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우영종,손영준,조한길,김영옥 대한소아신경학회 2010 대한소아신경학회지 Vol.18 No.1
Basal cell nevus syndrome(BCNS) is a rare autosomal dominant disorder characterized by variable developmental anomalies and predisposition to cancers. The main manifestations include multiple basal cell carcinomas of skins, odontogenic keratocysts, facial dysmorphism, skeletal abnormalities including scoliosis and bifid ribs, palmar and plantar pits, calcification of the falx cerebri, and biparietal frontal bossing. We experienced a case of 12-year-old girl with the clinical features of basal cell nevus syndrome. 기저세포모반증후군은 상염색체 우성 방식으로 유전되는 드문 질환으로 가족력과 특징적인 임상 및 방사선학적 소견으로 진단하며, 이에 대한 가족력이 없는 경우 질환을 조기 진단하는 데 어려움이 있다. 또한, 환아의 나이가 증가함에 따라 동반된 여러 이상을 가져올 수 있으므로 이에 대한 추적 관찰이 필요할 것으로 생각한다. 저자들은 12세 여아에서 우연히 발견된 치성 각화낭으로 인해 기저세포모반증후군으로 진단된 환아를 경험하였기에 문헌고찰과 함께 보고하는 바이다.
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우영종 대한의사협회 2009 대한의사협회지 Vol.52 No.8
Headache is one of the most common medical complaints of adolescents. Estimates are as high as 40 to 70% of the population having some form of headache with up to 75% of 15 year - olds having experienced of significant headaches. While it is apparent that recurrent headache causes significant functional disability, a majority of adolescents with recurrent headache are not likely to seek a medical care. The diagnosis is often overlooked because of this tendency to regard headache as a minor nuisance rather than true disease or the physician’s inability to appreciate its impact on the patient. In dealing with headaches in adolescents, physician must consider both physical and psychological factors in determining the correct diagnosis. The pathophysiology of specific headache type must be understood in order to provide the optimal treatment program. This article discuss the current classification and comprehensive management for chronic headache in adolescents.
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우영종 대한소아청소년과학회 2006 Clinical and Experimental Pediatrics (CEP) Vol.49 No.10
In this article, the following topics will be discussed: What is a developmental disability? What are the risk factors for developmental disability? What are the causes of delayed motor development? What are the early manifestations of developmental disability in young infants? What are the goals of early intervention and the forms or types of early intervention services? What are the dynamis of families with a developmentally delayed child? What is the role of a pediatrician for a evelopmentally disabled child and his or her family?
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6개월 여아에서 일측성으로 발생한 급성 파종성 뇌척수염 1례
박기원, 김근모, 안여현, 우영종, 마재숙, 류인훈(Ki Won Park, Keun Mo Kim, Y대 Hyeon Ahn, Young Jong Woo, Jae Sook Ma, In Hoon Ryu) 대한소아신경학회 1996 대한소아신경학회지 Vol.3 No.2
A six months old female infant was admitted with an abrupt onset of high fever and prolonged left hemiconvulsion followed by left hemiparesis. She hand a history of an acute upper respiratory tract infection with improvement two weeks before the admission. On admssion, EEG showed lazy phenomenon in the right side and diffuse slowing on the right hemisphere. MRI revealed multiple patchy lesions of high signal intensity in the right basal ganglia and periventricular areas on the T2WI. There was no abnormal finding on the magnetic resonance angiography. She had recovered with conservative treatmen and the unilateral brain lesions were progressively disappeared on the follow-up MRI studies. These unilateral demyelination lesions might be considered as one of the causes of acute infantile hemiplegia or hemiconvulsion-hemiplegia syndrome(HHS) in infancy, and the ADEM should be included as a cause of HHS.
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정상 성인과 Alzheimer병 환자 대뇌에서 주요 Gangliosides의 발현
이민철,우영종,김승업,Tadashi Tai 대한병리학회 2002 Journal of Pathology and Translational Medicine Vol.36 No.6
Background : GM1 ganglioside-bound amyloid beta-protein (GM1/A ) has been reported to be involved with senile plaque formation in Alzheimer disease. Methods : To investigate the binding of major gangliosides on senile plaques and neurofibrillary tangles of Alzheimer disease-specific pathology, we developed four monoclonal antibodies -- GM1, GD1a, GD1b, and GT1b -- employing the hydridoma technique, and applied them for immunohistochemical staining at the frontotemporal neocortex and hippocampus of Alzheimer disease brains and agematched control brains. Results : Moderate immunopositivity for GM1 and GD1a was noted on the senile plaques and neurofibrillary tangles. Mild immunopositivity for GD1b and GT1b on neurofibrillary tangles was noted. Strong GD1b immunopositivity was observed on a few neurons and neurites. Strong immunopositivity for GT1b, and moderate immunopositivity for GM1 and GD1a were noted on reactive astrocytes. Conclusions : These observations suggest that GM1 and GD1a may be involved in the formation of senile plaques as well as neurofibrillary tangles in Alzheimer disease brains.
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Primary psoas abscess confused with hip pathology
김영옥,우영종 대한소아청소년과학회 2006 Clinical and Experimental Pediatrics (CEP) Vol.49 No.5
대요근은 12번 흉추의 외연면에서 5번 요추까지의 외연면에 걸쳐 기시하여 대퇴골의 소전자에 부착되는 근육으로 고관절의 굴곡에 관여한다. 이는 후복막 기관으로 여러 혈관에서 풍부한 혈류 공급을 받으며, S자 결장, 공장, 충수 돌기, 요관, 대동맥, 신장, 췌장, 척추, 림프선 등 여러 장기와 인접하고 있어 감염의 호발 부위가 된다. 그러나, 요근 농양 발생시, 그 증상이 비특이적이고 건강한 소아에서 드물게 발병하는 이유로 고관절 질환으로 혼돈되기도 하여, 진단과 치료가 지연되는 예가 많다. 저자들은 평소 건강하던 14세 남아가 침대에서 떨어져 경한 우측 둔부 좌상을 입은 이후, 우측 둔부 통증과 발열 및 보행 불능을 호소하여 고관절 이상을 의심해 시행한 자기 공명 영상에서 우연히 우측 요근내 농양을 발견해 항생제 치료를 시행하였던 증례를 경험하였기에 보고하는 바이다. A healthy, 14-year-old boy presented with right hip pain and consequent fever after falling out of bed while sleeping. The patient could not walk and complained of severe pain with active and passive motion, which consisted mainly in extension and internal rotation of the right hip. Laboratory analysis of the peripheral blood identified leukocytosis and increased levels of acute phase reactants. Magnetic resonance imaging of the hip, which was performed with the expectation of right hip pathology, revealed cellulitis and abscess in the right psoas muscle and associated inflammatory changes in the adjacent presacral fat plane but showed no abnormal lesions in the adjacent pelvic bone and spine. Staphylococcus hominis was cultured from the blood. With empirical antibiotic therapy, the patient recovered fully. We report a case of primary psoas abscess confused with hip pathology in an immunocompetent child without underlying disease.
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