country,’ ‘cosmopolitan’ and ‘nomad’ identity. This finding challenges the assumption of a continuity of a homogeneous Korean identity and calls for attention to the gsubjective globalization of Korean society from within.
Cho, Sun Young,Lim, Gayoung,Oh, Seung Hwan,Lee, Hee Joo,Suh, Jin-Tae,Lee, Juhie,Lee, Woo-In,Lee, Hong Ghi,Yoon, Hwi-Joong,Park, Tae Sung Institute for Clinical Science] 2011 Annals of clinical and laboratory science Vol.41 No.3
<P>Because plasma cell leukemia (PCL) is a rare and distinct variant among plasma cell dyscrasias, recent clinical and cytogenetic studies have been performed in different ethnic groups to define the characteristics of these PCL patients. As far as we know, IGH rearrangements involving t(11;14) and (14;16) are significantly more frequent in PCL than in myeloma patients. However, PCL cases associated with t(6;14)(p21;q32) or IGH-CCND3 rearrangement are extremely rare in the literature; only one PCL case with t(6;14) has been documented. A 61-year-old female was admitted due to fatigue, weight loss, and exertional dyspnea. Plasmacytoid cells were counted up to 76% at a peripheral blood film, but bone marrow aspiration failed because of dry-tapping. Flow cytometric analysis showed positive for CD138 and cytoplasmic kappa light chain. Chromosome analysis revealed t(6;14)(p21;q32), which was confirmed by an IGH split-out probe in FISH analysis. Immunofixation electrophoresis also presented monoclonal bands identified as IgG and kappa light chain. Finally, she was diagnosed as primary PCL associated with t(6;14) and IGH rearrangement. Although considerable advances have been made in the understanding of the biology and molecular pathogenesis of PCL, further clinical, laboratory, and genetic studies of PCL associated with such a rare IGH rearrangement would be necessary in the future. To the best of our knowledge, this is the first report of PCL associated with t(6;14) as a sole chromosomal abnormality.</P>
Lee, Sang-Guk,Lim, Gayoung,Cho, Sun Young,Suh, Jin-Tae,Lee, Hee Joo,Baek, Sun Kyung,Lee, Woo-In,Yoon, Hwi-Joong,Park, Tae Sung S. Karger AG 2011 Acta haematologica Vol.126 No.3
<P>Abstract</P><P>No abstract available</P><P>Copyright © 2010 S. Karger AG, Basel</P>
Han, Kyung Ah,Woo, Doyeon,Kim, Seungjoon,Choii, Gayoung,Jeon, Sangmin,Won, Seoung Youn,Kim, Ho Min,Heo, Won Do,Um, Ji Won,Ko, Jaewon Society for Neuroscience 2016 The Journal of neuroscience Vol.36 No.17
<P>Neurotrophin-3 (NT-3) is a secreted neurotrophic factor that binds neurotrophin receptor tyrosine kinase C (TrkC), which in turn binds to presynaptic protein tyrosine phosphatase sigma (PTP sigma) to govern excitatory synapse development. However, whether and how NT-3 cooperates with the TrkC-PTP sigma synaptic adhesion pathway and TrkC-mediated intracellular signaling pathways in rat cultured neurons has remained unclear. Here, we report that NT-3 enhances TrkC binding affinity for PTP sigma. Strikingly, NT-3 treatment bidirectionally regulates the synaptogenic activity of TrkC: at concentrations of 10-25 ng/ml, NT-3 further enhanced the increase in synapse density induced by TrkC overexpression, whereas at higher concentrations, NT-3 abrogated TrkC-induced increases in synapse density. Semi-quantitative immunoblotting and optogenetics-based imaging showed that 25 ng/ml NT-3 or light stimulation at a power that produced a comparable level of NT-3 (6.25 mu W) activated only extracellular signal-regulated kinase (ERK) and Akt, whereas 100 ng/ml NT-3 (light intensity, 25 mu W) further triggered the activation of phospholipase C-gamma 1 and CREB independently of PTP sigma. Notably, disruption of TrkC intracellular signaling pathways, extracellular ligand binding, or kinase activity by point mutations compromised TrkC-induced increases in synapse density. Furthermore, only sparse, but not global, TrkC knock-down in cultured rat neurons significantly decreased synapse density, suggesting that intercellular differences in TrkC expression level are critical for its synapse-promoting action. Together, our data demonstrate that NT-3 is a key factor in excitatory synapse development that may direct higher-order assembly of the TrkC/PTP sigma complex and activate distinct intracellular signaling cascades in a concentration-dependent manner to promote competition-based synapse development processes.</P>
Ji Young Ha,Young Hun Choi,조연진,이승현,Seul Bi Lee,Gayoung Choi,Jung-Eun Cheon,Woo Sun Kim 대한영상의학회 2020 Korean Journal of Radiology Vol.21 No.10
Objective: To evaluate the incidence and risk factors of emetic complications associated with the intravenous administration of low-osmolality iodinated contrast media (ICM) in children undergoing computed tomography (CT). Materials and Methods: All children who underwent contrast-enhanced CT between April 2017 and July 2019 were included. Pediatric patients were instructed on the preparative dietary protocol at our institution. Experienced nurses in the radiology department monitored the children during the CT scans and recorded any emetic complications in their electronic medical records. These data were used to calculate the incidence of emetic complications. Various patient factors and technical factors, including fasting duration, the type and volume of ICM, and ongoing chemotherapy, were evaluated to identify risk factors for emetic complications using univariate and multivariate logistic regression analyses. Results: Among the 864 children (mean age, 8.4 ± 5.7 years) evaluated, 18 (2.1%) experienced emetic complications (6 experienced nausea only and 12 experienced nausea and vomiting). None of the children developed aspiration pneumonia. The mean fasting duration of patients with emesis was 7.9 ± 5.7 hours (range, 3–21 hours), whereas that of patients without nausea was 8.7 ± 5.7 hours (range, 0–24 hours). Fasting duration was not associated with the development of nausea and vomiting (p = 0.634). Multivariate logistic regression analysis revealed that ongoing chemotherapy (odds ratio [OR] = 4.323; 95% confidence interval [CI] = 1.430–13.064; p = 0.009), iomeprol use (OR = 7.219; 95% CI = 1.442–36.146; p = 0.016), and iohexol use (OR = 5.241; 95% CI = 1.350–20.346; p = 0.017) were independent risk factors for emetic complications. Conclusion: Only a small proportion (2.1%) of children experienced nausea or vomiting after exposure to low-osmolality ICM. Many children underwent excessive fasting; however, fasting duration was not associated with nausea and vomiting. Moreover, ongoing chemotherapy and the use of iomeprol or iohexol were identified as potential risk factors for emetic complications in children.